Lyon v. Wilson

443 P.2d 314, 201 Kan. 768, 1968 Kan. LEXIS 425
CourtSupreme Court of Kansas
DecidedJuly 13, 1968
Docket45,165
StatusPublished
Cited by35 cases

This text of 443 P.2d 314 (Lyon v. Wilson) is published on Counsel Stack Legal Research, covering Supreme Court of Kansas primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.

Bluebook
Lyon v. Wilson, 443 P.2d 314, 201 Kan. 768, 1968 Kan. LEXIS 425 (kan 1968).

Opinion

The opinion of the court was delivered by

O’Connor, J.:

This is a workmen’s compensation case where the employee, Lee Roy Lyon, died as the result of a coronary occlusion on September 12, 1962. The examiner’s denial of compensation to the claimant (widow, and guardian of the minor children) was approved by the director. On appeal the district court found that Lyon had sustained an injury by accident arising out of and in the course of his employment, and awarded compensation. The respondents (employer and insurance carrier) now appeal.

The principal question, as stated by the respondents, is whether or not there is substantial evidence to support the district court’s *769 finding that there was a causal connection between Lyon’s work and his heart attack.

Lyon was employed as a driller on a cable tool rig. On the morning of the day he died, Lyon arose at 5:30, ate breakfast, and left his home at 6:15. He arrived at the well site about 7:00 and began working. Lyon’s first task was to pull tubing, which required his running the brake and clutch on the rig. Following the pulling of the tubing, which took approximately two hours, Lyon assisted a member of his crew, Jacob Polzin, in making up a string of tools. Since this string of tools was needed in a hurry, the work was done by hand rather than by machinery, as was the customary practice. Although this was regular cable tool work, it was considered heavy work, and required the lifting, handling, and carrying of pieces of equipment, each varying in weight from 100 to 750 pounds. The entire operation took about an hour, during which period Lyon climbed up to the floor of the rig about six times. The work was completed by 10:00 a. m., the tools were put in the hole, and drilling was begun. Later, the hole was “baled,” requiring the use of a baler—two 150-pound sections of pipe—which was put on the line by hand. The baling and drilling continued until noon.

As Polzin and Lyon walked to the dog house for lunch, Lyon commented he had a pain in his chest and felt lightheaded. After arriving at the dog house, Lyon asked Polzin if he thought he, Lyon, was having a heart attack. Lyon then left the dog house, went back to the rig and laid his head on the floor, which was about four feet above the ground. His face was gray in color at that time, and he told Polzin he had to get to town as he was sick. Lyon was then taken to Hoisington and admitted to the hospital. Prior to the arrival of a doctor or the giving of medication, Lyon talked to an X-ray technician and told her he had had a sharp pain in his chest early that morning and had been working hard making up a string of tools. Dr. Robert Moore arrived, and during the ensuing examination Lyon also told the doctor he had been working hard and had been making up a string of tools. Lyon’s statement to Dr. Moore was verified by the claimant, who had gone to the hospital to be with her husband. An electrocardiogram was made and medical treatment administered, but Lyon died about an hour and a half after his admission to the hospital.

A postmortem examination of Lyon’s heart, which was performed by Dr. Richard J. Taylor, a pathologist in Wichita, showed the *770 lumen to be 75 percent occluded and at several points reduced to approximately 10 percent of normal size. The pathologist was of the opinion that a newly formed adherent thrombus which he found near an old thrombus caused the occlusion in the lumen resulting in Lyon’s death, and that this new thrombus was of probably less than four and certainly less than eight hours’ duration. The pathologist further found that Lyon had marked arteriosclerotic thickening of the vessels, and in his opinion, a coronary occlusion was inevitable because of the degree of coronary artery disease present.

The medical evidence concerning the relation of Lyon’s work to his coronary occlusion was sharply conflicting. None of the four doctors who testified had known or examined the workman during his lifetime. In fact, the record is void of any manifestations or symptoms of previous heart difficulty.

Dr. Renjamin Matassarin, who testified on behalf of the claimant, had not had the benefit of the pathologist’s report, but based upon a hypothetical question that Lyon had engaged in extremely heavy manual labor on the day he died, including making up a string of tools, the doctor was of the opinion the activties had an effect on the cardiovascular system and were a direct correlation to the progress of the disease and death. On cross-examination Dr. Matassarin steadfastly maintained his opinion as expressed, although he conceded that from statistical information and clinical experience a correlation between work and the formation of a fatal thrombus might be questionable.

Dr. Taylor, the pathologist, testified on behalf of the respondents. His testimony was apparently highly persuasive to the lower court, for in the journal entry of judgment the trial judge commented:

“The contention of the respondent and insurance carrier that the formation of a Thrombus in the lumen of a heart artery could not be caused by or have any relation to stress and exertion is refuted by . . . Dr. Taylor, the pathologist . . .”

The doctor testified that a person coming to him with shortness of breath, gray in color and ill at his stomach had symptoms of what might be a coronary occlusion; that any person with these symptoms should not be engaged in any kind of labor; that if he was truly having a coronary occlusion at that time, stress or continued physical activity would aggravate his condition. The doctor’s testimony continued as follows:

“Q. What causes—how does the exertion cause the occlusion?
“A. Well, I don’t know as I can say specifically how it causes the occlusion. *771 What it causes is an increased need of the myocardium of the heart for additional blood supply. Ordinarily, in a normal individual, this is accomplished by increase in heart rate, increase of blood pressure. This supplies more blood to the heart muscle. In a person with a very narrowed vessel due to an old thrombus or due to marked arteriosclerosis, the body is not able to meet this increased blood supply to the heart. As a result, the heart muscle will build up lactic acid, which is a breakdown product of its metabolism, and undergoes and ischemia, in other words, a deficiency of blood supply and is ordinarily associated with heart pain, also spasm of the coronary arteries. Due to the pain and the spasm and the narrowing of the vessels, certain idiocurrents are set up in the blood flow and sometimes these plaques are ruptured, bleed underneath them, and a new thrombus begins to form.
“Q. Doctor, if I understand you correctly, you said that stress, in your opinion, does, can bring on coronary occlusion.
“A. If I said it that way, I don’t really mean it that way. Let me restate it. Stress of any sort, physical or mental or emotional stress increases the heart’s need for oxygen. If these vessels are narrowed, then the heart doesn’t get adequate oxygen, the patients die without even any thrombus because of coronary insufficiency. I don’t know that anybody has demonstrated conclusively that the stress itself causes the thrombus.

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Cite This Page — Counsel Stack

Bluebook (online)
443 P.2d 314, 201 Kan. 768, 1968 Kan. LEXIS 425, Counsel Stack Legal Research, https://law.counselstack.com/opinion/lyon-v-wilson-kan-1968.