MR. JUSTICE McDONOUGH
delivered the Opinion of the Court.
This appeal and cross-appeal from the Worker’s Compensation Court involves a claim for widow’s benefits. The State Compensation Insurance Fund (Fund), appeals the lower court’s decision finding claimant’s husband suffered an injury resulting in death under the Montana Worker’s Compensation Act. The claimant, Irene Wood, cross appeals contending that the lower court erred in refusing to provide attorney fees due from the insurer in a lump sum. We affirm the lower court’s decisions in regard to the appeal and the cross-appeal.
Appellant’s Issue
Whether the Worker’s Compensation Court erred in awarding ben[355]*355efits to the widow of a worker based on the conclusion that the worker’s exposure to carbon monoxide caused his heart attack?
The relevant facts are as follows: Irene’s husband, Duane Wood, worked approximately ten years as a mechanic at Ulmer’s Car & Truck in Great Falls. Duane suffered from hypertension and an enlarged heart, musculoskeletal problems, low potassium levels, and cardiovascular disease. Duane was 49 years old when he died of a heart attack on October 29, 1984.
Duane’s doctor had diagnosed and treated most of his various ailments, but had no knowledge of his moderate to severe coronary artery disease until after Duane’s autopsy. The autopsy revealed moderate to severe narrowing due to coronary arteriosclerosis.
Duane smoked a pipe. He had switched from cigarettes to a pipe a few years before his death. Irene testified he did not inhale the pipe smoke. Duane also liked to drink one or two beers after work. The last two years his pace in completing repairs at Ulmer’s slowed considerably.
Duane worked the Friday before his death. On Friday evening Duane skipped dinner and went straight to bed complaining of extreme fatigue and shoulder pain. On Saturday morning he collapsed and was taken to the hospital. When he arrived at the hospital he was suffering from cardiac arrest probably due to ventricular fibrillation. Two days later he died. Duane’s behavior on Friday night indicated he experienced the onset of a myocardial infarction.
Irene testified that approximately two months before his death Duane began to exhibit loss of balance and appetite. During this period he also lost weight and experienced breathlessness and coughing. These symptoms, according to Irene, increased in September of 1984. Duane also complained of cold and fumes at work.
Ulmer’s Car & Truck operates in a building containing seven service bays. The business employs five mechanics and two mechanic’s helpers. The helpers perform routine service and the mechanics handle minor to major repairs. The bays are often full with vehicles undergoing servicing or repair.
Carbon monoxide escapes into the air from the exhaust of cars driven in and out of Ulmer’s shop. When an auto is run in the shop, workers hook a hose to the auto’s tail pipe. The hoses channel the carbon monoxide outside the building. Even when exhaust escapes outside through the hoses, leaky manifolds and other sources associated with running the cars inside contribute to the presence of carbon monoxide in the air of the garage.
[356]*356A twenty four inch fan in the wall of the garage and two ceiling fans help to circulate air and dissipate the carbon monoxide. Employees also help to rid the garage of the gas by opening windows and the doors that access the bays. Employees open the doors and windows more often on warmer days than on colder days. The high temperature recorded in Great Falls on Duane’s last day of work has 51 degrees Fahrenheit.
Irene hired Dr. Anderson to testify on the possible effects of carbon monoxide on individuals with cardiovascular disease. Anderson testified that individuals suffering from cardiovascular disease risk precipitating a heart attack by breathing carbon monoxide. The risk exists, according to Anderson, because carbon monoxide molecules bond more strongly to hemoglobin than oxygen molecules. Anderson testified that where concentrations of carbon monoxide in the air exist, the strength with which carbon monoxide binds to hemoglobin allows it to displace a portion of the oxygen one would normally obtain by breathing. The already reduced ability of individuals suffering from cardiovascular disease to supply the heart with oxygen, when combined with the effects of breathing air contaminated by concentrations of carbon monoxide, may precipitate a heart attack, according to Anderson.
Anderson considered the likelihood that inhalation of carbon monoxide caused Duane’s heart attack. Testing done on February 26, 1985, when the high temperature in Great Falls reached 43 degrees Fahrenheit demonstrated the presence of carbon monoxide in the air of the garage. Anderson testified that the levels of the gas present at that time could have posed a health risk to Duane. Anderson also noted inhalation of the gas in amounts sufficient to cause oxygen deprivation may result in headaches, dizziness, confusion, and coughing. Amounts too small to cause symptoms in ordinary individuals may nevertheless affect those suffering from cardiovascular disease.
