Mary Ragon as Personal Representative of the Estate of Larry Ragon v. Eli Lilly & Company

CourtIndiana Court of Appeals
DecidedSeptember 10, 2014
Docket93A02-1402-EX-80
StatusUnpublished

This text of Mary Ragon as Personal Representative of the Estate of Larry Ragon v. Eli Lilly & Company (Mary Ragon as Personal Representative of the Estate of Larry Ragon v. Eli Lilly & Company) is published on Counsel Stack Legal Research, covering Indiana Court of Appeals primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.

Bluebook
Mary Ragon as Personal Representative of the Estate of Larry Ragon v. Eli Lilly & Company, (Ind. Ct. App. 2014).

Opinion

Pursuant to Ind. Appellate Rule 65(D), this Memorandum Decision shall not be regarded as precedent or cited before any Sep 10 2014, 9:04 am court except for the purpose of establishing the defense of res judicata, collateral estoppel, or the law of the case.

ATTORNEYS FOR APPELLANT: ATTORNEYS FOR APPELLEE:

LINDA GEORGE BRIAN J. PAUL KATHLEEN A. FARINAS ANN H. STEWART TODD BARNES Ice Miller LLP ASHLEIGH M. RESETARITS Indianapolis, Indiana George & Farinas, LLP Indianapolis, Indiana

IN THE COURT OF APPEALS OF INDIANA

MARY RAGON AS PERSONAL ) REPRESENTATIVE OF THE ) ESTATE OF LARRY RAGON, ) ) Appellant-Plaintiff, ) ) vs. ) No. 93A02-1402-EX-80 ) ELI LILLY & COMPANY, ) ) Appellee-Defendant. )

APPEAL FROM THE FULL WORKER’S COMPENSATION BOARD OF INDIANA Linda Hamilton, Chairman Cause No. 0207812

September 10, 2014

MEMORANDUM DECISION – NOT FOR PUBLICATION

BAKER, Judge Mary Ragon (Mary), as personal representative of the Estate of Larry Ragon

(Larry), appeals the decision of the Indiana Worker’s Compensation Board (the Board)

finding that Larry had failed to meet his burden of proving, by a preponderance of the

evidence, that he suffers from the occupational disease asbestosis. Mary argues that the

board committed reversible error by failing to find that Larry was exposed to the hazards

of asbestos. She also contends that the Board erred when it found that Larry’s testimony

was not credible when appellee-defendant Eli Lilly (Lilly) presented no evidence to rebut

that testimony. Finding that the evidence in this case is disputed and that Mary’s

arguments constitute an impermissible request to reweigh the evidence, we affirm.

FACTS1

Larry was an employee at Lilly in varying capacities from 1965 to June 27, 1996.

During that time Larry worked as a trades helper in the carpenter shop from the mid-

1960s until 1969 or 1970. He then began working in the paint shop until 1972 or 1973;

he then worked in the pipe shop until 1974 or 1975; and he then worked in the sheet

metal shop until sometime in the early 1980s. From that time forward, Larry performed

work certifying laboratory equipment for Lilly, and he held that position until he left

Lilly in 1996. Larry held other jobs after leaving Lilly, including maintenance and

security work. He left the workforce in 2009.

1 We remind appellant’s attorney that, in accordance with Appellate Rule of Procedure 46(A)(6), parties are required to state the facts “in accordance with the standard of review appropriate to the judgment or order being appealed.” 2 Larry struggled with respiratory problems, including chronic bronchitis and

pneumonia. He was ultimately diagnosed with Chronic Obstructive Pulmonary Disease

(COPD) and pulmonary fibrosis, which is marked by a thickening and stiffening of tissue

in the lungs.

Larry believed that his exposure to asbestos during his employment with Lilly had

caused his respiratory disease and filed an Application for Adjustment of Claim on

December 15, 2010. Lilly denied Larry’s claim. Larry was then examined by Dr. David

Mares, and, on September 26, 2011, Larry filed an Amended Application for Adjustment

of Claim. At this time, Dr. Steven Smith, Lilly’s occupational and environmental disease

expert, examined Larry. Dr. Mares and Dr. Smith agreed that Larry is disabled, but

disagreed as to causation.

