COLEMAN, JUSTICE, FOR THE COURT:
¶1. On May 13, 2013, a former construction worker, Robert Lee Rankin Sr., filed a complaint in Jefferson County Circuit Court against American Optical Corpora[1064]*1064tion (AO) alleging an injury of “lung disease and silica related conditions caused by exposure to respirable crystalline silica” while using defective respirators1 manufactured by AO. Following trial on February 17-24, 2015, the jury returned a total verdict of $14 million in favor of Rankin.2
¶2. According to the verdict form, the jury found by a preponderance of the evidence that (1) Rankin had a “silica-induced lung condition;” (2) Rankin did not know nor should he have known before May 13, 2010, that he had the “lung injury” that he alleged in the lawsuit; (3) Rankin used a respirator manufactured or sold by AO “while working in harmful concentrations of silica;” (4) there was a defect in the design of an AO product that proximately caused Rankin’s silica-induced lung condition; and (5) there was a defect in the warning of an AO product that proximately caused Rankin’s silica-induced lung condition.
¶3. The jury awarded $4 million in non-economic damages and $10 million in economic damages. The jury assigned 45% of fault to AO and 55% to “Other Named and Unnamed Defendants and/or Parties.”3 The trial court entered a total judgment against AO of $6.3 million, reflecting the jury’s apportionment of fault. AO filed a motion to amend the judgment and a motion for a judgment notwithstanding the verdict (JNOV) or, alternatively, for a new trial. The trial court granted AO’s motion to amend the judgment in part and amended the noneconomic damages award from $1.8 million to $1 million to comply with the statutory cap on noneconomic damages. However, the trial court denied AO’s motion for a JNOV or, alternatively, for a new trial.
¶4. AO appeals, raising the following seven issues, restated for clarity:
I. Whether Rankin’s claims are barred by the three-year statute of limitations.
II. Whether Rankin proved that the design of the AO respirators proximately caused his injury and that there was a feasible design alternative.
III. Whether AO is entitled to judgment as a matter of law on Rankin’s failure to warn claim.
IV. Whether Rankin proved that his use of an AO respirator proximately caused his injuries.
V. Whether Rankin proved a reasonable basis for the allegation that his alleged damages were caused by silica exposure.
VI. Whether AO was deprived of its right to a fair trial in an impartial forum.
VII. Whether the $14 million verdict was against the overwhelming weight of the evidence.
¶5. It is necessary for the Court to address only the first issue raised.
FACTUAL BACKGROUND
¶6. AO manufactured and sold respiratory protection products, including the model 1050 and 1010 respirators or masks, which were introduced in 1976 and 1985 respec[1065]*1065tively.4 The 1050 and 1010 were single use, disposable respirators designed to protect individuals against pneumoconiosis and fibrosis producing dusts such as silica. Both AO respirators were marketed for use in textile, mining, quarrying, grinding, and rock crushing operations, but not in abrasive blasting operations.
¶7. Rankin worked in the construction business for various employers and independently from approximately 1965 though 2001. Rankin testified that he jackham-mered and sandblasted during his career in the construction business.5 Rankin identified several different brands of respirators that he used throughout his career while he jackhammered and sandblasted. Two of the respirators that Rankin used while he jackhammered and sandblasted were AO’s 1050 and 1010. Rankin could not recall how often or where he wore any specific respirator that he identified. Rankin never saw any boxes, packaging, or paperwork that came with the respirators, nor did anyone teach him how to use the respirators.
¶8. Larry Brown, Rankin’s former coworker, testified that he worked with Rankin from 1980 to 1985. Brown testified that he and Rankin performed carpentry, sandblasted, and jackhammered during the entire five year period that they worked together. Brown testified that there was “a lot of dust created” when they sandblasted and jackhammered concrete.6 Although Brown’s testimony was inconsistent, Brown ultimately testified that Rankin wore an AO mask while jackhammering and sandblasting from 1980 through 1985.
