State v. Albert Lynn Norton

CourtCourt of Criminal Appeals of Tennessee
DecidedJuly 20, 1999
Docket03C01-9707-CR-00270
StatusPublished

This text of State v. Albert Lynn Norton (State v. Albert Lynn Norton) is published on Counsel Stack Legal Research, covering Court of Criminal Appeals of Tennessee primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.

Bluebook
State v. Albert Lynn Norton, (Tenn. Ct. App. 1999).

Opinion

IN THE COURT OF CRIMINAL APPEALS OF TENNESSEE FILED AT KNOXVILLE July 20, 1999

Cecil Crowson, Jr. MAY 1999 SESSION Appellate C ourt Clerk

STATE OF TENNESSEE, ) ) Appellee, ) C.C.A. No. 03C01-9707-CR-00270 ) vs. ) Blount County ) ALBERT L. NORTON, ) Hon. D. Kelly Thomas, Jr., Judge ) Appellant. ) (DUI 2nd Offense) )

FOR THE APPELLANT: FOR THE APPELLEE:

GEORGE H. WATERS PAUL G. SUMMERS Asst. Public Defender Attorney General & Reporter 419 High Street Maryville, TN 37804 ERIK W. DAAB Assistant Attorney General Criminal Justice Division 425 Fifth Avenue North Nashville, TN 37243

PHILIP MORTON Asst. District Attorney General Blount County Courthouse 363 Court Street Maryville, TN 37804

OPINION FILED: _____________

AFFIRMED

JAMES CURWOOD WITT, JR., JUDGE OPINION

The defendant, Albert L. Norton, appeals his Blount County Circuit

Court jury conviction of second-offense DUI, a class A misdemeanor. He does not

challenge his jail sentence of eleven months, 29 days at 90 percent, to be probated

after serving 90 days, a $2,500 fine, and a two-year license revocation. Rather, he

challenges his conviction by raising the following issues:

1. The evidence was insufficient to support his

conviction.

2. The trial court erred by allowing evidence of the

defendant’s blood test result despite the failure to establish a

proper chain of custody of the blood sample.

3. The trial court erred in allowing evidence of drugs

being present in the defendant’s blood sample.

4. The trial court erred by admitting testimony that the

defendant failed to pass a horizontal gaze nystagmus field

sobriety test.

After a review of the record, the briefs of the parties, and the applicable law, we

affirm the judgment of the trial court.

Sergeant Mark Taylor of the Maryville Police Department arrested the

defendant for DUI on March 7, 1995, after watching the defendant swerve his car

across the street centerline on two or three occasions and make a wide left turn

during which he appeared to strike a curb. Taylor stopped the defendant, who had

blood-shot eyes, smelled of alcohol, fumbled with his wallet when trying to extract

his drivers license, and staggered upon getting out of the car. The defendant told

Taylor he had a couple of beers earlier in the day and that he was on three types

of medication, including a pain medicine and a muscle relaxant. Taylor

administered three field sobriety tests -- the horizontal gaze nystagmus (HGN) test,

the one-leg stand, and the walk and turn test. After explaining that the HGN test

2 involved an assessment of the effects of intoxicants on the muscles in the eyes,

Taylor testified that the defendant tested “positive” on all six “clues” for which the

testing officer looks. On the one-leg stand, the defendant dropped his foot a few

times while counting and finally put his foot down and said he could not finish the

test. During the walk and turn test, he stepped off the line twice, used his arms to

keep his balance, missed connecting heel to toe three times, and incorrectly

executed the turn.

Michael J. Lyttle, a forensic scientist with the Tennessee Bureau of

Investigation (TBI), analyzed the defendant’s blood sample for the presence of

drugs. He found the presence of 1.1 micrograms per milliliter of carisoprodol, a

muscle relaxant, and testified that the therapeutic range 1 for carisoprodol is ten to

40 micrograms per milliliter. He found the presence of 9.2 micrograms per milliliter

of meprobamate, which is a metabolite of carisoprodol. The therapeutic range for

meprobamate is 3.0 to 26 micrograms per milliliter. He found the presence of

dihydrocodeinone, a narcotic analgesic, for which the therapeutic range is .002 to

.024 micrograms per milliliter. The concentration of dihydrocodeinone was below

the level of .1 micrograms per milliliter, and by policy, the TBI lab does not

“quantitate” concentrations below this level. It simply refers to the result as being

“less than” .1 micrograms per milliliter. Nevertheless, Lyttle assessed the

dihydrocodeinone level in order to determine if it exceeded the .1 level and found

the level to be .05, which is less than .1 but about twice .024, the upper limit of the

therapeutic range. Lyttle testified that all of these drugs are depressants that in

general have a sedative effect.2

1 Lyttle testified that “therapeutic range” refers to “the range that you would expect to see when that drug is having its prescribed effect on the body.” 2 Although not mentioned by Lyttle in his testimony, the TBI lab report showed the following additional substances were present in the blood sample: cocaine, “less than .1 UG/ML”; cocaethylene, “less than .1 UG/ML”; “benzoylecgonine (cocaine metabolite) 1456 NG/ML.” At a jury-out hearing, the trial court excluded from Lyttle’s proposed testimony evidence of the cocaine and cocaine-related substances.

3 Jerry Main, another forensic specialist with the TBI, testified that he

conducted the analysis of the defendant’s blood sample for the purpose of

ascertaining the presence of alcohol. Specifically, he found an alcohol

concentration of .006 percent. The TBI considers results of .01 percent or less to

be “negative.”

At a jury-out hearing to determine the nature of her expertise and the

scope of her testimony, Jean Ezell, the director of the pharmacy at Blount Memorial

Hospital, testified that drug level data could not be used to determine the actual

effect that a given drug level would have on a specific individual, but that she could

opine generally as to the effects of drug dosages. She reviewed the TBI drug

screen report and opined that the meprobamate and dihydrocodeinone were at

levels significant enough to cause some impairment, especially when one considers

the likelihood of an “additive” or “synergistic” effect of combining the two drugs.

Before the jury, Ezell confirmed that the three substances mentioned by Lyttle were

depressants and that if she were filling prescriptions for these prescription-only

drugs, she would affix labels warning the consumer of the effects of sedation and

dizziness. The dihydrocodeinone was present in the defendant’s blood sample at

a level double the “normal peak.” The carisoprodol was present at a level below the

therapeutic range, but Ezell explained that it metabolizes into meprobamate and

that, when drugs metabolize into other active drugs such as meprobamate, the

metabolite itself may have more effect than the “original” drug. Furthermore, she

testified that the TBI report revealed the presence of cocaine,3 a central nervous

system stimulant, in the defendant’s blood sample at a level less than .1

micrograms per milliliter. She explained generally the increased potency that could

be expected because of the additive and synergistic effects of combining the drugs

found in the defendant’s blood, but she stressed that she could not opine as to the

3 Based upon Ezell’s knowledge of the effects of cocaine which was revealed through her proffered testimony, the trial court allowed Ezell to testify before the jury about the cocaine and cocaine-related substances mentioned in the TBI lab report.

4 specific effect of any of these drugs on the defendant. Because there are

“extensions of the effect of the drug, . . . it can vary from patient to patient. So, I

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