Shisslak v. Saul

CourtDistrict Court, D. Massachusetts
DecidedFebruary 7, 2020
Docket1:19-cv-10768
StatusUnknown

This text of Shisslak v. Saul (Shisslak v. Saul) is published on Counsel Stack Legal Research, covering District Court, D. Massachusetts primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.

Bluebook
Shisslak v. Saul, (D. Mass. 2020).

Opinion

UNITED STATES DISTRICT COURT DISTRICT OF MASSACHUSETTS ___________________________________ ) ABIGAIL ELIZABETH SHISSLAK, ) ) Plaintiff, ) ) Civil Action v. ) No. 19-10768-PBS ) ANDREW M. SAUL, ) Commissioner of Social ) Security Administration,1 ) ) Defendant. ) ______________________________ )

MEMORANDUM AND ORDER February 7, 2020 Saris, D.J. Introduction Plaintiff Abigail Elizabeth Shisslak brings this action under 42 U.S.C. §§ 405(g) and 1383(c)(3) for judicial review of a final decision denying her applications for Supplemental Security Income and Children’s Insurance Benefits. She claims that the Administrative Law Judge (“ALJ”) erred by failing to accord proper weight to the opinion of her primary treating physician, failing to fully consider the vocational expert’s testimony, and improperly disregarding Plaintiff’s testimony.

1 Andrew M. Saul has been substituted pursuant to Fed. R. Civ. P. 25(d) for Nancy A. Berryhill, the former Acting Commissioner of the Social Security Administration. For the following reasons, the Court DENIES Plaintiff’s motion for an order reversing the decision of the Commissioner (Docket No. 9) and ALLOWS Defendant’s motion for an order

affirming the Commissioner’s decision (Docket No. 15). Factual Background The following facts are taken from the administrative record. I. Education and Occupational History Plaintiff was 20 years old when she filed for Supplemental Security Income and Child Insurance Benefits in 2016, alleging disability as of January 1, 2016. She finished high school but did not complete any type of vocational training. Plaintiff has previously worked at two stores. As a salesclerk at a frozen yogurt store, she stood 6-8 hour a day serving customers. She also often lifted product

containers from the storage area to refill product stocks. Later, she worked on the sales staff at a toy store supervising 2-3 people. The job required her to sit 8-9 hours a day and to occasionally walk, stand, kneel, and crouch. II. Medical History Plaintiff stated that she suffered “a lot of pain while growing up,” which her pediatrician referred to as growing pains. R. 373. She also reported poor muscle mass and chronic fatigue since childhood. Symptoms became more severe in her mid- teens. In 2014, when she was eighteen years old, she began seeking treatment for many symptoms including memory issues, narcolepsy, migraines, blurry vision, spine issues, and muscle

weakness and numbness. From April 12, 2013 to November 21, 2016, Plaintiff was seen by a clinical social worker, June Atkind, LICSW for a psychiatric disorder. R. 369. Based on Atkind’s report produced on November 23, 2016, Plaintiff was diagnosed with an unspecific Adjustment Disorder, Ehlers-Danlos Syndrome (“EDS”),2 and attention and memory issues. Id. However, the social worker did not provide any treatment record. R. 369-71. Due to her sleep-related issues, Plaintiff participated in a sleep study on April 10 and 11, 2014. The results of her polysomnography, an all-night sleep study, were normal. However, the results of her multiple sleep latency test, or “daytime nap study,” showed “narcolepsy without cataplexy.”3

On January 31, 2015, Plaintiff met with neurologist Dr. Allan Ropper for a polysymptomatic illness and a number of unclear, tentative diagnoses. Among others, she reported she had been diagnosed with EDS for her connective tissue disorder.

2 EDS is a group of hereditary disorders that affect connective tissues and are generally characterized by joint hypermobility (overly flexible joints) and stretchy, fragile skin. 3 Cataplexy is a condition involving abrupt attacks of muscular weakness triggered by an emotional stimulus such as happiness, anger, fear, or surprise. However, Dr. Ropper could not provide a certain diagnosis, noting that “no genetic affirmation” or other traits except for hypermobility of joints would suggest an EDS diagnosis. R. 348.

On February 12, 2015, Plaintiff was seen by Dr. Steven M. Vandor, her primary care physician. R. 344. She reported narcolepsy, asthma and “a very complicated and difficult medical history” revolving around EDS Type III.4 Id. In particular, she reported many issues associated with her diagnosis of EDS, including joint hypermobility, cognitive issues, chronic joint and muscle pain, and visual issues. Dr. Vandor recommended aqua therapy and specialist evaluation. A medical report dated June 19, 2015 revealed that Dr. Vandor did not have a clear etiology for Plaintiff’s chronic history of joint pains and was waiting for her genetic testing results to confirm the diagnosis. He also noted that Plaintiff experienced headaches that appeared

“migrainous in nature.” R. 343. On July 30, 2015, Dr. Michael Erkkinen conducted a neurologic consultation to evaluate Plaintiff’s “multiple complaints,” including headaches, chronic fatigue and paresthesia (an abnormal sensation of the skin). R. 353.

4 EDS Type III, now known as “hypermobile EDS,” is mainly characterized by hypermobility in both large and small joints, leading to frequent dislocation and subluxation (partial dislocation) of joints. Unlike the other twelve subtypes of EDS, the genetic basis for EDS III is unknown. Physical examination revealed no abnormalities except for “very mild hyperextensibility at Plaintiff’s wrists and hands.” R. 356. Dr. Erkkinen also opined that Plaintiff’s headaches sounded

migrainous. R. 357. He was “suspicious” of the EDS diagnosis “particularly given her largely unrevealing genetic screen.” Id. With regard to Plaintiff’s cognitive and psychological functioning given her complaints concerning memory issues and episodes of sleepiness, Dr. Erkkinen suggested a formal neuropsychologic evaluation. Plaintiff met again with Dr. Vandor for headaches on September 11, 2015. She reported “some mild fatigue” but no concerning symptoms. R. 342. Dr. Vandor suggested continuing the use of amitriptyline, an antidepressant used to treat migraines, as “[s]he has had a good response with resolution of headache symptoms” on the medication. Id. He also noted her “chronic

joint pains.” Id. On October 2, 2015, Dr. Neal K. Lakdawala evaluated Plaintiff in the cardiovascular genetics clinic at Brigham and Women’s Hospital. He noted she had undergone comprehensive genetic testing for connective tissue disease, which identified a variant of uncertain significance in the gene MYH11. Although Plaintiff reported ongoing symptoms of joint pains and fatigue, Dr. Lakdawala found she had an “ambiguous cardiac phenotype” at worst. R. 358-59. He stated her genetic testing failed to identify genes associated with the genetic connective tissue disorders Marfan, Loeys Dietz, or the vascular type of EDS, and further noted her most prominent symptoms were likely related to

the abnormal relaxation and contraction of her blood vessels. From October 25 to 26, 2015, Plaintiff was hospitalized at St. Elizabeth Medical Centre for a sudden onset of back pain. However, a chest x-ray and a magnetic resonance imaging (MRI) report showed no abnormal results. On April 14, 2016, Plaintiff met again with Dr. Erkkinen. Plaintiff reported difficulties with organization and memory and attributed such difficulties to EDS. After examination, Dr. Erkkinen was not overly concerned about her memory problems. Dr. Erkkinen further stated that he was not convinced of her EDS diagnosis and concerned at “how strongly she anchor[ed] to this diagnosis and justifie[d] her medical issues to [it].” R. 362.

However, he did note “hyperextensible joints notably at her wrister, lesser extent at the elbow.” R.

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