United States Steel Mining Co. v. Workers' Compensation Appeal Board

859 A.2d 877, 2004 Pa. Commw. LEXIS 724
CourtCommonwealth Court of Pennsylvania
DecidedOctober 7, 2004
StatusPublished
Cited by7 cases

This text of 859 A.2d 877 (United States Steel Mining Co. v. Workers' Compensation Appeal Board) is published on Counsel Stack Legal Research, covering Commonwealth Court of Pennsylvania primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.

Bluebook
United States Steel Mining Co. v. Workers' Compensation Appeal Board, 859 A.2d 877, 2004 Pa. Commw. LEXIS 724 (Pa. Ct. App. 2004).

Opinion

OPINION BY

Senior Judge MIRARCHI.

United States Steel Mining Company, LLC (Employer) petitions this Court for review of an order of the Workers’ Compensation Appeal Board (Board) affirming a decision of a workers’ compensation judge (WCJ) that granted the fatal claim *879 petition of Nancy Sullivan (Claimant) for the work-related death of her husband Thomas Sullivan, Jr. (Decedent). We affirm.

In a 1985 workers’ compensation decision, it was determined that Decedent, then living, established that he became partially disabled as a result of contracting an occupational disease, namely anthraco-silicosis, while in the employ of Employer. This decision further established that Decedent was employed as a coal miner in Pennsylvania from 1946 to 1958 and from 1965 to 1983. He was employed by Employer from 1965 until 1983, and became partially disabled from his occupational disease in 1984. During his employment as a coal miner, Decedent was exposed to the hazards of coal and silica dust, and his occupational disease resulted from his cumulative exposure to these hazards. Decedent died in 2000, and at Claimant’s request, an autopsy was performed on Decedent, resulting in, among other things, a series of slides of Decedent’s lung tissue.

At the hearing on her fatal claim petition, Claimant presented the deposition testimony of Cyril H. Wecht, M.D. and Curtis S. Goldblatt, M.D. Dr. Wecht, a board-certified anatomic, clinical, and forensic pathologist, testified that he reviewed Decedent’s medical records, his occupational history, the autopsy report, and the slides made from tissue taken from the cadaver. Dr. Wecht concluded that Decedent suffered from a cancerous tumor with multiple metastases in the liver, lung, lymph nodes, omentum, and mesentery. He also suffered from a tumor-related obstruction of the common bowl duct and an inflammation of the abdominal cavity. Further, the Decedent had an enlarged heart with hardening of the arteries and the aorta. Additionally, he suffered from anthracosilicosis or coal workers’ pneumo-coniosis, and severe bilateral fibroscleritis. Dr. Wecht also testified that Decedent suffered from a heart and lung condition known as cor pulmonale.

Dr. Wecht opined that Decedent’s principal cause of death was his widespread cancer, but that Decedent’s coal workers’ pneumoconiosis, as a secondary disease process, was a substantial contributing factor to his death. He explained that the 50% increase in the thickness of the right side of Decedent’s heart, compared with a more modest increase in the thickness of the left side of the heart, indicated that these changes were caused by Decedent’s lung disease, not by his hypertension or arteriosclerosis. He further described Decedent’s lung disease as an additional burden upon his cardiovascular problems, one that “made the physiological burden for the cardiopulmonary cycle a greater one for the body to sustain.” Deposition of Dr. Wecht, p. 24. For these reasons, Dr. Wecht opined that Decedent’s pneumoconi-osis was a significant contributing cause of Decedent’s death.

On cross-examination, Dr. Wecht acknowledged that Decedent had a history of heavy cigarette smoking, the effects of which on their own could develop chronic obstructive pulmonary disease. Dr. Wecht noted, however, that cigarette smoking could not cause Decedent’s pulmonary fibrosis and the other conditions associated with the deposit of silicate crystals in his tissue.

Dr. Goldblatt, who is board-certified in the specialty of anatomic and clinical pathology, testified that he reviewed the autopsy slides of Decedent’s tissue, the various relevant medical records and reports, and Decedent’s occupational history. Dr. Goldblatt observed on the autopsy slides macules of coal workers’ pneumoconiosis ranging in size from less than .1 centimeter to ,8 centimeters within the lymph nodes. Dr. Goldblatt testified that from *880 his review of the slides of Decedent’s lung tissue, he was able to diagnose Decedent as suffering from simple coal workers’ pneumoconiosis, severe pulmonary emphysema, and metastatic endocarcinoma. Dr. Goldblatt further testified that the medical records and autopsy report, as well as the autopsy material, indicated that Decedent also suffered from widespread metastatic colon cancer, severe coronary artery disease, myocardial fibrosis, and an enlarged heart with thickening of the left and right ventricles. Because the right ventricle was markedly thicker than the left ventricle, Dr. Goldblatt opined that Decedent also suffered from cor pulmonale.