On March 26, 2013, Judge Diana Parsons of the Worker’s Compensation Board

held a single-member hearing on the matter. Larry was the only witness to testify in

person, as his counsel wanted Judge Parsons to be able to judge his credibility. In

addition to Larry’s testimony, Judge Parsons reviewed documentary evidence and the

depositions of both Larry’s and Lilly’s experts. Dr. Mares’s report found that Larry had

asbestosis to a reasonable degree of medical certainty. Additionally, Dr. Robert Daly, the

pulmonologist treating Larry at the time of the single-member hearing, testified that

Larry’s history was consistent with a diagnosis of asbestosis to a reasonable degree of

certainty. Dr. Daly’s opinion was based on the medical history provided to him by Larry

and Larry’s previous physicians.

3 Dr. Smith, Lilly’s expert, examined Larry’s medical records and interviewed Tom

Yoder, a former safety director at Lilly, who had knowledge about the work environment

Larry would have encountered during his time at Lilly. Dr. Smith found that “it is likely

that [Larry] was occupationally exposed to some level of respirable asbestos in

conjunction with his work within [Lilly] facilities . . . .” Tr. p. 171. However, he also

found that “[Larry] most certainly was not exposed to sufficient quantities of airborne

and respirable asbestos fibers within Lilly premises to have developed even very mild

Lilly employment-related asbestosis.” Id. at 171-72. Moreover, Dr. Smith found that

“[Larry] definitely does not even have pulmonary asbestosis.” Id. at 172.

After reviewing the evidence, Judge Parsons issued her order on April 6, 2013.

Among her Case Summary, Commentary, and Findings were the following:

5. There is a dispute as to whether the Plaintiff’s pulmonary fibrosis is idiopathic or was caused by the inhalation of airborne asbestos fibers while the Plaintiff worked for the Defendant. The Plaintiff has offered expert medical opinions from pulmonologists Dr. David Mares and Dr. Robert Daly to support his contention that his pulmonary fibrosis was caused by the inhalation of airborne asbestos fibers.

...

7. The Plaintiff was also evaluated, and treated, for his pulmonary condition by Dr. Daly. Dr. Daly initially diagnosed the Plaintiff with idiopathic pulmonary fibrosis. When he learned of the Plaintiff’s alleged asbestos exposure, he subsequently opined that it was unclear if the Plaintiff’s fibrosis was a mix of asbestosis and/or idiopathic changes. Dr. Daly then subsequently opined that the Plaintiff had asbestosis. Dr. Daly’s occupational history from the Plaintiff was that he was a pipefitter for the Defendant. This is an incomplete and inaccurate statement of the Plaintiff’s occupational history. Dr. Daly’s opinion was also informed by the “extensive occupational history documented by [Dr.] Mares” as referenced

4 by Dr. Daly in his November 23, 2011 report. Dr. Mares’ occupational history, as noted above, was incorrect.

8. The Defendant stipulates that, during the period of Plaintiff’s employment, many buildings owned by the Defendant has asbestos- containing materials installed in them. The Defendant disputes, however, that the Plaintiff suffers from asbestosis as a result of the inhalation of airborne asbestos fibers.

10. The Defendant contends that the Plaintiff’s pulmonary fibrosis is idiopathic and offers Dr. Smith’s report in support of this contention. Dr. Smith conducted an extensive record and professional literature review and issued a detailed report in which he concludes, in part, that the Plaintiff’s [alleged] asbestos exposure would not have been sufficient to have posed a risk for the development of asbestosis. In support of this opinion, Dr. Smith provided a detailed analysis of the specific tasks allegedly performed by the Plaintiff, their known propensity for airborne asbestos exposure, and the cumulative putative asbestos exposure likely to have resulted therefrom. He noted that, while there can be a release of respirable asbestos fibers during some of the industrial piping work the Plaintiff contends he performed, the level of release would have been minimal, extremely low, and incapable of causing asbestosis. Dr.

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