[200] LUSK, J.
This is an appeal from a judgment for the defendant based on the verdict of a jury in a medical malpractice action.
The defendant, Dr. J. W. Dowsett, is a duly licensed physician and surgeon who maintains an office in Portland, Oregon, and specializes in obstetrics. He attended the plaintiff, Arloine Mayor, a married woman, during her pregnancy and at the birth of her second child. Under his direction, Dr. L. M. Sutherland administered a spinal anesthetic shortly before the birth of the child. Following the birth plaintiff became paralyzed from the neck down and has so remained. The evidence indicates that this condition is permanent.
The plaintiff alleged in her complaint that her paralysis was caused by the negligence of the defendant in improperly positioning, handling, placing and securing plaintiff, thereby causing and permitting the spinal anesthetic solution injected in plaintiff’s spine to contact, shock and destroy the motor nerves in plaintiff’s spine. There were other charges of negligence, but this was the only one submitted to the jury by the trial judge.
The defendant moved for a directed verdict on the ground of insufficiency of the evidence of negligence or proximate cause. The motion was denied and the defendant urges in this Court that it should have been allowed. We will consider this question first.
Defendant does not now contend that there is no evidence of negligence. His claim is that there is no evidence that the alleged negligence was the cause of plaintiff’s injury.
The plaintiff was 34 years of age in 1959. She was employed in the office of a contracting firm and also [201] was a vocalist and derived an income from ¡her singing. There is substantial evidence that she was in good health during her pregnancy and at the time of the birth of her child. She had no illness during this period except that for a week in September, 1959, she suffered from a cold which, however, caused her no loss of time from her job, but made it necessary for her to cancel several singing engagements. Dr. Dow-sett prescribed for her cold fluids, aspirin and cough medicine. He testified that she surmounted that complaint. The last time that Dr. Dowsett saw the plaintiff before she went to the hospital was on November 2, 1959. He testified that at that time her condition was “real good.” During delivery, Dr. Dowsett testified, “[h]er condition was excellent. She complained of nothing at all. She was real happy, * '* There is contradictory evidence as to plaintiff’s general condition of health, but for present purposes it need not be considered.
Plaintiff was admitted to Emanuel Hospital in Portland on the evening of November 6, 1959. The following morning shortly before six o’clock she was taken to the delivery room. The defendant arrived at the hospital about seven o’clock, a.m., and attended her until the baby was born. At 9:48 a.m. Dr. L. M. Sutherland, a resident physician at the hospital, administered a saddle block spinal anesthetic at the direction of the defendant. The injection was in the lower part of the spine between the fourth and fifth lumbar vertebrae. The anesthetic, as described in a hypothetical question addressed to a medical witness for the plaintiff, was “a two and a half per cent Novocain, being ten per cent Novocain or Procaine in saline solution that was diluted by * * * spinal fluid from the patient herself, * * * making it one part of [202] anesthetic solution and three parts of spinal fluid, a total of two cubic centimeters being injected into the * * * spinal canal # * *.” It is not disputed that the injection was made at a proper place and all the evidence shows that the solution used was a mild one. At 9:52 a.m. the plaintiff gave birth to a normal baby boy.
The hospital record shows that at 11:20 a.m. on November 7 the plaintiff complained of intermittent chilling, and Dr. Dowsett was notified; that at 11:45 a.m. after the plaintiff was removed to her private room, she complained of shortness of breath; and at 12:30 p.m. the following entry made by the nurse appears : “BP 104/80. Pt rigid and movement of arms spastic—nauseated.” Again at 8 p.m. on November 7 the notation “Shortness of breath” appears, and at 9 p.m.: “Patient seems to have difficulty breathing.”