Based on the presence of carbon monoxide concentrations in the garage, the compatibility of Duane’s symptoms with the effects of carbon monoxide in the blood, and a lengthy hypothetical propounded by claimant’s counsel incorporating Irene’s testimony and Duane’s autopsy report, the following exchange occurred:
“Q. Taking into account the symptoms suggested by the question over the preceding month period and on the night of decedent’s collapse, and taking into account the levels of carbon monoxide concen[357]*357trations reported in the question, in your opinion did such levels of carbon monoxide aggravate his underlying heart disorder?
“A. It certainly is probable that it did.
“Q. Would the symptoms listed by the decedent be consistent with the levels of carbon monoxide reported in the question?
“A. Yes.
“Q. Taking into account the decedent’s heart condition, as reported in the autopsy, and the levels of carbon monoxide reported, could these levels trigger or precipitate a new stage of pathology or a new dysfunction in the heart condition?
“A. Yes, it could.
“Q. In your opinion, did the levels of carbon monoxide aggravate, hasten, trigger or precipitate a new stage of pathology or a new dysfunction in his heart condition?
“A. It was a precipitating factor, I think.
“Q. And in your opinion, did they do so?
“A. All I can say is that it’s probable that the high levels of carbon monoxide were a precipitating factor in the final stage of events that led to his death.”
On cross-examination, Anderson testified as follows:
“Q. Okay, I think I asked you earlier, but as I understand it, it is not your opinion that the carbon monoxide exposure triggered his heart attack?
“Mr. Bottomly (claimant’s counsel objecting): Wait a minute, that is contrary to his opinion.
“Q. Well, let’s ask you what your opinion is.
“A. Okay, my opinion is that it’s a risk factor that certainly could have triggered — we don’t know, you know, it did with certainty, but it’s certainly a risk factor.
“Q. It’s a risk factor, but you don’t know for certain?
“A.
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MR. JUSTICE McDONOUGH
delivered the Opinion of the Court.
This appeal and cross-appeal from the Worker’s Compensation Court involves a claim for widow’s benefits. The State Compensation Insurance Fund (Fund), appeals the lower court’s decision finding claimant’s husband suffered an injury resulting in death under the Montana Worker’s Compensation Act. The claimant, Irene Wood, cross appeals contending that the lower court erred in refusing to provide attorney fees due from the insurer in a lump sum. We affirm the lower court’s decisions in regard to the appeal and the cross-appeal.
Appellant’s Issue
Whether the Worker’s Compensation Court erred in awarding ben[355]*355efits to the widow of a worker based on the conclusion that the worker’s exposure to carbon monoxide caused his heart attack?
The relevant facts are as follows: Irene’s husband, Duane Wood, worked approximately ten years as a mechanic at Ulmer’s Car & Truck in Great Falls. Duane suffered from hypertension and an enlarged heart, musculoskeletal problems, low potassium levels, and cardiovascular disease. Duane was 49 years old when he died of a heart attack on October 29, 1984.
Duane’s doctor had diagnosed and treated most of his various ailments, but had no knowledge of his moderate to severe coronary artery disease until after Duane’s autopsy. The autopsy revealed moderate to severe narrowing due to coronary arteriosclerosis.
Duane smoked a pipe. He had switched from cigarettes to a pipe a few years before his death. Irene testified he did not inhale the pipe smoke. Duane also liked to drink one or two beers after work. The last two years his pace in completing repairs at Ulmer’s slowed considerably.
Duane worked the Friday before his death. On Friday evening Duane skipped dinner and went straight to bed complaining of extreme fatigue and shoulder pain. On Saturday morning he collapsed and was taken to the hospital. When he arrived at the hospital he was suffering from cardiac arrest probably due to ventricular fibrillation. Two days later he died. Duane’s behavior on Friday night indicated he experienced the onset of a myocardial infarction.
Irene testified that approximately two months before his death Duane began to exhibit loss of balance and appetite. During this period he also lost weight and experienced breathlessness and coughing. These symptoms, according to Irene, increased in September of 1984. Duane also complained of cold and fumes at work.
Ulmer’s Car & Truck operates in a building containing seven service bays. The business employs five mechanics and two mechanic’s helpers. The helpers perform routine service and the mechanics handle minor to major repairs. The bays are often full with vehicles undergoing servicing or repair.
Carbon monoxide escapes into the air from the exhaust of cars driven in and out of Ulmer’s shop. When an auto is run in the shop, workers hook a hose to the auto’s tail pipe. The hoses channel the carbon monoxide outside the building. Even when exhaust escapes outside through the hoses, leaky manifolds and other sources associated with running the cars inside contribute to the presence of carbon monoxide in the air of the garage.