¶9. Rankin’s “long and complicated medical history” was well documented at trial. Rankin first was diagnosed with silicosis, an occupational lung disease, on January 14, 2014, eight months after Rankin had filed his complaint in the present case. Rankin also suffered from breathing problems since 2000; had a heart attack in 2001; underwent open heart surgery; had multiple strokes; had blood clots in his legs; suffered pulmonary emboli; had seizure disorders; had Type II diabetes; underwent leg amputátion below the knee due to diabetes; had peripheral vascular disease; had high cholesterol; suffered from hypertension; had coronary artery disease; had congestive heart failure; had chronic obstructive pulmonary disease (COPD), a chronic lung disease; and suffered from pulmonary interstitial lung disease.
1Í10. Dr. Steven Haber, a physician board certified in internal and pulmonary medicine as well as a certified B-reader, testified on behalf of Rankin. A B-reader is a physician certified by the National Institute for Occupational Safety and Health (NIOSH) to read x-rays for dust diseases of the lung such as silicosis and asbestosis. Dr. Haber explained that silicosis is a type of pneumoconiosis or “dust disease of the lung” caused by inhaling ciystalline silica. Dr. Haber testified four criteria are required before a diagnosis of silicosis can be made, which are: (1) an x-ray showing scar tissue; (2) a history of exposure to silica; (8) a latency period; and (4) a clinical evaluation to determine if there was another reason for the scar tissue.
[1066]*1066¶11. Dr. Haber testified that respirable silica may be generated during construction activities such as jackhammering and sandblasting. He said that disrupting or breaking up materials through jackham-mering and sandblasting causes crystalline silica to become airborne in high concentrations. Dr. Haber testified that the inhalation of silica causes scar tissue or fibrosis to form in the interstitial or tissue part of the lung, which results in an incurable and progressive lung disease known as silicosis. .
¶12. On January 14, 2014, Dr. Haber personally interviewed and examined Rankin “regarding, possible occupational lung disease.” Dr. Haber took Rankin’s occupational history and reviewed Rankin’s medical records, including x-rays and CT scans, as well as deposition testimony of Rankin and his coworkers. Dr. Haber gave the following summary of Rankin’s occupational history based on his interview with Rankin:
[Rankin] was in the construction indus-. try from about 1966 until 2001 when he left work. He had significant occupational exposures to silica dust. The sources of that silica dust was from sandblasting, jackhammering, construction work and being around others who were doing that sort of .activity. He, over his like 36 years, of work, had done about ten years or so of sandblasting. He worked around other sandblasting or jackhammering or doing activities that were producing silica dust. And that he would experience sand- in the nose or the mouth while he’s doing some of these activities and over man years, primarily from sandblasting and concrete work starting in the 1960s,
¶13. On January 14, 2014, Dr. Haber diagnosed Rankin with chronic silicosis that was incurable and likely to progress. Dr. Haber testified that, although Rankin’s heart problems were not caused by silicosis, the heart problems were “exacerbated” by silicosis.7
¶14. Dr. Obie McNair, a board certified internal medicine physician and former board certified pulmonolgist, also testified for Rankin. Dr. McNair opined that Rankin’s x-rays revealed “findings consistent with silicosis.” Dr, McNair testified that Rankin’s OOPD was caused by simple silicosis, by exclusion, Dr. McNair based the conclusion on “articles [that] said that silic[a] exposure can also cause COPD.” Dr. McNair; explained that he “deduced” that Rankin’s COPD was “quite possibly related to his silica exposure.”
