Hayes v. Decker

822 A.2d 228, 263 Conn. 677, 2003 Conn. LEXIS 210
CourtSupreme Court of Connecticut
DecidedJune 3, 2003
DocketSC 16692
StatusPublished
Cited by21 cases

This text of 822 A.2d 228 (Hayes v. Decker) is published on Counsel Stack Legal Research, covering Supreme Court of Connecticut primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.

Bluebook
Hayes v. Decker, 822 A.2d 228, 263 Conn. 677, 2003 Conn. LEXIS 210 (Colo. 2003).

Opinions

Opinion

SULLIVAN, C. J.

The defendant, Mark J. Decker, a physician, appeals from the judgment of the Appellate Court reversing the trial court’s judgment in his favor and granting the plaintiff, Gilbert Hayes, a new trial. The sole question in this certified appeal is “[d]id the Appellate Court properly conclude that the plaintiffs expert’s proffered testimony regarding the effect of discontinuation of blood pressure medication should have been admitted into evidence?” Hayes v. Decker, 259 Conn. 928, 793 A.2d 253 (2002). We conclude that the Appellate Court’s conclusion was correct and, accordingly, affirm the judgment of the Appellate Court.

The opinion of the Appellate Court sets forth the following relevant facts and procedural history. “On May 12,1995, the plaintiff, who was fifty years old, went [679]*679to the defendant, an internist, for a physical examination. During the examination, he reported sexual dysfunction and multiple cardiac risk factors, including age greater than forty, high cholesterol, overweight, chest pain, smoking, and a family history of vascular disease and hypertension, for which he was taking Procardia XL as prescribed by his kidney physician. The defendant suggested that the plaintiff lose weight to control his blood pressure and that he stop taking Procardia for a trial period as a possible solution to his impotence problem.

“By the time of his next visit to the defendant’s office on June 2, 1995, the plaintiff had lost fifteen pounds and his systolic and diastolic blood pressure reading had dropped from 160 over 110 to 120 over 90. The defendant again suggested that the plaintiff stop taking Procardia as a possible cure for his impotence. The defendant did not consult with the plaintiffs kidney physician before making this recommendation and did not prescribe any substitute blood pressure medication, as he apparently believed that the plaintiffs blood pressure could be controlled adequately by weight reduction and exercise. Approximately one week later, the plaintiff stopped taking Procardia. Two weeks later, his blood pressure reading was 140 over 95.

“On July 9,1995, the plaintiff suffered a massive heart attack. During the attack, his diastolic pressure rose to 120. At the hospital, he underwent catheterization and primary angioplasty. In performing this procedure, physicians discovered that his left anterior descending artery was totally occluded and that there was significant stenosis of the right coronary artery. Two months later, the plaintiff underwent triple bypass surgery.

“In his complaint dated June 30, 1997, the plaintiff claimed that the defendant negligently failed to recognize and treat symptoms of cardiac ischemia and per[680]*680mitted him to discontinue the Procardia without substituting another blood pressure medication. The case was tried to a jury, which returned a verdict for the defendant. Thereafter, the court denied the plaintiffs motion to set aside the verdict and rendered judgment for the defendant.” Hayes v. Decker, 66 Conn. App. 293, 295-96, 784 A.2d 417 (2001).

“At trial, the defendant filed a motion to exclude proposed testimony by the plaintiffs expert witness, Richard Friedlander, pursuant to State v. Porter, 241 Conn. 57, 698 A.2d 739 (1997) (en banc), cert. denied, 523 U.S. 1058, 118 S. Ct. 1384, 140 L. Ed. 2d 645 (1998). Friedlander was a board certified physician in internal medicine and cardiology who had treated hundreds, perhaps thousands, of patients during his career, and who at one time directed cardiovascular clinical research for a pharmaceutical company. At the hearing on the motion, Friedlander stated his ‘belief and ‘opinion’ that, although the discontinuation of the plaintiffs blood pressure medication did not cause his heart attack, it did cause his blood pressure to rise and resulted in more tissue damage than otherwise would have occurred had he not stopped taking the medication. Friedlander based his opinion on the fact that numerous studies show, and it is generally accepted within the scientific community, that an increase in blood pressure results in an increase in the demand of heart muscle tissue for oxygen, and that increased blood pressure and oxygen demand during the acute phase of a heart attack result in increased tissue death. He also testified that studies have shown that Procardia, a well known blood pressure medication, lowers blood pressure in most individuals.

“Friedlander acknowledged, however, that he did not know of any research or completed study documenting a link between the discontinuance of blood pressure medication and an increase in the severity of a subse[681]*681quent heart attack. He also could not point to any scientific articles, studies or treatises concluding that specific increases in blood pressure result in specific amounts of heart muscle damage. He testified that such studies would be impossible to conduct because ‘you are comparing what is to what would have been’ in a single heart attack patient. Moreover, he did not attempt to quantify how much additional heart muscle damage the plaintiff might have suffered as a result of discontinuing his medication.

“The [trial] court ‘reluctantly’ ruled, on the basis of the standard articulated in Porter for the admissibility of scientific evidence, that because there was no study concluding that the withdrawal of Procardia will increase the severity of a heart attack, Friedlander’s proposed testimony was ‘speculative’ and hence inadmissible. In denying the plaintiffs subsequent motion to set aside the verdict, the [trial] court similarly stated that, in the absence of any evidence in the form of treatises or publications establishing that the withdrawal of Procardia increases the severity of a heart attack, it saw no reason to change its opinion.”1 Hayes v. Decker, supra, 66 Conn. App. 296-98.

[682]*682Following judgment for the defendant, the plaintiff appealed, claiming that the trial court improperly had granted the defendant’s motion to exclude scientific testimony because it had misapplied the standard for the admission of scientific testimony as set forth in State v. Porter, supra, 241 Conn. 57. The Appellate Court reversed the judgment of the trial court, concluding that “the [trial] court incorrectly applied the law as set forth in Porter. Friedlander’s testimony as to whether the discontinuation of the plaintiffs blood pressure medication increased the severity of his heart attack should have been considered under the standards for the admissibility of expert opinion testimony; his testimony on the well documented effect of Procardia in lowering blood pressure and the relationship between blood pressure and tissue damage during a heart attack should have been considered under the standard for the admissibility of scientific evidence under Porter, or simply admitted on a showing of relevance. The [trial] court never made this crucial distinction and, in applying the Porter analysis to Friedlander’s opinion rather than to the scientific evidence on which it was based, improperly excluded all of his proposed testimony on Procardia, blood pressure and the amount of tissue damage that may occur during a heart attack.” Hayes v. Decker, supra, 66 Conn.

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Bluebook (online)
822 A.2d 228, 263 Conn. 677, 2003 Conn. LEXIS 210, Counsel Stack Legal Research, https://law.counselstack.com/opinion/hayes-v-decker-conn-2003.