[356]*356A twenty four inch fan in the wall of the garage and two ceiling fans help to circulate air and dissipate the carbon monoxide. Employees also help to rid the garage of the gas by opening windows and the doors that access the bays. Employees open the doors and windows more often on warmer days than on colder days. The high temperature recorded in Great Falls on Duane’s last day of work has 51 degrees Fahrenheit.
Irene hired Dr. Anderson to testify on the possible effects of carbon monoxide on individuals with cardiovascular disease. Anderson testified that individuals suffering from cardiovascular disease risk precipitating a heart attack by breathing carbon monoxide. The risk exists, according to Anderson, because carbon monoxide molecules bond more strongly to hemoglobin than oxygen molecules. Anderson testified that where concentrations of carbon monoxide in the air exist, the strength with which carbon monoxide binds to hemoglobin allows it to displace a portion of the oxygen one would normally obtain by breathing. The already reduced ability of individuals suffering from cardiovascular disease to supply the heart with oxygen, when combined with the effects of breathing air contaminated by concentrations of carbon monoxide, may precipitate a heart attack, according to Anderson.
Anderson considered the likelihood that inhalation of carbon monoxide caused Duane’s heart attack. Testing done on February 26, 1985, when the high temperature in Great Falls reached 43 degrees Fahrenheit demonstrated the presence of carbon monoxide in the air of the garage. Anderson testified that the levels of the gas present at that time could have posed a health risk to Duane. Anderson also noted inhalation of the gas in amounts sufficient to cause oxygen deprivation may result in headaches, dizziness, confusion, and coughing. Amounts too small to cause symptoms in ordinary individuals may nevertheless affect those suffering from cardiovascular disease.
Based on the presence of carbon monoxide concentrations in the garage, the compatibility of Duane’s symptoms with the effects of carbon monoxide in the blood, and a lengthy hypothetical propounded by claimant’s counsel incorporating Irene’s testimony and Duane’s autopsy report, the following exchange occurred:
“Q. Taking into account the symptoms suggested by the question over the preceding month period and on the night of decedent’s collapse, and taking into account the levels of carbon monoxide concen[357]*357trations reported in the question, in your opinion did such levels of carbon monoxide aggravate his underlying heart disorder?
“A. It certainly is probable that it did.
“Q. Would the symptoms listed by the decedent be consistent with the levels of carbon monoxide reported in the question?
“A. Yes.
“Q. Taking into account the decedent’s heart condition, as reported in the autopsy, and the levels of carbon monoxide reported, could these levels trigger or precipitate a new stage of pathology or a new dysfunction in the heart condition?
“A. Yes, it could.
“Q. In your opinion, did the levels of carbon monoxide aggravate, hasten, trigger or precipitate a new stage of pathology or a new dysfunction in his heart condition?
“A. It was a precipitating factor, I think.
“Q. And in your opinion, did they do so?
“A. All I can say is that it’s probable that the high levels of carbon monoxide were a precipitating factor in the final stage of events that led to his death.”
On cross-examination, Anderson testified as follows:
“Q. Okay, I think I asked you earlier, but as I understand it, it is not your opinion that the carbon monoxide exposure triggered his heart attack?
“Mr. Bottomly (claimant’s counsel objecting): Wait a minute, that is contrary to his opinion.
“Q. Well, let’s ask you what your opinion is.
“A. Okay, my opinion is that it’s a risk factor that certainly could have triggered — we don’t know, you know, it did with certainty, but it’s certainly a risk factor.
“Q. It’s a risk factor, but you don’t know for certain?
“A. Right.
“Q. And there were a number of other risk factors in this case?
“A. That’s correct.
“Q. The cigarette smoking, the hypertension; correct?
“A. Right, for developing coronary artery disease, right.
“Q. Was carbon monoxide any greater risk factor than those factors?
“A. Carbon monoxide was a risk factor just in the final event, not in the development of the coronary artery disease, so the others played a role in developing the coronary artery disease, put him at higher risk for having a myocardial infarction but the carbon mon[358]*358oxide was only with respect to having symptoms from coronary disease and eventually developing a myocardial infarction.
“Q. I guess what I’m asking here is can you attribute his heart attack and his sudden death to carbon monoxide as opposed to cigarette smoking, hypertension or anything else, for that matter.
“A. No.”