Dr. Dowsett left the hospital at noon on November 7. At 7:15 on the morning of November 8 he received a message that the plaintiff had a convulsion and was totally unconscious. He returned immediately to the hospital. Dr. Sutherland, Dr. Bruce Kvemland, a neurological surgeon, and Dr. D. W. Clouser, an anesthesiologist, were also summoned. The hospital record shows that at 7 a.m. on November 8th the plaintiff was breathing with grunts, and at 8:30 respiration ceased. She was limp all over her body. Her neck and face and extremities were flaccid. Dr. Sutherland testified that the patient was unconscious, that she “was eyanosed and she was moving her extremities in a restless way, but she obviously was not responding to knowing what she was doing.” A tracheotomy was done by Dr. Kvernland, and a tracheotomy tube was inserted and connected with a Bird respirator, a device [203] that breathes automatically for the patient by the use of oxygen and carbon dioxide. By this means respiration was restored and maintained.
Dr. Kvernland made a working diagnosis of myasthenia gravis, a disease characterized by paralysis of the muscles, and to test this assumption, administered a drug called Prostigmin, following which the plaintiff was able to wiggle her fingers and toes. He also found that by tickling the bottom of her feet and pricking her feet and hands with a needle, she had feeling.
On November 27, 1959, the plaintiff was removed to the University of Oregon Medical School Hospital, where she remained until May 10, 1961. While there has been some improvement in her condition, at the time of the trial, in June, 1963, she was still partially paralyzed from the neck down, and could breathe only with artificial aid—the Bird respirator by day, and a mechanical rocking bed at night.
The plaintiff testified:
“Q Did anything unusual happen during the course of their attempting to deliver the baby?
“A They were having a hard time with the baby and he kept saying I was going to have to work harder. And it seemed like two nurses came on in each side and held my arms, and helped me struggle, and then two doctors, and it seemed like I was being pulled apart like a chicken, and got mj?- legs up quite high and got my neck down until it got pretty hard to breathe. And they got my feet here and my arms up here and my neck was being pinched.
“Q Did your chin come in contact with your chest?
“A Yes.
[204] “Q Did that interfere with yonr breathing?
“A Yes, it did. And I thought it would be over in a minute. And I kept struggling and everything went.
“Q Can you describe a little bit more for us what you meant or what sensation you had when you say, everything went?
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[200] LUSK, J.
This is an appeal from a judgment for the defendant based on the verdict of a jury in a medical malpractice action.
The defendant, Dr. J. W. Dowsett, is a duly licensed physician and surgeon who maintains an office in Portland, Oregon, and specializes in obstetrics. He attended the plaintiff, Arloine Mayor, a married woman, during her pregnancy and at the birth of her second child. Under his direction, Dr. L. M. Sutherland administered a spinal anesthetic shortly before the birth of the child. Following the birth plaintiff became paralyzed from the neck down and has so remained. The evidence indicates that this condition is permanent.
The plaintiff alleged in her complaint that her paralysis was caused by the negligence of the defendant in improperly positioning, handling, placing and securing plaintiff, thereby causing and permitting the spinal anesthetic solution injected in plaintiff’s spine to contact, shock and destroy the motor nerves in plaintiff’s spine. There were other charges of negligence, but this was the only one submitted to the jury by the trial judge.
The defendant moved for a directed verdict on the ground of insufficiency of the evidence of negligence or proximate cause. The motion was denied and the defendant urges in this Court that it should have been allowed. We will consider this question first.
Defendant does not now contend that there is no evidence of negligence. His claim is that there is no evidence that the alleged negligence was the cause of plaintiff’s injury.
The plaintiff was 34 years of age in 1959. She was employed in the office of a contracting firm and also [201] was a vocalist and derived an income from ¡her singing. There is substantial evidence that she was in good health during her pregnancy and at the time of the birth of her child. She had no illness during this period except that for a week in September, 1959, she suffered from a cold which, however, caused her no loss of time from her job, but made it necessary for her to cancel several singing engagements. Dr. Dow-sett prescribed for her cold fluids, aspirin and cough medicine. He testified that she surmounted that complaint. The last time that Dr. Dowsett saw the plaintiff before she went to the hospital was on November 2, 1959. He testified that at that time her condition was “real good.” During delivery, Dr. Dowsett testified, “[h]er condition was excellent. She complained of nothing at all. She was real happy, * '* There is contradictory evidence as to plaintiff’s general condition of health, but for present purposes it need not be considered.
