State of Arizona v. John Robert Esser

Court of Appeals of Arizona·Decided May 23, 2003·No. 2 CA-CR 2001-0033 - 2 CA-CR 2002-0046 (consolidated)·Published

Opinion

IN THE COURT OF APPEALS

STATE OF ARIZONA

DIVISION TWO

THE STATE OF ARIZONA, )

)

Appellee, )

)

v. ) 2 CA-CR 2001-0033 )

JOHN ROBERT ESSER, )

)

Appellant. )

)

)

THE STATE OF ARIZONA, ) 2 CA-CR 2002-0046 ) (Consolidated)

Appellee, ) DEPARTMENT B )

v. ) OPINION )

JOSE MUNOZ VIDAL, )

)

Appellant. )

)

APPEAL FROM THE SUPERIOR COURT OF PIMA COUNTY Cause Nos. CR-65286 and CR-64727 Honorable Paul S. Banales, Judge Pro Tempore AFFIRMED

Terry Goddard, Arizona Attorney General By Randall M. Howe, Diane Leigh Hunt, and Tucson Joseph L. Parkhurst Attorneys for Appellee

Isabel G. Garcia, Pima County Legal Defender Tucson By Alex Heveri Attorneys for Appellant Esser By Robb P. Holmes Attorneys for Appellant Vidal

P E L A N D E R, Presiding Judge.

BACKGROUND

¶1 After a jury trial, appellant John Robert Esser was found guilty of aggravated driving with an alcohol concentration of 0.10 or greater within two hours of driving in violation of A.R.S. §§ 28-1381(A)(2) and 28-1383(A)(1).1 The trial court sentenced him to an aggravated, 7.5-year term of imprisonment, enhanced by one historical prior felony conviction. After a separate jury trial, appellant Jose Munoz Vidal was found guilty of four felonies involving alcohol and driving2 and was sentenced to concurrent, presumptive, ten-year terms of imprisonment, enhanced by two historical prior felony convictions. Each appellant challenges the trial court’s denial of his motion to suppress the results of his alcohol breath tests, which had been conducted using an Intoxilyzer 5000 device.

¶2 We consolidated the appeals because the cases were consolidated at the trial level for purposes of the motion to suppress the breath test results, because only one set of exhibits was introduced at that proceeding, and because the arguments each appellant raises on appeal about the denial of that motion are virtually identical. Pursuant to Rule 31.26, Ariz. R. Crim. P., 17 A.R.S., we address in this opinion only appellants’ issue relating to the breath-testing device. In a separate memorandum decision filed this date, we address two other issues Vidal raises, neither of which are opinion worthy or require reversal. See Ariz. R. Sup. Ct. 111, 17A A.R.S. We affirm the convictions and sentences of both appellants.

1 Section 28-1381(A)(2) was subsequently amended to proscribe driving with an alcohol concentration of 0.08 or greater within two hours of driving. 2001 Ariz. Sess. Laws, ch. 95, § 5.

2 Aggravated driving under the influence (DUI) of an intoxicant while license is suspended or revoked; aggravated driving with an alcohol concentration of 0.10 or more while license is suspended or revoked; aggravated DUI with two DUI convictions in the previous sixty months; and aggravated driving with an alcohol concentration of 0.10 or more with two DUI convictions in the previous sixty months.

DISCUSSION

I. Physiology-based Challenge to Intoxilyzer 5000

¶3 Vidal filed a motion to suppress the evidence of his alcohol breath test results, raising statutory and Frye 3 challenges to the admissibility of those results based on the science and respiratory physiology underlying the tests. Esser and five other defendants joined in that motion.4 After three hearings featuring extensive expert testimony and after reviewing the numerous exhibits, primarily scientific articles,5 that had been introduced into evidence, the trial court denied the motion as to all defendants. We review the denial of a motion to suppress evidence for a clear abuse of discretion, viewing the evidence presented at the suppression hearing in the light most favorable to upholding the trial court’s factual findings and reviewing its legal conclusions de novo. State v. Sanchez, 200 Ariz. 163, ¶5, 24 P.3d 610, ¶5 (App. 2001).

¶4 The motion to suppress was based on the conclusions of Dr. Michael P. Hlastala, an expert in pulmonary physiology who testified for the defense about alcohol breath tests, and on a Department of Health Services (DHS) regulation that requires breath specimens to be “alveolar in composition.” Ariz. Admin. Code R9-14-403(B)(1). Hlastala explained that the theory of alcohol breath testing originated decades ago when the scientific understanding of pulmonary physiology was primitive and before the development of sophisticated testing devices. In what Hlastala termed the “old paradigm,” alcohol was thought to be exchanged between the

3 Frye v. United States, 293 F. 1013 (D.C. Cir. 1923).

4 Esser and Vidal also sought suppression below on due process grounds, but they have not pursued that claim on appeal.

5 The state erroneously asserts that these exhibits are not part of Vidal’s record on appeal.

See Rule 31.8(a)(1) (record on appeal composed in part of “all documents, papers, books and photographs introduced into evidence”).

blood and the breath in the alveoli, the tiny air sacs deep in the lungs, in the same manner as oxygen and carbon dioxide are exchanged between blood and breath in the alveoli. The alcohol was thought to enter the breath in the alveoli in equilibrium with the percentage of alcohol in the blood in the pulmonary capillaries that line the alveoli. The airways, comprising the mouth, trachea, and bronchi, were considered dead air spaces in which no significant alcohol exchange occurred. Under this paradigm, the alcohol concentration of an exhaled breath from a subject with alcohol in his or her blood is expected to be initially very low as breath is expelled from the airways; to rise as the exhalation begins to incorporate alveolar air; and, finally, to level out at equilibrium when the exhalation contains only alveolar air, at what is termed the “alveolar plateau.”

¶5 Based on his own research and that of others, however, Hlastala had determined that the alveolar plateau does not occur. Rather, breath exhaled from the alveolar region and passed through the airways shows a gradual, almost linear increase in alcohol concentration for as long as the subject exhales, leveling off only at the end of the breath when the exhalation ceases. In addition, tests of controlled “rebreathed” exhalations, in which the subject breathes in and out of a bag before being tested, result in alcohol readings that approximate the equilibrium level and are higher than the reading from a single exhalation by the same subject, belying the old paradigm concept that testing a single exhalation accurately measures the alcohol exchanged in the alveoli. Moreover, Hlastala had found that breath tests results can be significantly altered by pretest breathing patterns such as hyperventilation and holding one’s breath, anomalies that could not be explained by the old paradigm. Based on these and other findings, Hlastala concluded that the old paradigm does not accurately reflect the physiology of breath alcohol exchange and arrived at his own paradigm.

¶6 Noting that alcohol is a gas highly soluble in water and affected by temperature, Hlastala theorized that a significant portion of alcohol-breath exchange occurs as the breath passes over the mucous-bearing tissues in the airways, which heat and humidify inhaled air before it reaches the alveoli. Hlastala concluded that inhaled air absorbs alcohol along with moisture as it passes over those mucous membranes so that, by the time it reaches the alveoli, the breath alcohol concentration is already in equilibrium with blood alcohol concentration, and no further alcohol exchange occurs in the alveoli. He further concluded that, during exhalation, alcohol is initially desorbed back into the mucous membranes, which have become somewhat depleted of alcohol and moisture during the inhalation. Less and less alcohol is desorbed as the mucous membranes become more resaturated with moisture and alcohol during the exhalation, explaining the absence of an alveolar plateau and the steady rise in alcohol concentration for the duration of an exhalation.

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