Sheppard v. A.C. & S. Co.

498 A.2d 1126, 1985 Del. Super. LEXIS 1314
CourtSuperior Court of Delaware
DecidedMay 16, 1985
StatusPublished
Cited by32 cases

This text of 498 A.2d 1126 (Sheppard v. A.C. & S. Co.) is published on Counsel Stack Legal Research, covering Superior Court of Delaware primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.

Bluebook
Sheppard v. A.C. & S. Co., 498 A.2d 1126, 1985 Del. Super. LEXIS 1314 (Del. Ct. App. 1985).

Opinion

POPPITI, Judge.

The matter before the Court is a Motion for Summary Judgment by several defendants, based upon provisions of the statute of limitations, 10 Del.C. § 8119. 1 The undisputed facts are that plaintiff Deronda Sheppard (hereinafter plaintiff) was an employee at the E.I. DuPont deNemours and Co. Inc.’s (hereinafter DuPont) Edgemoor Plant in Edgemoor, Delaware from 1937 until 1938, and again from 1945 until 1975. The plaintiff alleges that during both time periods he was exposed to asbestos fibers in dangerous concentrations. The defendants in this action were engaged in the mining, manufacturing, or distributing of asbestos or asbestos-related products which the plaintiff alleges were supplied to DuPont.

Plaintiff’s symptoms of illness first became apparent in the early 1970s, when he experienced shortness of breath. 2 In 1978 *1128 or 1979 he began to experience thickening of saliva and, in 1979, he had blood-streaked sputum and breathing problems. Apparently as a result of these more significant complaints he was examined by Dr. Robert G. Altschuler, a specialist in internal medicine, on September 28, 1979. In preparation for that examination, Dr. Louis Copman performed a chest x-ray. Dr. Cop-man interpreted the x-ray and forwarded the results in a written report to Dr. Alt-schuler by letter dated September 4, 1979. Therein Dr. Copman articulated his medical diagnosis as follows:

IMPRESSION: BILATERAL PLEURAL THICKENING ALONG THE LATERAL CHEST WALLS, GREATER ON THE LEFT, WHICH HAS INCREASED VERY SLIGHTLY SINCE THE PREVIOUS EXAMINATION OF 1/2/74 AND IS BELIEVED TO BE A MANIFESTATION OF ASBESTOSIS. IT WOULD BE INTERESTING TO KNOW IF THE PATIENT HAS HAD SUCH EXPOSURE. THERE IS NO MANIFESTATION OF ACTIVE PULMONARY DIS-EASE_ 3 (Underscore added.)

As a result of the x-ray and the accompanying report, Dr. Altschuler during the September 28, 1979 examination discussed with plaintiff his possible exposure to asbestos. Plaintiff recalls discussing his previous exposure to asbestos-containing products with Dr. Altschuler during that examination and further recalls that Dr. Altschu-ler advised him that he thought he was suffering from pulmonary asbestosis.

Dr. Altschuler again saw plaintiff on December 5, 1979 and again discussed with plaintiff his impression that plaintiff was suffering from an asbestos-related health problem. As a result of this examination plaintiff was referred to Dr. John J. Cha- *1129 balko, a pulmonary specialist who examined the plaintiff on January 3, 1980. He forwarded a report to Dr. Altschuler on January 8, 1980 which reads in pertinent part as follows:

His occupation for 43 years has been as a welder of all types of metals.... There has been a rare asbestos exposure when he either applies or removes insulating material from pipes. The work has occurred both outdoors and indoors. He occasionally has sawed pieces of asbestos as well. He feels that his exposure or potential exposure to asbestos has been very minimal.
My assessment at this time is a man with a long history of cigarette smoking and a history compatible with chronic bronchitis which appears to be improving. The history of hemoptysis six months ago is a bit bothersome especially in a person who has been a cigarette smoker. Mr. Sheppard seemed to pass this off as being not very important. Bronchoscopy is usually indicated in patients who have history of cigarette smoking and hemoptysis in that endo-bronchial malignancy can be responsible for this symptom. The presence of bilateral pleural disease in a patient with asbestos exposure certainly raises the possibility that the pleural disease is consequent to asbestos exposure. I could find no other good explanation for its presence.
Mr. Sheppard seemed somewhat reluctant to have investigations performed although I did not mention the possibility of bronchoscopy. I did, however, schedule him for a pulmonary function evaluation and arterial blood gases to be performed at St. Francis. These results will be reported to you. Mr. Sheppard was not interested in any further follow-up at this office.... Insofar as his pleural disease is concerned, I think it would be reasonable to follow this along with chest x-rays at regular intervals. Oblique views might help in visualizing additional pleural disease or calcification of the diaphragm. If things continue to progress, we should consider pleural biopsy. (Underscore added.)

Because the plaintiff was still experiencing breathing problems in 1981, Dr. Altchu-ler referred him to Dr. Zakir Hossain, a thoracic and cardiopulmonary specialist. Dr. Hossain performed a bronchoscopie examination on January 21, 1981 and filed a report with Dr. Altschuler by letter dated January 27, 1981 which reads in pertinent part as follows:

My clinical impression at this time is that he is suffering from chronic bronchitis secondary to chronic cigarette smoking.

On September 23, 1981 at the request of the DuPont Company, the plaintiff was examined by Dr. Joseph F. Kestner, a pulmonary specialist and associate of Dr. Chabal-ko. Dr. Kestner filed a report with the DuPont Company which reads in pertinent part as follows:

His chest radiographs show bilateral lateral pleural thickening and his pulmonary function tests support the presence of minimal obstructive impairment....
The bilateral lateral pleural thickening seen on chest radiographs is most likely related to previous asbestos exposure. (Underscore added.)

Finally on May 24,1982, the plaintiff was examined by Dr. Susan Daum, an occupational disease specialist. As a result of the examination and some tests Dr. Daum concluded that the plaintiff had asbestosis as well ás asbestos-related pleural disease, and she so informed him. The instant suit was filed on December 6, 1982.

There is no dispute that the provisions of the two-year statute of limitations, 10 Del. C. § 8119, govern the case at bar. The statute provides that the limitations period begins to run “from the date upon which it is claimed such injuries were sustained.” Plaintiff argues in essence that in the case at bar the statute began to run from the date of the actual clinical diagnosis of asbestosis by Dr. Daum on May 24,1982. On the other hand, the defendants’ position is *1130 that the cause of action is time-barred due to presence of general symptoms, namely, shortness of breath, as early as 1974 and actual physician-identified clinical symptoms of asbestos-related health problems by Dr. Altschuler and Dr. Copman in 1979. The diagnosis by Dr. Altschuler and Dr. Copman in 1979 was pleural thickening, the substance and etiology of which was communicated to and discussed with the plaintiff.

The issue as to when an injury is “sustained” within the context of an asbestos-related disease (an inherently unknowable disease) has recently been addressed by this Court in an opinion and order which was affirmed on appeal by the Delaware Supreme Court.

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Bluebook (online)
498 A.2d 1126, 1985 Del. Super. LEXIS 1314, Counsel Stack Legal Research, https://law.counselstack.com/opinion/sheppard-v-ac-s-co-delsuperct-1985.