Plaintiff was admitted to Emanuel Hospital in Portland on the evening of November 6, 1959. The following morning shortly before six o’clock she was taken to the delivery room. The defendant arrived at the hospital about seven o’clock, a.m., and attended her until the baby was born. At 9:48 a.m. Dr. L. M. Sutherland, a resident physician at the hospital, administered a saddle block spinal anesthetic at the direction of the defendant. The injection was in the lower part of the spine between the fourth and fifth lumbar vertebrae. The anesthetic, as described in a hypothetical question addressed to a medical witness for the plaintiff, was “a two and a half per cent Novocain, being ten per cent Novocain or Procaine in saline solution that was diluted by * * * spinal fluid from the patient herself, * * * making it one part of [202] anesthetic solution and three parts of spinal fluid, a total of two cubic centimeters being injected into the * * * spinal canal # * *.” It is not disputed that the injection was made at a proper place and all the evidence shows that the solution used was a mild one. At 9:52 a.m. the plaintiff gave birth to a normal baby boy.
The hospital record shows that at 11:20 a.m. on November 7 the plaintiff complained of intermittent chilling, and Dr. Dowsett was notified; that at 11:45 a.m. after the plaintiff was removed to her private room, she complained of shortness of breath; and at 12:30 p.m. the following entry made by the nurse appears : “BP 104/80. Pt rigid and movement of arms spastic—nauseated.” Again at 8 p.m. on November 7 the notation “Shortness of breath” appears, and at 9 p.m.: “Patient seems to have difficulty breathing.”
Dr. Dowsett left the hospital at noon on November 7. At 7:15 on the morning of November 8 he received a message that the plaintiff had a convulsion and was totally unconscious. He returned immediately to the hospital. Dr. Sutherland, Dr. Bruce Kvemland, a neurological surgeon, and Dr. D. W. Clouser, an anesthesiologist, were also summoned. The hospital record shows that at 7 a.m. on November 8th the plaintiff was breathing with grunts, and at 8:30 respiration ceased. She was limp all over her body. Her neck and face and extremities were flaccid. Dr. Sutherland testified that the patient was unconscious, that she “was eyanosed and she was moving her extremities in a restless way, but she obviously was not responding to knowing what she was doing.” A tracheotomy was done by Dr. Kvernland, and a tracheotomy tube was inserted and connected with a Bird respirator, a device [203] that breathes automatically for the patient by the use of oxygen and carbon dioxide. By this means respiration was restored and maintained.
Dr. Kvernland made a working diagnosis of myasthenia gravis, a disease characterized by paralysis of the muscles, and to test this assumption, administered a drug called Prostigmin, following which the plaintiff was able to wiggle her fingers and toes. He also found that by tickling the bottom of her feet and pricking her feet and hands with a needle, she had feeling.
On November 27, 1959, the plaintiff was removed to the University of Oregon Medical School Hospital, where she remained until May 10, 1961. While there has been some improvement in her condition, at the time of the trial, in June, 1963, she was still partially paralyzed from the neck down, and could breathe only with artificial aid—the Bird respirator by day, and a mechanical rocking bed at night.
The plaintiff testified:
“Q Did anything unusual happen during the course of their attempting to deliver the baby?
“A They were having a hard time with the baby and he kept saying I was going to have to work harder. And it seemed like two nurses came on in each side and held my arms, and helped me struggle, and then two doctors, and it seemed like I was being pulled apart like a chicken, and got mj?- legs up quite high and got my neck down until it got pretty hard to breathe. And they got my feet here and my arms up here and my neck was being pinched.
“Q Did your chin come in contact with your chest?
“A Yes.
[204] “Q Did that interfere with yonr breathing?
“A Yes, it did. And I thought it would be over in a minute. And I kept struggling and everything went.
“Q Can you describe a little bit more for us what you meant or what sensation you had when you say, everything went?