¶15. Dr. McNair testified that Rankin’s diabetes, stroke, heart attack, peripheral vascular disease, seizure disorder, hypertension, high cholesterol, and heart failure were not caused by silica exposure. Dr. McNair also testified that Rankin’s medications for diabetes and cholesterol were not related to silica exposure.8 Dr. McNair [1067]*1067was asked on cross examination whether Rankin’s medication isordil had anything to do with silica exposure. In response, Dr. McNair mentioned a peer reviewed journal article (Liu Article) containing a study of 40,000 Chinese workers who were exposed to silica. Over the objection of AO’s counsel, Dr. McNair testified that, according to the article, people who had low level exposure to silica were at an increased risk of dying from heart related complications. '
¶16. Dr. McNair testified that he did not know whether Rankin’s, silicosis would progress at all. Dr. McNair also testified that Rankin’s life expectancy would be shortened due to the number of his medical problems. Dr. McNair also opined that Rankin’s COPD and chronic silicosis contributed to or exacerbated Rankin’s right-sided heart disease. Dr. McNair also testified that Rankin’s left-sided heart disease is not attributable to COPD. When asked whether Rankin’s left-sided heart disease was attributable to silicosis, Dr. McNair again mentioned that, according to the Liu article, people with exposure to silica had an increased risk of ischemic heart disease. Dr. McNair was asked whether Cozaar, a medication for congestive heart failure, was related to silica. Dr. McNair explained that silica could be indirectly related to congestive..heart failure, according to the Liu article. Dr. McNair opined that damaged lungs generally affect the cardiovascular system.
¶17. At the close of Rankin’s case in chief, AO moved for a directed verdict, arguing that Rankin’s claims are barred by the statute of limitations because Rankin knew he had a lung injury before May 13, 2010. The trial court denied the motion and AO proceeded with its case in chief. AO called Dr. Malcom Taylor, a cardiologist who treated Rankin in 2013, to the stand. Dr. Taylor testified that Rankin had left-sided heart disease and the right side of his heart was normal. Although Dr. Taylor did not see any problem with Rankin’s lungs, he referred Rankin to a pulmo-nologist at the request of Rankin’s wife, Jean. Dr. Taylor testified that silicosis could lead to COPD, and silicosis could cause lung damage.
¶18. AO also called to the stand Dr. Demondes Haynes, a board certified pulmonary and critical care physician. Dr. Haynes opined that Rankin did not have silicosis or any occupational lung disease. Dr. Haynes opined that silicosis does not cause congestive heart failure or make congestive heart failure worse. Dr; Haynes testified that Rankin’s x-rays dated January 19, 2010, and April 3, 2013, did not indicate silicosis. Dr. Haynes opined that Rankin did not have COPD. Dr. Haynes also opined that Rankin did not have lung disease or interstitial lung disease.
STANDARD OF REVIEW
¶19. The- Court applies a de novo standard of review to statutes of limitation. Phillips 66 Co. v. Lofton, 94 So.3d 1051, 1059 (¶ 12) (Miss. 2012). Likewise, the Court- reviews a trial court’s decision to grant or deny a motion for a directed verdict de novo. Moreno v. TLSL, Inc., 187 So.3d 127, 129 (¶8) (Miss. 2016).
¶20. “A motion for a directed verdict challenges the legal sufficiency of the evidence; it asks if the plaintiff met the burden of going forward on the evidence.” Roop v. S. Pharm. Corp., 188 So.3d 1179, 1184 (¶ 15) (Miss. 2016). “Th[e] Court will consider the evidence in the light most favorable to the appellee, giving that party the benefit of all favorable inference that may be reasonably drawn from [1068]*1068the evidence.” Spotlite Skating Rink, Inc. v. Barnes ex rel. Barnes, 988 So.2d 364, 368 (¶ 10) (Miss. 2008). The Court will affirm the denial of a motion for a directed verdict where there is substantial evidence to support the verdict; but we will reverse if the evidence, as applied to the elements of a party’s case, is either so indisputable, or so deficient, that the necessity of a trier of fact has been obviated. Hyundai Motor America v. Applewhite, 53 So.3d 749, 752-53 (¶¶ 9) (Miss. 2011).
