Clarence Hobbs v. Clinchfield Coal Company Director, Office of Workers Compensation Programs, United States Department of Labor

45 F.3d 819, 1995 U.S. App. LEXIS 940, 1995 WL 36147
CourtCourt of Appeals for the Fourth Circuit
DecidedJanuary 18, 1995
Docket93-2314
StatusPublished
Cited by49 cases

This text of 45 F.3d 819 (Clarence Hobbs v. Clinchfield Coal Company Director, Office of Workers Compensation Programs, United States Department of Labor) is published on Counsel Stack Legal Research, covering Court of Appeals for the Fourth Circuit primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.

Bluebook
Clarence Hobbs v. Clinchfield Coal Company Director, Office of Workers Compensation Programs, United States Department of Labor, 45 F.3d 819, 1995 U.S. App. LEXIS 940, 1995 WL 36147 (4th Cir. 1995).

Opinion

Affirmed by published opinion. Judge MICHAEL wrote the opinion, in which Judge WIDENER and Judge MOTZ joined.

OPINION

MICHAEL, District Judge:

Clarence Hobbs (“Hobbs”) appeals from a decision of the Benefits Review Board (“BRB”) affirming the decision of an Administrative Law Judge (“ALJ”) denying him black lung benefits. The ALJ found that although Hobbs suffers from pneumoconiosis and is totally disabled, the pneumoconiosis is not a contributing cause of his total disability pursuant to 20 C.F.R. § 718.204(c)(4) (1994). We affirm.

I.

Hobbs filed a claim for black lung benefits on September 23, 1982. He has worked in coal mining for thirty-six years and has never smoked. Initially, an ALJ denied Hobbs’ claim, finding that his total disability is not due to pneumoconiosis. The BRB affirmed, but this court vacated the ALJ’s decision because it was unclear whether he applied the correct standard of causation. See Hobbs v. Clinchfield Coal Co., 917 F.2d 790, 791-92 (4th Cir.1990) (stating that pneumoconiosis need only be a contributing cause of a claimant’s total disability rather than the exclusive cause) (citing Robinson v. Pickands Mather & Co., 914 F.2d 35, 38 (4th Cir.1990)). As a result, we remanded for a determination of whether pneumoconiosis was a contributing cause of Hobbs’ total disability. 1

. On remand, an ALJ again denied benefits to Hobbs. He accorded great weight to the opinions of Drs. Sargent, Kress, and Stewart, all of whom found that Hobbs’ disability is caused by obesity and arthritis. In contrast, the ALJ attributed less weight to some doctors who found that pneumoconiosis is a cause of Hobbs’ total disability. After reviewing all of the medical evidence, the ALJ found that pneumoconiosis is not a contributing cause of Hobbs’ total disability. The BRB affirmed the decision of the ALJ, based largely upon his discretion to weigh the medical evidence.

In reviewing the decision below, we are required to adhere carefully to the principle that ve must affirm the ALJ’s factual findings and weighing of the medical evidence where these conclusions of the ALJ are supported by substantial evidence. Thorn v. Itmann Coal Co., 3 F.3d 713, 718 (4th Cir.1993). After reviewing the record, we conclude that substantial evidence supports the ALJ’s decision that pneumoconiosis is not a contributing cause of Hobbs’ total disability. The ALJ set forth reasons why he attributed greater weight to certain medical opinions, and he specifically addressed each medical opinion which disagreed with his ultimate conclusion.

II.

The ALJ attributed great weight to the opinions of Drs. Sargent, Kress, and Stewart in concluding that pneumoconiosis is not a contributing cause of Hobbs’ total disability pursuant to 20 C.F.R. § 718.204(c)(4). 2 Hobbs asserts that the ALJ erred in doing so *821 because those doctors premised their conclusions upon faulty diagnoses of no pneumoco-niosis. See Grigg v. Director, Office of Workers’ Compensation Programs, 28 F.3d 416, 419 (4th Cir.1994) (stating that an opinion finding no respiratory or pulmonary impairment that is premised upon an erroneous finding of no pneumoconiosis is “not worthy of much, if any, weight”). Contrary to Hobbs’ contention, however, these doctors did not premise their conclusions upon findings of no pneumoconiosis. 3 Drs. Sargent and Kress stated only that Hobbs does not suffer from “coal workers’ pneumoconiosis” and they presumed that any respiratory impairment that was present in Hobbs was attributable to coal dust. For example, Dr. Sargent stated:

It is my impression that this man has little or no evidence of interstitial lung disease secondary to coal workers’ pneumoconio-sis.... He does have a fairly impressive history for chronic productive cough and occasional wheezing. In a man who is a non-smoker and has never been a smoker therefore we must infer that this chronic productive cough is due to his exposure to coal dust.

In addition, Dr. Kress found that Hobbs suffers from chronic bronchitis attributable to coal dust exposure, but that his disability results from skeletal problems rather than pneumoconiosis. Although Drs. Sargent and Kress found that Hobbs suffers from respiratory problems arising out of coal mine employment, they both concluded that his total disability was caused by obesity and arthritis, but not by pneumoconiosis.

Hobbs’ argument fails to distinguish the particular medical affliction “coal workers’ pneumoconiosis” from the much broader legal definition of pneumoconiosis used to consider requests for black lung benefits.

For the purpose of the Act, pneumoconio-sis means a chronic dust disease of the lung and its sequelae, including respiratory and pulmonary impairments, arising out of coal mine employment. This definition in-eludes, but is not limited to, coal workers’ pneumoconiosis, anthracosilicosis, • anthra-cosis, anthrosilicosis, massive pulmonary fibrosis, progressive massive fibrosis, silicosis or silicotuberculosis, arising out of coal mine employment. For purposes of this definition, a disease “arising out of coal mine employment” includes any chronic pulmonary disease resulting in respiratory or pulmonary impairment significantly related to, or substantially aggravated by, dust exposure in coal mine employment.

20 C.F.R. § 718.201 (1994).' Although “coal workers’ pneumoconiosis” may be" used synonymously with pneumoconiosis in medical circles, the two terms are distinct legally. First, § 718.201 includes coal workers’ pneumoconiosis as only one of several possible ailments which could satisfy the legal definition of pneumoconiosis. Furthermore, the comparative breadth of the legal definition contained in § 718.201 is indicated by its inclusion of certain disorders which medically are different from pneumoconiosis. Clinically, pneumoconiosis may be described in simple terms as a chronic lung disease marked by an overgrowth of connective tissue caused by the inhalation of certain dusts. J.E. Schmidt, M.D., Attorney’s Dictionary of Medicine and Word Finder P-239 (1993). In contrast, anthracosilicosis, for example, is a darkening and hardening of the lungs caused by the inhalation of certain dusts. Id. at A-289. Although all of the disorders explicitly mentioned in § 718.201 are medically similar, what is important is that a medical diagnosis finding no coal workers’ pneumoco-niosis is not equivalent to a legal finding of no pneumoconiosis.

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Bluebook (online)
45 F.3d 819, 1995 U.S. App. LEXIS 940, 1995 WL 36147, Counsel Stack Legal Research, https://law.counselstack.com/opinion/clarence-hobbs-v-clinchfield-coal-company-director-office-of-workers-ca4-1995.