“A At first I was helping, you know, working as hard as I could and he kept telling me to push down on all the muscles that I could, and, I think, I was doing a good job helping. And all of a sudden I felt there was no push left and everything collapsed.
“Q Could you move your arms and legs?
“A They were holding me with my arms and legs and I don’t know at that time if I could have worked them myself or not.
“Q Were you conscious when the baby was born?
“A Yes, and when he cried.
“Q Do you know whether you lost consciousness at any time?
“A No, I really don’t know.
“Q What happened when the baby was born?
“A They moved me back to the cart they brought me down on.
“Q Was that done immediately?
“A As soon as they kind of cleaned me up and put me over there.
“Q How long did you stay on the cart in the delivery room?
“A I think it was approximately a half hour. It could have been more or less.
“Q Do you remember going back down to the room?
“A Yes, I remember going to the room.
[205] “Q Do you remember being put in the bed?
“A I remember being put in the bed and they came soon after for lunch.
“Q 'What happened when lunch came ?
“A I really don’t know, I was so tired. But the potatoes and gravy looked so good and I tried to reach up for it and I couldn’t And I thought I was just so tired.
“Q How long was that after you came down from the delivery room?
“A About a half hour.
“Q Soon after?
“A Yes, I don’t think I had been in my room but a few minutes when they came with the lunch.
“Q After you noticed you couldn’t move your arms, thereafter could you move?
“A No, I couldn’t move.
“Q Was anything else other than your arms involved?
“A I didn’t try to move anything else and I was so tired, and I tried to keep flat because of the spinal. And from then on everything went hazy.”
As to the question of impairment of sensory perception, the plaintiff testified:
“A I Avas completely numb for the first four months. The doctors Avould come in and stick pins in my feet and go up my leg and I couldn’t feel them, and after about four months, about the time I started to be able to move, then I began to feel the pin pricking at the bottom of my feet. And then it Avas about a month later that I began to feel them in my legs, but then, it was about two or three months later that I felt them in my arms. But it wasn’t until the tenth month that I could feel in my fingertips. My husband would bring the baby and put him by my bed and I couldn’t feel him up until ten months.
[206] “Q Did the feeling -and the muscle movement come back about the same time and about the same degree?
“A It started about the -same time, yes.”
We go to the expert testimony.
There is evidence that it is the standard practice in Portland to place a pillow under the patient’s head when administering a spinal anesthetic in order to elevate the head and guard -against the danger of the anesthetic ascending the spinal canal and attacking the phrenic nerve which controls the -diaphragm and the nerves in the -cranial area. The plaintiff testified that no pillow was used in her ease. The testimony of the defendant and Dr. Sutherland is to the contrary, but, of course, this issue was for the jury.
There is evidence that -one would not expect permanent partial paralysis from the neck down to result from the delivery of a child following a spinal anesthetic when due care and proper practice -are observed. The defendant so testified, and further, that he was familiar with errors in technique or traumatic introduction of the spinal anesthetic that can cause paralysis. The percentage of such cases is very small. One study showed that of 482 patients who had 'had neurological complaints following spinal anesthesia, in the case of four paralysis was caused by the anesthesia.
Dr. -Charles C. Carter, a witness for the defendant, testified that he had -seen eight to twelve cases of permanent partial paralysis resulting from a spinal anesthetic. Dr. Carter is a neurologist, and assistant professor in neurology at the University of Oregon Medical School. He had -supervision -of the plaintiff’s case while she was in the Medical School Hospital.
Besides the defendant, Dr. Sutherland, and Dr. Carter, the medical witnesses on behalf of the defend[207] ant were Dr. Kvernland and Dr. Clouser. All these witnesses gave it as their opinion that the plaintiff’s paralysis was not caused by the anesthetic.
One of the reasons for this opinion was that the plaintiff suffered no impairment of the sensory nerves, but only of the motor nerves. The motor nerves and the sensory nerves emerge at the same level from the spinal cord and come together and fuse. The plaintiff’s phrenic nerve, in the neck area, which supplies the diaphragm, an important muscle in regard to respiration, was damaged, as were the cranial nerves. Had the damage to these motor nerves been caused by the anesthetic the sensory nerves would necessarily have been affected; but, as previously stated, Dr. Kvernland testified to plaintiff’s retention of sensory function demonstrated by pricking her extremities with a needle, and similar testimony was given by the defendant and by Dr. Carter.