DISCUSSION
I. Whether Rankin’s claims are barred by the three-year statute of limitations.
¶21. AO argues that the'trial court erred by failing to grant its motion for a directed verdict because Rankin’s claims are barred by the three-year statute of limitations. AO contends that Rankin’s claims accrued when he discovered his lung injury. Specifically, AO argues Rankin’s claims accrued when he was diagnosed with COPD in November 2007 or, at the latest, in January 2010 when his x-ray revealed “pulmonary fibrotic pathology.” Rankin contends that the timeliness of his complaint was a factual issue that was properly resolved by the jury. The special verdict form posed the question, “Do you find by a preponderance of the evidence that [Rankin] knew or should have known before May 13, 2010, that he had the lung injury alleged in this lawsuit?” To this, the jury answered “No.” Rankin argues that “under the unique facts of this case, [he] invoked his right to file suit even though he had not yet received a full diagnosis-only a strong suspicion he was exhibiting signs of silicosis.”
¶22. Under Mississippi Code Section 15-1-49, a plaintiffs cause of action for a latent injury or disease accrues at the point at which he discovered, or by reasonable diligence should have discovered the injury. Section 15-1-49 provides in relevant part:
(1) All actions for which no other period of limitation is prescribed shall be commenced within three (3) years next after the cause of such action accrued, and not after.
(2) In actions for which no other period of limitation is prescribed and which involve latent injury or disease, the cause of action does not accrue until the plaintiff has discovered, or by reasonable diligence should have discovered, the injury.
Miss. Code Ann. § 15-1-49 (Rev. 2012).
¶ 23. Causes of action accrue upon discovery of the injury, not discovery of the injury and its cause. Ridgway Lane & Assocs., Inc. v. Watson, 189 So.3d 626, 629 (¶ 9) (Miss. 2016). Knowledge of the cause of the injury is irrelevant to the analysis; rather, the inquiry is when the plaintiff knew or should have known of an injury. Lincoln Elec. Co. v. McLemore, 54 So.3d 833, 838 (¶ 23) (Miss. 2010). In determining whether a plaintiff knew or reasonably should have known that he had an injury, the Court considers the actions taken by the plaintiff. Id. at 837-38 (¶¶ 22-23). For example, “seeking medical attention for side effects or symptoms” may confirm that the plaintiff knew he was injured. PPG Architectural Finishes, Inc. v. Lowery, 909 So.2d 47, 51 (¶ 16) (Miss. 2005).
¶24. The question of whether the statute of limitations is tolled by the discovery rule often turns on the factual determination of what the plaintiff knew and when. Peoples Bank of Biloxi v. McAdams, 171 So.3d 505, 509-10 (¶ 18) (Miss. 2015). The discovery of an injury is an issue of fact to be decided by a jury when there is a genuine dispute. Watson, 189 So.3d at 629 (¶ 9). Although occasionally, the question of whether the suit is barred [1069]*1069by the statute of limitations is a question of fact for the jury, the question may be taken.away from the jury if reasonable minds could not differ as to the conclusion, as with other putative fact questions. McAdams, 171 So.3d at 510 (¶ 9).
¶25. In the case sub judice, Rankin’s wife testified that Rankin had breathing problems dating back to 2000. She explained that Rankin suffered from shortness of breath and at times could “hardly get his breath.” On November 27, 2007, Rankin sought medical treatment at the emergency room of Natchez Regional Medical Center. He complained of shortness of breath, asthma, and other respiratory distress. Upon a physical examination, his breathing came in wheezes and rales, and he exhibited crackles in the lung bases. He was suffering from cardiomegaly (enlarged heart), had increased lung markings, and was coughing yellow sputum. He had a history of emphysema, congestive heart failure, and coronary artery disease, as well as a prior myocardial infarction (heart attack). He also had prior cardiac catheterization and bypass surgery in 2001. A prior stroke left him with right-sided weakness, and he also had a history of hypertension, diabetes, and hyperlipide-mia.
