Kinney v. State Industrial Accident Commission

423 P.2d 186, 245 Or. 543, 1967 Ore. LEXIS 639
CourtOregon Supreme Court
DecidedFebruary 1, 1967
StatusPublished
Cited by10 cases

This text of 423 P.2d 186 (Kinney v. State Industrial Accident Commission) is published on Counsel Stack Legal Research, covering Oregon Supreme Court primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.

Bluebook
Kinney v. State Industrial Accident Commission, 423 P.2d 186, 245 Or. 543, 1967 Ore. LEXIS 639 (Or. 1967).

Opinion

LUSK, J.

This is an appeal by the defendant State Industrial Accident Commission (now the Workmen’s Compensation Board: OES 656.002 (3)) from a judgment of the circuit court on an appeal from the order of the Commission denying plaintiff’s claim for compensation. In a jury trial, after the plaintiff had rested and the defendant, without introducing any evidence, also rested, both sides moved for a directed verdict. The jury was thereupon discharged and the court found that the plaintiff had sustained a compensable injury and entered an order referring the case back to the Commission for the fixing of the plaintiff’s compensation.

The only substantial question for decision is whether the plaintiff suffered an “injury” within the meaning of that word as used in the Workmen’s Compensation Law.

Plaintiff at the time of the incident involved was employed as a maintenance man by a firm known as Plastics Northwest. He was 51 years of age. On June 2, 1963, an elevator in the building where plaintiff was working fell into the pit and a fellow employee who *545 was in the elevator was injured. Plaintiff undertook to assist him. He tried to move a safety gate weighing 150 to 175 pounds which was wedged in the elevator, but was unable to do so. He then went to the second floor of the building, got a ladder and put it up to the elevator spool that hangs on the ceiling with the purpose of respooling and raising the elevator. He climbed to the top of the ladder, but was forced to come down because he experienced shortness of breath, dizziness, and pressure on his chest. He was taken to Holladay Park Hospital. The hospital record shows the following diagnosis: “Acute anterior myocardial ischemia due to coronary artery disease — probable arteriosclerotic basis. Aortic and mitral stenosis due to rheumatic fever, childhood. Hypertensive cardiovascular disease.”

Plaintiff was discharged from the Holladay Park Hospital on June 14, 1963, but did not return to work.

On July 8, 1963, the plaintiff suffered an early morning onset of shortness of breath and pressure upon his chest. He was taken to the Veterans Administration Hospital in Portland and treated there for aortic stenosis. He remained in the hospital until August 13, 1963, and thereafter was treated as an outpatient until September 10, when he was readmitted and on October 22, 1963, it was determined that surgery was indicated. On February 10, 1965, open heart surgery was performed at the University of Oregon Medical School Hospital and the plaintiff was fitted with a Starr-Edwards stainless steel prosthetic valve, replacing his own aortic valve which had been narrowed as a result of aortic stenosis.

The only medical witness was Dr. Leonard W. Eitzmann, Professor of Medicine at the University of *546 Oregon Medical School and head of the cardiology section of the Veterans Administration Hospital in Portland. Dr. Eitzmann supervised treatment of the plaintiff after he entered the Veterans Hospital.

In explaining the anatomy and physiology of the heart Dr. Eitzmann testified that the left ventricle is “a pumping chamber over the rest of the body” and the “outlet of the left ventricle is into the aorta, the main blood vessel, which leads blood to all the rest of the body.”

The plaintiff had a heart condition, he testified, known as aortic stenosis, by which is meant a narrowing or shrinking of the aorta. The result “of aortic stenosis on the heart muscle is predominantly to increase the workload and the way the heart compensates for this is for the heart muscle to get thicker and stronger” and due to the narrowing of the aortic valve there is a “type of pressure difference which we call an aortic systolic type of contraction pressure grading” which enables a physician to make a certain diagnosis of the presence of stenosis. A patient may have this type of deformity for many years and have no symptoms. He may

“* * * have been completely active, maybe even been very active in terms of sports and the like, even though they have the condition. Somewhere along the line a patient then usually will develop some type of symptoms from this disease, however, the symptoms which we mentioned, and it is at the time that symptoms develop that we feel it is necessary to step in with a form of therapy, because the condition is really a serious one, once the symptoms have developed, leading very commonly to death within several years.”

The symptoms are often the same as those of severe coronary artery disease, that is, angina pectoris or *547 heart pain, shortness of breath, and fainting or dizziness. In aortic stenosis angina results because “the heart itself is now thickened and * * * the blood supply to the heart is no longer able to keep up with feeding the extra amount of muscle.” The witness thus explained the reason why stress produces symptoms in a patient having aortic stenosis:

“Well, when stress occurs, this puts an extra workload on the left ventricle to produce blood for the rest of the body which uses an increased amount of oxygen and requires extra oxygen to be transported by the vascular system. The heart itself has to supply this and hence when the heart is trying to put out this extra load, if it is unable to receive enough blood supply itself in order to take care of this extra work, this is the place where symptoms of angina occur.”

Eespecting the cause of the plaintiff’s symptoms he testified:

“My opinion is that the unusual stress that he underwent in trying to raise this elevator and the other tenseness of the situation, the extra work that he went through at that time, was the precipitating factor for his first symptoms associated with his aortic stenosis.”

He testified, in effect, that where there are no symptoms aortic stenosis is not disabling. When symptoms appear, it becomes disabling. In order to determine whether there was coronary artery disease he and other doctors examined all the electrocardiograms which were available and made other tests. The results were negative. It was admitted by counsel for the plaintiff on the trial that there was no myocardial infarction.

The statute in effect at the time this case arose provided that a workman who sustains “an accidental *548 injury * * * arising out of and in the course of his employment” is entitled to compensation as provided in other sections of the Act: former OES 656.152. The statute contains no definition of injury. The defendant contends that it means “a lesion or pathological change in a tissue or organ of the human body,” and that, since there is no evidence of such a change, there was no compensable injury. In the statutes of a few of the states injury is defined in accordance with the defendant’s contention, and there is some judicial authority for that view in states having no statutory definition of the word: Bohanan v. Schlozman Ford, Inc., 188 Kan 795, 366 P2d 28; Burns’s Case, 218 Mass 8, 105 NE 601, Ann Cas 1916A 787; Sullivan’s Case, 265 Mass 497, 164 NE 457, 62 ALR 1458.

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Bluebook (online)
423 P.2d 186, 245 Or. 543, 1967 Ore. LEXIS 639, Counsel Stack Legal Research, https://law.counselstack.com/opinion/kinney-v-state-industrial-accident-commission-or-1967.