SAIF Corp. v. Lewis

12 P.3d 498, 170 Or. App. 201, 2000 Ore. App. LEXIS 1680
CourtCourt of Appeals of Oregon
DecidedOctober 4, 2000
Docket97-04909; CA A103052
StatusPublished
Cited by10 cases

This text of 12 P.3d 498 (SAIF Corp. v. Lewis) is published on Counsel Stack Legal Research, covering Court of Appeals of Oregon primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.

Bluebook
SAIF Corp. v. Lewis, 12 P.3d 498, 170 Or. App. 201, 2000 Ore. App. LEXIS 1680 (Or. Ct. App. 2000).

Opinions

[203]*203LINDER, J.

Employer seeks judicial review of a Workers’ Compensation Board (Board) order concluding that claimant’s claim for a “toxic exposure condition” is compensable. The compensability issue centers on the meaning and application of the statutory requirement that the claim must be “established by medical evidence supported by objective findings.” ORS 656.005(7)(a); ORS 656.802(2)(d). We conclude that the requirement of “objective findings” is not satisfied if a medical expert merely listens to a patient’s description of his or her symptoms and, believing the patient and without any verification process, relies on that description to form a diagnosis. We also conclude that, in this case, claimant failed to present “medical evidence supported by objective findings” to establish his claim. We therefore reverse the Board’s order.

The Board found the following facts:

“Claimant worked as a bioscience research technician for [employer]. On February 11, 1997, claimant’s work involved cleaning a building which contained insecticides, herbicides and fungicides in liquid, powder and granular forms. Claimant and his co-workers moved the chemicals and cleaned the room. Claimant also scraped paint from the ceiling and walls to prepare them for repainting. The room was dusty and the work stirred up dust.
“Claimant wore protective clothing, including a charcoal respirator mask. The mask leaked. Claimant also wore goggles part of the time, but he took them off when they became fogged.
“During the job, claimant experienced fatigue and eye irritation. At about 3 p.m., after working, claimant felt ‘flat,’ disoriented, confused, and ‘funny in the eyes.’ He experienced eye irritation, tearing, coughing, and wheezing on his way home. That evening, claimant noticed a ‘yellowish-whitish’ powder in his nostrils. He had difficulty concentrating. By the next morning, claimant had a sore throat, sore neck, fatigue, dizziness, tinnitus, headache, sinus congestion, bright yellow phlegm and sputum, a chemical taste in his mouth, and vision abnormalities.”

Claimant did not seek medical attention until three weeks after his February 11, 1997, exposure. On March 3, [204]*2041997, he saw Dr. Huff, who noted that claimant reported “persistent [symptoms] of Tinnitus, [headache] and fatigue primarily.” Huff observed that the case was a “difficult one to assess as there’s no objective data.” He referred claimant to Dr. Stringham, an industrial medicine specialist.

Stringham saw claimant only once, on March 4, 1997. In the “subjective” portion of his report, Stringham recounted claimant’s description of his symptoms, which had “mostly subsided” by that time:

“By the next day [after the exposure], [claimant] had a mild headache, sense of malaise, weakness, and difficulty concentrating. He had a pressure in his head and his sinuses. He noted ringing in the ears. He had a sore throat. He felt like he was disoriented at times. He felt on the morning of 02/12/97 he brought up some yellow sputum and then blood tinged sputum later in the day. On 02/12/97, he returned to the area [where he had been exposed] and painted with spray paint the next day. Over the next few days, he developed fatigue, anorexia, difficulty concentrating. Although he didn’t feel well, he presumed the symptoms would subside. He continued to have malaise, tinnitus, headache, sinus ache, through the following week of 02/17/97 through 02/21/97. By 02/21/97, he started to feel more normal, although he still had some tinnitus and still felt some pressure in the back of the head around the right ear. He still had a mild headache.”

In the “objective” portion of his report of his examination of claimant, Stringham noted that claimant was “in no acute distress.” Claimant’s sinuses were “nontender.” His hearing was “normal to normal conversation.” All laboratory results were normal, except for “an elevated bilirubin total for reasons unclear, but possibly related to the exposure.” Stringham’s “assessment” of claimant’s condition was “[t]oxic exposure to multiple chemicals.” Stringham further noted that claimant was “getting better spontaneously.”

Later, Stringham reported that, when he saw claimant in early March 1997, claimant was “feeling that he had recovered about 70%.” Stringham believed that claimant was “entirely credible.” He reported:

[205]*205“On a clinical basis he has an exposure. He developed symptoms in a time frame consistent to the exposure. The symptoms have resolved. In my mind it is far more probable on a medical basis that the exposure, given the chemicals involved and the circumstances of his exposure, is the cause of the symptoms.”

Stringham concluded “to a reasonable medical probability” that claimant’s symptoms, “including visual changes, malaise, tinnitus, and headache were related to his toxic exposure on February 11,1997.”

In May 1997, claimant saw Drs. Quarum, Berlin, and Burton, all specialists in occupational and environmental toxicology. Quarum directed that an audiogram be performed on claimant, which showed normal hearing in both ears. Quarum concluded that “[t]here [was] no evidence that [claimant] actually had a specific or toxic exposure, and there [was] also no correlation of his tinnitus with any of the chemicals that he was exposed to based on the information provided.” Quarum’s “best guess” as to the etiology of claimant’s tinnitus, “given his negative audiologic history,” was “possibly due to Aspirin, which is well known as a salicylate to cause tinnitus.” Claimant admitted taking aspirin “on a regular basis,” although the amounts he said he took “would not be considered sufficient to cause this condition unless he actually is taking quite a bit more.” Burton and Berlin concluded that claimant’s complaints were not work-related and more likely were due to some other medical condition that coincided with the possible toxic exposure at work. Quarum and Burton both suggested that any link between claimant’s symptoms and his possible exposure to toxic chemicals was based solely on conjecture.

SAIF denied the claim, and claimant requested a hearing. Following the hearing, the administrative law judge (ALJ) concluded that claimant had not proved the compensability of his claim. The ALJ viewed the case as a battle of the experts regarding the causal link between claimant’s work and the symptoms that he experienced. The ALJ determined that Stringham’s opinion supporting compensability was insufficient because of a “vacuum in the medical literature” regarding the effects of exposure to the multiple chemicals to which claimant was exposed.

[206]*206On review, a divided Board reversed. The Board rejected the ALJ’s conclusion that proving compensability necessarily requires support from “medical literature.” The Board determined that Stringham’s opinion was “more persuasive than those of Drs.

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SAIF Corp. v. Lewis
12 P.3d 498 (Court of Appeals of Oregon, 2000)

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Bluebook (online)
12 P.3d 498, 170 Or. App. 201, 2000 Ore. App. LEXIS 1680, Counsel Stack Legal Research, https://law.counselstack.com/opinion/saif-corp-v-lewis-orctapp-2000.