¶26. Notably, both Rankin and his wife discussed his medical history, examination, results, diagnosis, and treatment plan with Dr. Barry Tillman. Dr. Tillman, in turn, counseled Rankin and his family regarding lab/radiological results, diagnosis, and need for follow-up. Rankin previously had suffered from symptoms similar to those exhibited on November 27, 2007. According to Dr. Tillman’s report, Rankin was evaluated by the emergency room physicians and was diagnosed with exacerbation of chronic obstructive pulmonary disease, consistent with Dr. Tillman’s clinical impression. Rankin was admitted to the emergency room as a result. A follow-up examination on November 30, 2007, showed that the “[g]eneral appearance of [Rankin’s] chest is essentially unchanged compared to previous 11/27/07. Technique of the chest is somewhat different but the lung fields are essentially unchanged. Car-diomegaly persists.”
¶27. A close inspection of the medical records reveals that Rankin had knowledge of the injury to his lungs because he and his wife relayed his medical history to the medical staff of the Natchez Regional Medical Center on November 27, 2007. Dr. Tillman, in turn, counseled Rankin regarding his lab results, diagnosis, and treatment. Rankin certainly knew his own history of construction work, involving jackhammering and sandblasting, from approximately 1965-2001. Rankin argues that his claim did not begin to run until January 14, 2014—eight months after filing suit—when he was diagnosed with silicosis. The testimony of his own expert, Dr. Obie McNair, contravenes that argument. According to Dr. McNair, silicosis caused Rankin’s COPD.
¶28. At trial, Dr. McNair testified that Rankin’s COPD, a chronic lung disease, was caused by simple silicosis by exclusion. On February 4,' 2008, Rankin went to the ER due to a “sudden onset of acute shortness of breath that was complicated with profound diaphoresis.” He was diagnosed with acute shortness of breath, recent, pulmonary embolism of extremity, coronary artery disease, and atherosclerotic cardiovascular disease. On July 17, 2009, a radiology report noted that Rankin had a “low probability of pulmonary embolic pathology.” On January 19, 2010, Rankin underwent an x-ray at the Jefferson County Hospital, which revealed “pulmonary fi-brotic pathology” or lung scarring disease. At trial, Dr. Haber testified that silicosis causes lung fibrosis or scarring.
[1070]*1070¶29. On an unknown date, Rankin’s attorneys retained Dr. Haber to perform a B-reading of Rankin’s x-rays. On October 22, 2012, Dr. Haber performed a B-reading of a Rankin’s x-ray dated August 10, 2012. On October 23, 2012, Dr. Haber submitted a report to Rankin’s attorney stating that his findings were “consistent with silicosis, assuming an adequate exposure history, sufficient latency, and exclusion of other more probable causes.” Dr. Haber also stated that no diagnosis was made or implied and recommended clinical correlation.
¶30. On another unknown date, Rankin’s attorneys arranged for him to undergo a physical and clinical evaluation by Dr. Haber “regarding possible .occupational lung disease.” On January 14, 2014, Dr, Haber performed an examination of Rankin and completed a report of his findings and diagnosis. The same day, which was eight months after Rankin had. filed his complaint against AO, Dr. Haber diagnosed Rankin with chronic .simple silicosis.
,¶31. Rankin filed his complaint against AO on May 13, 2013. Rankin claimed that his injury was “lung disease and silica related conditions, caused by exposure to respirable crystalline silica.” Rankin also alleged that his “lungs and body were injured” and he had “premature disability” as a result of silica inhalation. Rankin’s complaint indicates that he already had been diagnosed with silicosis at the time he filed his complaint. Rankin’s complaint contained a section “DISCOVERY RULE,” in which he alleged that he “suffers from an occupational illness which has a latency period,” and his “occupational illness did not distinctly manifest itself until [he] was diagnosed ivith silicosis.”9 However, Rankin’s position ultimately was that he was first diagnosed with silicosis on January 14, 2014, eight months after he filed his complaint,
¶32. At trial, Rankin maintained the position that he was first diagnosed with silicosis on January 14, 2014, by his retained expert pulmonologist, Dr. Haber. AO did not. dispute the diagnosis date of January 14, 2014; AO’s position at trial was that Rankin did not have silicosis at all. There is no dispute [Rankin] has never been diagnosed with silicosis- or any other occupational lung injury -by any of his treating physicians.10
¶33. At trial, in response to AO’s motion for a directed verdict, Rankin’s counsel argued that Rankin had filed within the statute of limitations because “Mr. Rankin was not diagnosed until—J mean, was not diagnosed with silicosis until three years prior to him filing a suit.” Rankin argued in its written response to AO’s post trial motion: “In cases involving latent injury such as silicosis, the claim only arises once the Plaintiff is diagnosed with the disease.”