Dr. Goldblatt opined that Decedent’s death was caused by cardiac failure precipitated by the numerous disease processes that were occurring within him. Dr. Gold-blatt further opined that coal workers’ pneumoconiosis, as a major component of Decedent’s lung disease, was a significant contributing factor in Decedent’s death. Dr. Goldblatt described Decedent’s lung disease as simple coal workers’ pneumoco-niosis and severe pulmonary emphysema, which was in part caused by the simple coal workers’ pneumoconiosis. The lung disease decreased the oxygen supply to the heart, and the right ventricular hypertrophy, also related to Decedent’s coal workers’ pneumoconiosis, caused a demand for additional oxygen to serve the larger muscle mass. Thus, according to Dr. Gold-blatt, these conditions, both related to Decedent’s coal workers’ pneumoconiosis, caused the heart to fail.

Dr. Goldblatt described Decedent’s coal workers’ pneumoconiosis as a secondary cause of death, which on its own would not have caused his death. In combination with Decedent’s other disease processes, however, the coal workers’ pneumoconiosis had a significant role to play in Decedent’s demise. Dr. Goldblatt was aware of Decedent’s history of smoking.

Employer presented the deposition testimony of Stephen T. Bush, M.D. and Everett F. Oesterling, Jr., M.D. Dr. Bush, who is board-certified in the specialty of anatomic and clinical pathology, testified that he reviewed the autopsy slides of Decedent’s tissue, various medical records and reports, and Decedent’s occupational history. Dr. Bush opined that the autopsy slides did not reveal any evidence of coal workers’ pneumoconiosis, and that the black pigment evident, absent a scarring, was not an indicator of coal workers’ pneu-moconiosis but of metastatic carcinoma. He further opined that Decedent’s lung disease consisted only of a mild-to-moderate centrilobar emphysema, attributable to his cigarette smoking but not to coal dust. Dr. Bush also opined that Decedent’s cause of death was his metastatic colon cancer. Dr. Bush did not believe that Decedent’s heart disease contributed to his death; nor did he believe that coal workers’ pneumoconiosis was a factor either. In fact, he denied that Decedent ever suffered from coal workers’ pneumoconiosis. Dr. Bush further testified that he did not believe that Decedent suffered from cor pulmonale, and that the bi-ventricular hypertrophy was caused by chronic hypertension, not lung disease.

Dr. Oesterling, who is board-certified in the specialty of anatomic and clinical pathology and nuclear medicine, testified that he reviewed the autopsy slides of Decedent’s tissue, various medical records and reports, and Decedent’s occupational history. Dr. Oesterling noted that the black pigment found around the pulmonary vessels did contain birefringement silica crystals, or coal mine dust.

Free access — add to your briefcase to read the full text and ask questions with AI

Related

G. DeBellis v. WCAB (Dermatology, LTD)
Commonwealth Court of Pennsylvania, 2019
PA Department of Environmental Resources v. WCAB (Vicinelly)
Commonwealth Court of Pennsylvania, 2018
T. Hughes v. WCAB (Pep Boys)
Commonwealth Court of Pennsylvania, 2018
M. Edwards v. WCAB (DPW)
Commonwealth Court of Pennsylvania, 2016
Glaze v. Workers' Compensation Appeal Board
41 A.3d 190 (Commonwealth Court of Pennsylvania, 2012)
O'Neill v. Workers' Compensation Appeal Board
29 A.3d 50 (Commonwealth Court of Pennsylvania, 2011)
City of Pittsburgh v. Workers' Compensation Appeal Board
11 A.3d 1071 (Commonwealth Court of Pennsylvania, 2011)

Cite This Page — Counsel Stack

Bluebook (online)
859 A.2d 877, 2004 Pa. Commw. LEXIS 724, Counsel Stack Legal Research, https://law.counselstack.com/opinion/united-states-steel-mining-co-v-workers-compensation-appeal-board-pacommwct-2004.