Keel v. H & v. INC.

421 S.E.2d 362, 107 N.C. App. 536, 1992 N.C. App. LEXIS 758
CourtCourt of Appeals of North Carolina
DecidedOctober 6, 1992
Docket9110IC770
StatusPublished
Cited by53 cases

This text of 421 S.E.2d 362 (Keel v. H & v. INC.) is published on Counsel Stack Legal Research, covering Court of Appeals of North Carolina primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.

Bluebook
Keel v. H & v. INC., 421 S.E.2d 362, 107 N.C. App. 536, 1992 N.C. App. LEXIS 758 (N.C. Ct. App. 1992).

Opinion

LEWIS, Judge.

The evidence before the Commission tended to establish the following:

Plaintiff-employee, Roxie Keel, is a 42 year old woman (she was 36 years old at the time of initial diagnosis) with a tenth grade education who worked for defendant dry cleaner from October 1985 until June 1986. For the first three months, plaintiff washed, pressed and removed spots. In January 1986 she began operating a dry-cleaning machine. During the required maintenance of the machine, plaintiff was regularly exposed to perchloroethylene (PCE), a chemical component of the dry-cleaning solution, while cleaning the filters. Plaintiff contended that contact with the fumes of the dry-cleaning solution caused eye irritation and tears, dizziness, perspiration, coughing and later, shortness of breath. The fumes were so strong that on occasion plaintiff was forced to sit down. By June 1986 plaintiff’s shortness of breath became so pronounced that she left the job.

*538 Plaintiff was treated by her family physician until November 1986 when she was referred to a pulmonary specialist, Dr. Albert Driver. Dr. Driver diagnosed plaintiffs condition as interstitial pulmonary fibrosis which he attributed to her exposure to fumes in the workplace. An industrial hygienist, at Dr. Driver’s request, tested defendant’s dry-cleaning premises on 12 November 1986. He found airborne concentrations of PCE that were “only 7°/o of the recommended exposure limits” and determined that there were “no recognized significant health hazards.” The hygienist testified via deposition that the “[potential health effects associated with perchloroethylene are basically irritation of the eyes and upper respiratory system, central nervous system depression, and possible liver/kidney damage.” He stated that human studies revealed no response to seven hours continuous exposure to PCE in concentrations of 100 parts per million. The hygienist speculated that prior occupational exposure might have contributed to plaintiff’s illness, though no evidence of such exposure was present.

Dr. Driver examined plaintiff and was unable to find signs of any causative agent except chemical exposure, nor were symptoms of other causative agents documented in plaintiff’s old medical records. A chest x-ray taken in 1982 was “entirely normal.” As a progressive disease, evidence of interstitial fibrosis would have shown up on x-ray or through other symptoms had the disease pre-dated plaintiff’s employment with defendant. Dr. Driver specifically excluded plaintiff’s history of smoking as a causative factor. He stated that plaintiff did not attribute her illness to work related chemical exposure, but named PCE in answer to specific questions regarding her exposure to chemical fumes in the workplace.

Due to the strong “circumstantial or . .. chronological] association” between the evolution of plaintiff’s symptoms and her employment, Dr. Driver testified: “there’s a reasonable certainty in my mind that the occupational exposure to fumes in the work place were significant factors in the development of interstitial fibrosis.” Dr. Driver pointed to three case reports in the literature which, though not linking PCE to interstitial fibrosis, did report studies “where these solvents ha[d] been injurious to the lungs and, I, I see no reasons why these chemicals could not have affected this particular patient.” As plaintiff has experienced minimal improvement in her condition, Dr. Driver testified that plaintiff is “almost totally impaired” and is unable to “perform any physical exertion.”

*539 Prior to her employment with defendant, plaintiff had held various jobs including four and one-half years in the dry cleaning industry, but had never operated a dry cleaning machine and had never encountered significant respiratory problems. Plaintiff is unable to perform her former jobs or any other job which would require physical exertion. She is without the education or the training to perform other types of work. Plaintiff has been unemployed since leaving defendant’s business in June of 1986.

“For an injury or death to be compensable under our Workmen’s [now Workers’] Compensation Act it must be either the result of an ‘accident arising out of and in the course of the employment’ or an ‘occupational disease.’ ” Booker v. Duke Medical Center, 297 N.C. 458, 465, 256 S.E.2d 189, 194 (1979). “A disease is an occupational disease compensable under N.C. Gen. Stat. 97-53(13) if claimant’s employment exposed him “to a greater risk of contracting this disease than members of the public generally, . . .” and such exposure “significantly contributed to, or was a significant causal factor in, the disease’s development.” Gay v. J. P. Stevens & Co. Inc., 79 N.C. App. 324, 330, 339 S.E.2d 490, 494 (1986) (citing Rutledge v. Tultex Corp., 308 N.C. 85, 101, 301 S.E.2d 359, 369-70 (1983)). Workplace exposure is a significant factor if without the exposure “the disease would not have developed to such an extent that it caused the physical disability which resulted in claimant’s incapacity for work.” Id. (citing Rutledge, 308 N.C. at 102, 301 S.E.2d at 370). The significance of exposure may be determined by medical evidence as well as “(1) the nature and extent of claimant’s occupational exposure, (2) the presence or absence of other non-work-related exposures and components which contributed to the disease’s development, and (3) correlations between claimant’s work history and the development of the disease.” Id. at 331, 339 S.E.2d at 494 (citing Rutledge, 308 N.C. at 105, 301 S.E.2d at 370). Claimant has the burden of proof, but, “if the occupational exposure in question is such that it augments the disease process to any degree, however slight, the employer is liable.” Id. (citing Rutledge, 308 N.C. at 89, 301 S.E.2d at 362); see also, Hansel v. Sherman Textiles, 304 N.C. 44, 53, 283 S.E.2d 101, 106 (1981) (where the claimant has non-occupational infirmities, the Commission should consider whether the occupation accelerated or aggravated the condition and contributed to claimant’s disablement.) “In addition, the substance to which plaintiff was last injuriously exposed need not be in a substance known to cause the disease.” Gay, 79 N.C. App. at 330-31, *540 339 S.E.2d at 494 (citing Caulder v. Waverly Mills, 314 N.C. 70, 74, 331 S.E.2d 646, 649 (1985)).

“Where the Commission awards compensation for disablement due to an occupational disease encompassed by G.S. 97-53(13), the opinion and award must contain findings as to the characteristics, symptoms and manifestations of the disease from which the plaintiff suffers, as well as a conclusion of law as to whether the disease falls within the statutory provision.” Hansel, 304 N.C. at 54, 283 S.E.2d at 106-07.

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Bluebook (online)
421 S.E.2d 362, 107 N.C. App. 536, 1992 N.C. App. LEXIS 758, Counsel Stack Legal Research, https://law.counselstack.com/opinion/keel-v-h-v-inc-ncctapp-1992.