Another reason, according to the defendant’s expert witnesses, for ruling out the anesthetic as the cause of plaintiff’s paralysis was that during the process of delivery the plaintiff’s blood pressure remained normal. Had the anesthetic ascended to the cranial nerves, it would have affected the sympathetic nerves which control blood pressure, and, as Dr. Clouser testified, “if many of these nerves are knocked out by spinal anesthetic, the blood pressure tends to drop.” Dr. Clouser concluded that, since the blood pressure did not drop “this spinal stayed down in the saddle block area where it is supposed to.” Dr. Dowsett testified:
“Well, if it had been an immediate reaction to the anesthesia, the patient would have gone into profound collapse, and I mean real collapse. The blood pressure would completely disappear, her [208] breathing would have stopped, she would have been cyanotic and real extreme emergency procedures would have had to be carried out to save her life immediately at the time the spinal was given. If it had been a delayed reaction or a reaction from the anesthetic agent, her paralysis for the anesthesia would not recover, [sic] she would continue to be paralyzed from the time the administration of her spinal block.”
As above noted, however, within less than three hours after the plaintiff’s child was born, her blood pressure dropped to 104/80.
Other reasons for ruling out the spinal anesthetic as the causative agent were that tests of the plaintiff’s spinal fluid made at Emanuel Hospital 22 hours after delivery disclosed only a slight increase of protein count to 62 milligrams per cent and no white blood cells. Dr. Carter testified that “in a majority of cases” if something is wrong with the nerves that come out of the spinal cord it is reflected in abnormality in the spinal fluid, particularly in elevation of the protein count, and that the increase shown was without significance. Dr. Dowsett’s testimony is to the same effect. He said that white blood cells “reveal an irritating agent” within the spinal canal.
Finding nothing abnormal in the spinal fluid, and since “[t]he motor malfunction, the weakness was way out of proportion to any impairment of sensation that we could find,” Dr. Carter directed his “interest away from the anatomy adjacent to the spinal cord in the spinal canal to ether anatomy.” He made a muscle test which indicated that there was impairment of nerve supply to the muscle and that the nerves were not functioning well due to some disease process. The place of impairment was “in the nerve out.”
“Q So that the trouble was as the nerve got [209] closer out of the extremities and not closer to the spinal column, is that so?
“A That was our contention.”
Dr. Carter called the plaintiff’s affliction “infectious polyneuropathy mainly because this is a specific disease entity which doctors recognize as an illness like polio or like appendicitis or like pneumonia and so forth.” “Neuropathy” means “there is something pathologically wrong with that nerve.” He also termed the illness “infections polyneuritis” indicating “an inflammation of the nerve.” This, he said, is a “little more specific term” than neuropathy. The cause of the disease is “undetermined.” The final diagnosis as shown by the hospital record was “Multiple Neuropathy, Chronic, Cause Undetermined.”
On cross-examination Dr. Carter testified:
“Q Now, Doctor, I take it that you relied strongly, as I understood your testimony, in making your final diagnosis, on the fact that you said that Mrs. Mayor’s sensory feeling didn’t ever disappear, is that correct?
“A This is one of the important points, yes.
“Q What difference, in your diagnosis, would there be, if any, if the loss of ¡sensory function and the loss of motor function was paralyzed?
“A Then, of course, I would have to re-evaluate the area of the body involved and I would have to re-evaluate the causes for such involvement.
“Q And that would point back again to the spinal fluid, wouldn’t it?
“A Probably would.”