¶34. A summary of the foregoing pertinent dates to the statute of limitations issue is provided by the Court:
1Í35. AO argues that McLemore is directly on point with the facts of the pres[1071]*1071ent case. On November 14, 2005, a welder, McLemore, filed a products liability suit against manufacturers of welding rods, claiming that his exposure to harmful welding fumes from the rods resulted in “serious neurological injuries” and his eventual diagnosis of manganism, a neurological disease caused by high exposure to manganese. McLemore, 54 So.3d at 833-35 (¶¶ 1-2, 9). The manufacturers argued that McLemore’s claims were time barred because he knew he had an injury on September 3, 2002, when a neurologist had diagnosed him with Parkinsonism and had informed him that his condition may have been related to his occupation as a welder. Id. at 835 (¶ 12). McLemore argued that his cause of action did not accrue until October 2005, when he was specifically diagnosed with manganism. Id, at 835 (¶ 13). McLemore argued that he did riot know that the welding fumes had caused his damages until the manganism diagnosis. Id. at 835 (¶ 13). At trial,- the physician who had diagnosed McLemore with man-ganism testified that manganism was a syndrome with features of atypical Parkin-sonism. Id. at 834 (¶ 8).
¶36. The Court held that McLemore’s cause of action was time barred because he knew of his injury in September 2002 when he was “informed of the correlation between his symptoms and welding.” Id. at 837, 839 (¶¶ 23-31). The Court held that, while notice of the causal relationship is irrelevant to the accrual of a cause of action, it showed McLemore’s knowledge of his injury at that time. Id. at 837 (¶ 23). The Court determined that the following “events and actions” taken by McLemore showed that he knew or should have known that he had an injury no later than September 2002: he had difficulty using his left hand in December 2001; he was informed that his Parkinsonism might have been related to his welding work; he sought legal advice and he filed a complaint in 2004 prior to his manganism diagnosis alleging “serious neurological injury” from exposure to manganese products. Id. at 835, 838 (¶¶ 9, 24).
¶37. AO also relies on the district court’s decision in Langston v. Pangborn Corp., 2014 WL 347718 (S.D. Miss. Jan. 31, 2014) (not reported) in support of its argument. On May 14, 2012, Langston filed a complaint, which seems to be strikingly similar to Rankin’s complaint in the présent case, against personal respiratory equipinent manufacturers, including AO. Id. at *1. Like Rankin, Langston alleged that he suffered from “lung disease and silica related conditions” as a result of béing exposed to crystalline silica while working as a sandblaster. Id. The defendants argued that Langston had knowledge of his “lung injuries” before May 14, 2009, three years prior to the filing of his complaint. Id. at * 2. Langston argued that his claim did not accrue until he was diagnosed with silicosis on November 7, 2012, which was six months after he filed his complaint. Id. at **4-5. The district court addressed the issue of the post-complaint silicosis diagnosis:
Without overlooking the obvious, the Court notes that Langston’s November 7, 2012, silicosis diagnosis occurred almost six (6) months after he filed suit on May 14, 2012. The diagnosis was made by Dr. Steven Haber, Langston’s retained medical expert. No medical record showing a diagnosis of silicosis from any of Langston’s treating physicians has been presented to the Court. Clearly then, the subject diagnosis is immaterial to the issue of when the claims alleged in the Complaint accrued for statute of limitations purposes and fails to give rise to a genuine dispute for trial. Any contrary argument from Langston is “disingenuous and without merit.” Powe v. Byrd, 892 So.2d 223, 228 (¶ 17) (Miss. [1072]*10722004) (rejecting the plaintiffs argument that the statute of limitations did not start to run until December 1, 2000, when her complaint was filed on August 3, 2000). Further, the accrual rule cited by the Mississippi Supreme Court in Morgan, Kinsey, and Kelly is inapplicable here since the plaintiffs in those actions filed suit subsequent to each respective silicosis diagnosis. See [Empire Abrasive Equip. Corp. v.] Morgan, 87 So.3d [455,] 462 (¶25) [(Miss. 2012)]; Kinsey [v. Pangborn Corp.], 78 So.3d [301,] 307 (¶ 18) [ (Miss. 2011) ]; [Clark Sand Co. v.] Kelly, 60 So.3d [149,] 162 (¶ 46) [ (Miss. 2011) ].