The Fund contends that Irene failed to carry the burden of proving inhalation of carbon monoxide caused an injury resulting in death under §§ 39-71-721(1), 39-71-119, MCA (1983):
“39-71-721. Compensation for injury causing death.
(1) if an injured employee dies and the injury was the proximate cause of such death . . .
“39-71-119. Injury or injured defined.
‘Injury’ or ‘injured’ means:
“(1) a tangible happening of a traumatic nature from an unexpected cause or unusual strain resulting in either external or internal physical harm and such physical condition as a result therefrom and excluding disease not traceable to injury, except as provided in subsection (2) of this section . . .
“(3) death resulting from injury.”
The Fund cites several cases where this Court affirmed the Worker’s Compensation Court because the claimant failed to demonstrate causation even though “medical possibility” evidence existed. See, Brown v. Ament (Mont. 1988), [231 Mont.158,] 752 P.2d 171, 45 St. Rep. 508; Currey v. 10 Minute Lube (Mont. 1987), [226 Mont. 445,] 736 P.2d 113, 44 St. Rep. 790, Ferdinand v. Lodge No. 456 (Mont. 1986), [221 Mont. 436,] 719 P.2d 775, 43 St. Rep. 955, Wheeler v. Carlson Transport (1985), 217 Mont. 254, 704 P.2d 49, 42 St. Rep. 1177. The Fund contends that the similarity of this case to the cited cases mandates the conclusion that Irene failed to demonstrate causation. We disagree.
In Brown, the claimant failed to come forward with sufficient evidence to corroborate testimony revealing the medical possibility of causation. Brown, 752 P.2d at 175. In Currey, the medical evidence conflicted. Currey, 736 P.2d at 116. The medical experts in Ferdinand never offered testimony that the claimed injury more probably than not caused claimant’s heart attack. Ferdinand, 719 P.2d at 777. And in Wheeler, conflicting evidence also led this court to affirm denial of benefits. Wheeler, 704 P.2d at 54.
Here, Anderson testified carbon monoxide was probably a precipitating factor in causing Duane’s death. Irene’s testimony cor[359]*359roborates Anderson’s opinion. Thus, substantial evidence supports the decision of the lower court.
The Fund contends that Anderson contradicted his earlier testimony under cross-examination when he stated that he could not attribute the heart attack to carbon monoxide as opposed to other factors which could have caused the attack. However, the Fund questioned Anderson only on whether or not he was certain that carbon monoxide caused the fatal illness. Anderson never refuted or explained away his opinion that carbon monoxide probably precipitated events leading to Duane’s death. Moreover, the lower court has the duty to resolve conflicts in the evidence, and this Court may not substitute its judgment for the judgment of the lower court when substantial evidence supports the lower court’s decision. Ferdinand, 719 P.2d at 776. Thus we affirm the lower court’s finding on causation.
The Fund also contends that insufficient evidence supports the lower court’s finding that the heart attack met the requirement of time definiteness for finding an injury under § 39-71-119, MCA. We disagree.
Evidence disclosed the worsening effects of oxygen deprivation on Duane’s health two months prior to his death. The lower court found that the night before his death, oxygen deprivation precipitated the heart attack. Anderson testified that Irene’s description of Duane’s symptomatology on the Friday before he collapsed indicated the onset of a myocardial infarction probably triggered by inhalation of carbon monoxide. Thus, while Duane’s cardiovascular disease developed over time, the event that deprived his heart of enough oxygen to continue pumping probably occurred on his last day of work. Under these circumstances, substantial evidence supports the finding of time definiteness by the lower court. Daniels v. Kalispell Regional Hospital (Mont. 1988), [230 Mont. 407,] 750 P.2d 455, 45 St. Rep. 310.
Cross Appellant’s Issue
Whether the lower court erred in denying lump sum attorney fees for future benefits?
On cross-appeal Irene contends that the lower court erred in refusing to provide the attorney fees due from the Fund for future benefits in a lump sum. We disagree. The applicable statute, § 39-71-611, MCA (1983), provides for an award of attorney fees and costs from the insurer. The amount and kind of fees, lump sum or [360]*360periodic, must be reasonably established by the Worker’s Compensation Court. The lower court acts reasonably and within its discretion by refusing to lump sum attorney fees owed for benefits which may never accrue. Davis v. Jones (Mont. 1987), [229 Mont. 158,] 745 P.2d 362, 365, 44 St. Rep. 1859, 1862. Thus, we affirm the cross appeal.
MR. CHIEF JUSTICE TURNAGE and MR. JUSTICES HARRISON and GULBRANDSON concur.