The sole medical witness for the plaintiff was Dr. Smith, a general practitioner in Portland. Dr. Smith took care of the plaintiff after she returned home from the University of Oregon Medical School Hospital. He had had experience in administering spinal [210] anesthetics. He claimed no expertness in the field of neurology, but based his testimony on his experience, the history of the case and a study of the relevant medical literature. He testified that in his opinion Mrs. Mayor’s paralysis was either directly or indirectly related to the administration of the spinal anesthetic, that “the sequence of events between the administration of the anesthetic and the development of the paralysis is too close and too significant to be ignored.”
On cross-examination Dr. Smith conceded that if the anesthetic reached the upper levels of the spinal canal he would expect an immediate fall in blood pressure, embarrassment in the patient’s breathing and cyanosis, that she would have a cardiac arrest “sooner or later,” and that if the spinal fluid showed no white blood cells and no elevation of protein within 24 or 36 hours after the anesthetic had been given, he would consider that there was no serious effect from the spinal. Later, he testified that he would not know how to interpret the absence of white blood cells, and that a “virus could have caused this condition, certainly that is the basis for the whole diagnosis as it is of an infectious polyneuritis, supposedly caused by a virus.” He said that it was “basically true” that the plaintiff had involvement of the motor nerves, but not the sensory nerves, that he did not know whether this indicated that the damaging agent was not an intradural agent, but that if a neurologist would say that this was the diagnostic test he would not dispute it. Nevertheless, he stated that from “the story * * * I have heard,” the anesthetic rising up the spinal canal could very likely be the explanation of the case. He added that from his study of the literature he had found “that typical quadriplegia, the paralysis that [211] can develop, has developed and has been reported following spinal anesthetic, and the symptoms sometimes come on after the anesthesia has worn off, and that it takes very small concentrations of anesthetic to produce symptoms.” He read from a report: “The incidence of severe neurological complications from spinal anesthesia in San Diego County in a series of 32,828 cases in a five-year period was 0.012 per cent.” He further testified that his experience in spinal anesthesia has been that the anesthetic wears off within from one to three to four hours.
The proof of causation in a medical malpractice case need not always be direct and positive; it may be circumstantial: Olson v. McAtee, 181 Or 503, 517, 182 P2d 979; Clemens v. Smith, 170 Or 400, 407, 134 P2d 424; Carter v. Howard, 160 Or 507, 512, 86 P2d 451; Lippold v. Kidd, 126 Or 160, 174, 269 P 210, 59 ALR 875. Prom the evidence that the defendant was negligent in failing to place a pillow under the plaintiff’s head when the anesthetic was administered, in accordance with the standard practice, a legitimate inference might be drawn that this omission was the proximate cause of the injury sustained by the plaintiff, since this precaution is taken to avoid the danger of the anesthetic ascending the spinal canal. See Champion v. Bennetts, 37 Cal 2d 815, 821, 236 P2d 155. The anesthetic, if it encountered the nerves emanating from the higher levels of the spinal cord, was capable of inflicting the injury which the plaintiff actually suffered. No other specific cause of the injury was testified to by any witness. Neither myasthenia gravis, Dr. Kvernland’s diagnosis, nor infectious polyneuritis, Dr. Carter’s diagnosis (but rejected by Dr. Kvernland), nor multiple neuropathy, the final diagnosis at the University of Oregon Medical School [212] Hospital, indicates a cause, but a result. “Multiple neuropathy” could just as well be used to describe the plaintiff’s paralysis resulting from damage to the nerves caused by the anesthetic as by some unidentified cause.
If it were established without contradiction by the medical evidence that all the manifestations of injury to which the witnesses for the defendant testified must concur in every ease before a conclusion would be justified that the damage came from within the spinal canal and, further, that some of these manifestations—-reduced blood pressure, cyanosis, difficulty in breathing—must have occurred immediately after the introduction of the anesthetic into the spinal canal, defendant’s position might be sustainable. See 2 Harper and James, The Law of Torts, 1100-1101, § 19.11. But this is not the state of the record.