Langston, 2014 WL 347718, at *5.
¶38. The key distinguishable fact from the present case, however, was that Lang-ston testified that in 2007 he had researched silicosis and learned that it is caused by the inhalation of silica dust after he had found out he had “lung trouble.” Id. at *3. The district court held that “the extensive and consistent medical treatment sought by Langston for his breathing problems confirms that he either knew or should have known of his lung injuries prior to 2009.” Id. at *6. And “the accrual of Langston’s cause of action was not dependent upon an expert witness placing the label of silicosis on his injury.” Id. The district court concluded that Langston’s claims were time barred under Section 15-1^49 because the record established that Langston knew or should have known of his “lung disease and silica related conditions,” “injury to his lungs and body,” and “premature disability” more than three years prior to the filing of his complaint. Id.
¶39. In Phillips 66 Co. v. Lofton, 94 So.3d 1051, 1056 (¶ 1) (Miss. 2012), Lofton, a former oil rig worker, filed a complaint on May 19, 2004, alleging that he suffered from asbestosis11 as a result of exposure to a defendant’s product. Lofton began experiencing shortness of breath in 1995, and chest x-rays from 1995 and 1996 revealed pulmonary fibrosis. Id. at 1057 (¶ 5). Lofton first sought treatment for his pulmonary symptoms in September 2003 with a pulmonologist, who diagnosed Lofton with pulmonary fibrosis. Id.
¶40. In 2004, the same pulmonologist noted that Lofton likely was suffering from pneumoconiosis, also known as asbestosis. Id. Lofton was diagnosed conclusively with asbestosis in 2010, several years after he had filed his complaint. Id. The defendant argued that the statute of limitations began running in 1995, or at the latest 1996, when physicians discovered pulmonary fibrosis in Lofton’s lungs. Id. at 1059 (¶ 14). However, Lofton argued that “he could not have reasonably known of his lung injury until he sought treatment from [a pulmonologist] and was diagnosed with pulmonary fibrosis, or scarring of the lungs in September 2003.” Id.
¶41. The Court sated: “[According to Lofton’s testimony, he was seeking medical treatment for health concerns unrelated to his lungs in 1995 and 1996 and was not made aware of the fibrosis until [a pulmonolgist] diagnosed him in 2003.” Id. at 1059 (¶ 16). “Accordingly, Lofton could not reasonably have known about his injury until he sought treatment in September 2003 for symptoms associated with his asbestosis and was diagnosed with pulmonary fibrosis.” Id.
¶42. Because the discovery of the injury was a disputed issue of fact, the trial court submitted the following question of discov[1073]*1073ery to the jury: If you determine that Mr. Lofton knew or by reasonable diligence should have known of his injury before May, 2001, then you must return a verdict for the defendant. Id, at 1059-1060 (¶ 17). The Court held that the issue of whether the statute of limitations barred Lofton’s claim was a disputed issue of fact that was resolved by the jury in favor of Lofton. Id.
¶43. The record does not reveal when Rankin sought legal advice related to his lung injury. However, it is undisputed that Rankin began experiencing breathing problems in 2000. It also is undisputed that Rankin sought medical treatment for shortness of breath and had been diagnosed with exacerbation of COPD in 2007.
