Faciane v. Southern Shipbuilding Corp.

446 So. 2d 770
CourtLouisiana Court of Appeal
DecidedJanuary 16, 1984
DocketCA 0222, CA 0223
StatusPublished
Cited by28 cases

This text of 446 So. 2d 770 (Faciane v. Southern Shipbuilding Corp.) is published on Counsel Stack Legal Research, covering Louisiana Court of Appeal primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.

Bluebook
Faciane v. Southern Shipbuilding Corp., 446 So. 2d 770 (La. Ct. App. 1984).

Opinion

446 So.2d 770 (1984)

Freddie E. FACIANE
v.
SOUTHERN SHIPBUILDING CORP., Employers Mutual Liability Insurance Company.

Nos. CA 0222, CA 0223.

Court of Appeal of Louisiana, Fourth Circuit.

January 16, 1984.
Rehearing Denied March 21, 1984.

*771 Bendana & Carlton, Orlando G. Bendana, New Orleans, for plaintiff-appellant.

Robert P. Hogan, Covington, for defendants-third party plaintiffs-appellants.

Frank J. Peragine, Daniel J. Caruso, Michael R. Daigle, New Orleans, for American Optical Corp.

Before BYRNES, GULOTTA and AUGUSTINE, JJ.

BYRNES, Judge.

This is an appeal from the granting of an exception of no cause of action in a suit by an injured worker against the executive officers of his former employer, Southern Shipbuilding Corporation (hereinafter "Southern"). We reverse.

Appellant was employed by Southern for approximately 18 years as a welder/gauger. His work required him to spend most days inside the hulls of vessels under construction, in an atmosphere laden with various potentially harmful particles such as silica, asbestos, and iron oxide. After experiencing increasing shortness of breath, *772 appellant sought medical help in May of 1977, and was diagnosed as having silicosis, a disabling lung disease.

Two months later, in July 1977, appellant filed suit against the executive officers and various supervisory personnel of Southern, alleging that their negligence resulted in his exposure to the particles which caused his disease. A products liability suit was also filed against the manufacturer of various protective devices which were meant to protect workers from inhalation of the harmful particles, but allegedly did not.

The executive officers filed an exception of no cause of action alleging that appellant's cause of action accrued after the 1976 amendment to the Louisiana Workers' Compensation Act prohibiting tort suits against executive officers. A motion for summary judgment was also filed by several past insurers of the executive officers who contended that their occurrence-type liability policies did not cover damage sustained after the expiration date of those policies. The trial judge granted the motion for summary judgment and maintained the exception of no cause of action, ruling that "plaintiff's cause of action arose May 7, 1977, when his lung disease was diagnosed." It is with this conclusion of the trial judge that we disagree.

THE DISEASE

Silicosis is "a chronic pulmonary disease due to inhalation of dust with a high concentration of silica (SIO2), characterized by widespread fibrosis and clinically by shortness of breath and increased susceptibility to tuberculosis." Blakiston's GOULD MEDICAL DICTIONARY, (4th Ed.1979).

Medical experts agree that silicosis is caused by the body's reaction to the presence of silica particles. These particles react chemically with the body of the victim, creating toxic compounds which irritate bodily tissues and lead to the destruction of certain cells vital to the immunological process. The body's natural cleansing mechanisms, maimed and overworked by a combination of the presence of silica and its toxic effects, break down. The compounds produced in reaction to the presence of silica stimulate the growth of connective tissues which in turn lead to fibrosis, a state where a fibrous growth obscures and impairs healthy tissue. Eventually this process damages the lungs to such an extent that disability occurs. The degree of exposure necessary to create this condition varies from person to person by reason of the individual's make up and bodily chemistry, as well as various environmental influences.

The condition is progressive in nature in the sense that once the disease process begins, it continues to worsen even when the victim is removed from exposure to silica particles. There is no known cure for this disease. Those suffering from silicosis not only experience decreased pulmonary function, but are more vulnerable to diseases of any type, due to lowered body resistance.

By its very nature this type of injury is not immediately manifest but develops slowly over a period of time. The rate at which the disease develops depends on many factors including the type of particle involved, the environment in which the exposure occurs, the type of protective equipment used, the number of years of exposure, and the health and habits of the particular worker. For a good general discussion of the causes and development of silicosis see, Hamilton & Hardy's INDUSTRIAL TOXICOLOGY, (4th Ed.) Revised by Asher J. Finkel, and CLINICS IN CHEST MEDICINE, (Vol. 2 No. 2, May 1981), Occupational Lung Diseases, by Stuart M. Brooks.

THE CAUSE OF ACTION

There is no dispute among medical authorities, or among the parties to this suit, that regardless of the time at which the disability caused by silicosis arises, the disease process itself invariably begins years earlier. The question posed by this appeal is; when does a plaintiff's cause of action for damages caused by silicosis arise?

Appellee argues strenuously that a cause of action accrues only when the disease manifests itself by physical symptoms and *773 disability. This contention is based on the theory that negligence without injury or damage is not actionable. While this is a correct statement of the law, it begs the question at hand. This appeal is concerned with what constitutes injury, in a legal sense, in cases of silicosis, and when that injury occurs. Resolution of this question seems to hinge primarily on how one defines the injury which, when coupled with a defendant's negligence, gives rise to a cause of action.

The trial judge took the position that injury does not occur until the disease is diagnosed or manifested by physical symptoms. While this theory has the advantage of being easy to apply, it ignores the reality of the situation. There is no doubt whatsoever that by the time a plaintiff is diagnosed as having silicosis, the disease has been present for some time. It would be the purest fiction to assert that no injury has occurred before the date of manifestation. Injury occurred when the cumulation of exposure reached the point where the plaintiff contracted the disease. The fact that the symptoms of the disease manifest themselves years later does not change the essential fact that the damage has already been done.

We have concluded that in the unique circumstances presented by cases of cumulative diseases such as silicosis, it is the contraction of the disease which creates a cause of action and not the later manifestation of the consequences of that disease. To hold otherwise would be to ignore the facts. While the ultimate manifestation of the disease by physical disability may be a convenient, easily fixed point at which to find that a cause of action accrues, we cannot disregard the fact that but for the earlier contraction of the disease, there would be no symptoms to manifest themselves.

The problem with this approach is that it is extremely difficult to accurately fix the point in time at which the disease is contracted. This difficulty does not change the unavoidable reality that the disease exists, in the sense that irreversible biological changes which will inevitably result in disease and disability, take place before manifestation of the condition by way of disabling symptoms. There is universal medical opinion that the date of detection is not the date at which the disease is contracted.

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