Dr. Smith testified that the symptoms sometimes come on after the -anesthesia has worn off and that it may be as much as four hours before it wears off. Dr. Dowsett recognized that there might be “a delayed reaction” from the anesthetic agent in which case, he said, “she would continue to be paralyzed from the time [of] the administration of her spinal block.” If it were “an immediate reaction to the anesthesia,” then the symptoms of reduced blood pressure, difficulty in breathing and cyanosis would appear immediately. The defendant argues that the evidence shows without dispute that the plaintiff recovered the full use of -her legs while in the delivery room, but we think that the evidence is susceptible of a different interpretation.
Dr. Carter testified that if there had been loss of sensory function as well as motor function he would [213] have to re-evaluate the cause and that would probably point to the spinal fluid.
In any appraisal of the evidence, of course, the plaintiff’s testimony is not to be ignored. She was a competent witness as to whether she had suffered a loss of feeling and she testified that she was “completely numb” for the first four months and that it was after this that she began to recover some muscle movement and at the same time to recover sensory perception.
Her testimony that during delivery “everything collapsed,” that she was unable to reach up for the food they brought her when she was returned to her private room from the delivery room, that she could not move her arms thereafter and did not try to move “anything else” because she was “so tired,” indicates that the process of paralysis had already set in. She experienced shortness of breath within two hours after the birth of the baby, within three hours a marked drop in blood pressure, and on the early morning of November eighth she had a convulsion, was cyanotic, totally unconscious, and respiration had ceased.
While the evidence discloses that absence of white cells and of elevated protein count indicate that the cause of the injury was not an agent within the spinal canal, yet it cannot be said that the medical testimony shows that this evidence is conclusive. Dr. Carter testified that “in the majority of cases,” if something is wrong with the nerves that come out of the spinal cord, it is reflected in the spinal fluid.
We are further of the opinion that res ipsa loquitur applies to this case. The rule as first formulated in Scott v. London and St. Katherine Docks Co., 3 H & C 596, 601, 159 Eng Rep 665, 667, and quoted [214] with approval in. Gow v. Multnomah Hotel, Inc., 191 Or 45, 52, 224 P2d 552, 228 P2d 791, is as follows:
“There must be reasonable evidence of negligence. But where the thing is shown to be under the management of the defendant or his servants, and the accident is such as in the ordinary course of things does not happen if those who have the management use proper care, it affords reasonable evidence, in the absence of explanation by the defendants, that the accident arose from want of care.”
As generally stated today, the conditions necessary for the application of the principle of res ipsa loquitur are these:
“* * * (1) the accident must be of a kind which ordinarily does not occur in the absence of someone’s negligence; (2) it must be caused by an agency or instrumentality within the exclusive control of the defendant; (3) it must not have been due to any voluntary action or contribution on the part of the plaintiff. * * *” Prosser, Law of Torts (2d ed) 201-202, §42.
We have several times said that this doctrine has no place in an action for medical malpractice: Eckleberry v. Kaiser Foundation et al, 226 Or 616, 626-627, 359 P2d 1090, 84 ALR2d 1327; Malila v. Meacham, 187 Or 330, 354, 211 P2d 747; Ritter v. Sivils, 206 Or 410, 413, 293 P2d 211. But these were all cases which arose out of treatment of a patient or surgery for the purpose of effecting a cure, and in which to apply res ipsa loquitur would have impinged upon the established principle that a physician is not, as stated in Ritter v. Sivils, supra, “a warrantor of cure, and if a good result does not ensue from his efforts the doctrine of res ipsa loquitur is not available to his erstwhile patient.” The courts have properly adopted [215] this principle as a measure of needed protection to those engaged in the healing arts against unfounded lawsuits (see Johnston v. Bodis (DC Dist Col) 151 F Supp 345, reversed on other grounds, 102 App D C 209, 210, 251 F2d 917), and we sanction no departure from it, nor from the rule ihat “[i]f the issue turns upon some fact beyond the ken of laymen, expert testimony must be produced showing that the practitioner’s treatment fell below the standard of the profession and was the proximate cause of the condition for which the plaintiff seeks an award of damages.” Ritter v. Sivils, supra. But to say that in an action to recover for personal injury based on negligence—which is all that a malpractice action is—a rule of evidence founded in “common sense”