¶44. The district court in Bryant v. 3M Co., 2014 WL 2761553, *3 (S.D. Miss. June 18, 2014) (not reported), addressed a set of facts nearly identical to the present case under Mississippi law. On April 9, 2013, Bryant, a sandblaster for several years in the 1970s, filed a products liability complaint claiming that he had developed “lung disease and silica related conditions” because he had used defective personal respiratory equipment from certain manufacturers. Id. at *1. Bryant claimed in his discovery responses that he suffered from “shortness of breath, silicosis, and any condition caused by his exposure to respirable silica.” Id. at *2. The issue before the district court was whether Bryan knew or reasonably should have known of his alleged injuries before April 9, 2010. Id. at *2.
¶45. Bryant experienced both wheezing and shortness of breath as early as 1992; was diagnosed with both emphysema and severe COPD as early as 2000. In 2005, a radiologist identified markings on Bryant’s lungs that “could be chronic fibrotic type changes,” but made no definitive diagnosis. He was referred to a pulmonologist in 2007. Id. The Court stated: “First, [Bryant] did not know about the ‘B reading’ until February 2014-after he had already filed suit. Similarly, [Dr.] Haber first diagnosed [Bryant] with silicosis on January 14, 2014-after [he] had already filed suit.”12 The district court stated that, as a result, neither Dr. Haber’s B-reading nor his diagnosis report could have provided Bryant with the knowledge necessary to file suit. Id. at *4.
¶46. In applying the Court’s holdings in Lofton and McLemore, the district court held that there was a genuine issue of material fact regarding when a plaintiff knew or reasonably should have known of his alleged “silicosis/pulmonary fibrosis.” Id. at *4. However, the district court granted the defendant’s motion for summary judgment in part with respect to Bryant’s claims for “COPD, emphysema, [1074]*1074shortness of breath, wheezing, and any other injury or medical condition diagnosed before April 9, ■ 2010.” Id.
¶47. The Court stated in Lofton that the plaintiff could not reasonably have known about his injury until he sought treatment for symptoms associated with his alleged injury and was diagnosed. Lofton, 94 So.3d at 1059 (¶ 16). The Court has held that “absolute certainty nor an expert opinion vest the right to a cause of action under [Mississippi’s] products liability statute.” Lowery, 909 So.2d at 52 (¶18). The post-complaint diagnosis of the occupational injury, silicosis, is irrelevant to the analysis, because the Court has emphasized that “the cause of action accruefe] upon discovery, of the,injury, not discovery of the. injury and its cause.” Angle v. Koppers, Inc., 42 So.3d 1, 5 (¶ 9) (Miss. 2010).
¶48. Rankin’s complaint alleged that his injury was “lung disease and silica related conditions, caused by exposure to respira-ble crystalline silica.”-Rankin also alleged that his “lungs and body were injured” and he had “premature disability” as a result of silica inhalation. Reasonable minds could not have differed in answering the question on the special verdict form: “Do you find by a preponderance of the evidence that [Rankin] knew or should have known before May 13, 2010, that he had the' lung injury alleged in this lawsuit?” It is undisputed that Rankin was aware of and sought treatment for lung disease, COPD, in 2007. Moreover, Rankin’s experts opined that Rankin’s myriad of remaining medical conditions, of which he was aware and for which he sought treatment before May 13,2010, were related “in part” or “exacerbated” by silica exposure. Accordingly, we hold that the trial court erred by failing to grant AO’s motion for a directed verdict because Rankin’s claims are time barred.
CONCLUSION
¶49, Because Rankin’s claims are time barred, we reverse the jury verdict, vacate the judgment in favor of Rankin, and render a judgment as a matter of law in favor of AO.
¶50. REVERSED AND RENDERED.
WALLER, C. J., DICKINSON AND RANDOLPH, P.JJ., MAXWELL AND BEAM, JJ.,-CONCUR. CHAMBERLIN, J.,-CONCURS IN PART AND IN RESULT WITHOUT SEPARATE WRITTEN OPINION. KITCHENS, J., DISSENTS WITH SEPARATE WRITTEN OPINION JOINED BY KING, J.,