Rogers v. State Bd. of Med. Examiners

371 So. 2d 1037
CourtDistrict Court of Appeal of Florida
DecidedJanuary 9, 1979
DocketEE-454
StatusPublished
Cited by6 cases

This text of 371 So. 2d 1037 (Rogers v. State Bd. of Med. Examiners) is published on Counsel Stack Legal Research, covering District Court of Appeal of Florida primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.

Bluebook
Rogers v. State Bd. of Med. Examiners, 371 So. 2d 1037 (Fla. Ct. App. 1979).

Opinion

371 So.2d 1037 (1979)

Robert J. ROGERS, M.D., Petitioner,
v.
STATE BOARD OF MEDICAL EXAMINERS of Florida, Respondent.

No. EE-454.

District Court of Appeal of Florida, First District.

January 9, 1979.
Rehearing Denied January 31, 1979.

*1038 Andrew A. Graham, Cocoa, for petitioner.

Michael Schwartz, of Slepin & Schwartz, Tallahassee, for respondent.

BOYER, Acting Chief Judge.

By this proceeding review is sought of an order of the State Board of Medical Examiners of Florida (the Board) approving and effectuating a recommended order of a duly appointed hearing officer. The question presented is relatively narrow, viz: To what extent does the State of Florida have constitutional authority to prohibit a nonharmful mode of medical treatment by a licensed physician after full disclosure to, and election by, a patient? Petitioner also seeks a declaration of invalidity of F.S. 458.1201(1)(p).[1]

Petitioner is, and at all material times has been, a practicing physician in Brevard County, Florida. In June of 1974 a nonresident daughter of one of petitioner's patients wrote a letter to the Brevard County Medical Association (BCMA) inquiring about a medical procedure or methodology known as chelation therapy, as a result of which correspondence BCMA commenced an inquiry into chelation therapy and its use by petitioner. Several hearings were held and ultimately the BCMA ordered petitioner to discontinue the use or practice of chelation therapy. Petitioner refused and was expelled from the BCMA. As a result of that expulsion, pursuant to F.S. 458.1201(1)(p), the respondent Board issued an administrative complaint following which a hearing was held on September 10, 1976. In due course the hearing officer filed a recommended order which was thereafter approved and adopted by the Board. Petitioner was reprimanded; ordered to immediately cease and desist from engaging in the utilization of chelation therapy in the treatment of arteriosclerosis unless and until authorized by the Florida State Board of Medical Examiners and his license to practice medicine was placed on probation for a period of one year. Review was then sought in this court.

The record reveals that chelation therapy consists of a series of intravenous injections of a chelating drug, usually disodium ethylenediamine tetraacetic acid (hereafter disodium EDTA, Na2 EDTA, or EDTA). Each injection takes approximately three to four hours to administer, and a normal course of *1039 treatment usually involves twenty such injections. The treatments are specifically intended to treat arteriosclerosis (hardening of the arteries), therosclerosis (deposits on the inner lining of the arteries), and other generalized circulatory deficiencies caused by excess calcium in the circulatory vessels. The common chelating agent, Na2 EDTA, was originally discovered by I.G. Farben in the 1930's and is widely used as a food preservative. EDTA intravenous treatment is often recommended for lead poisoning and other diseases requiring removal of heavy metals from the body. Chelation treatments were widely used during World War II in treating sailors who had contracted poisoning from leaded paint. Since World War II more than 1500 scientific articles and studies have been published concerning practically every application of the chelating process in the body. Many of those articles are contained in the record.

The precise chemical reaction whereby metals, or calcium, are removed from the body through chelation is not yet completely known nor understood. However, through the years, many doctors have observed that after EDTA infusion, the urine calcium level rises and remains at relatively high levels for some months after administration of the treatments. The theory, now generally agreed upon by chelation proponents, is that the chelating salt binds with ionic calcium in the blood, causing a temporary calcium deficiency in the blood. This is rapidly replaced by calcium in precipitate form ionizing in the bloodstream. This calcium, known as metastatic calcium, comes from the walls of the blood vessels and from calcium precipitate in every cell. Many experts believe that the metastatic calcium sludge in each cell causes the cells gradually to disfunction. This phenomenon, coupled with the better known effects of calcium deposits on the interior walls of the blood vessels, results in a gradual decline in blood flow and cell function. Thus, if the pernicious calcium buildup can be prevented or reversed, cells and vessels can continue to function well into old age, reducing the inevitable effects of hardening of the arteries and other vascular occlusive diseases.

The record is replete with claimed instances of dramatic restoration of blood flow to the extremities resulting in arrest of gangrene, restored sensation, increased temperature and return of normal color to toes, fingers, hands and feet after chelation treatment. In one exceptionally dramatic case history, Reynolds Hall, a patient of petitioner, allegedly regained his sight during his seventh chelation treatment.

Chelation therapy is, then, infusion of a chelating agent (generally Na2 EDTA) into the blood stream over several hours, a treatment which is repeated about 20 times, generally over a period of a month or more.

The record reveals that over one million persons die each year in the United States from the effects of vascular occlusive disease. Ultimately, vascular occlusive disease affects all persons to a greater or lesser degree. Yet few treatments are known which combat the disease. Bypass surgery may be effective if the patient can tolerate surgery,[2] if the occlusion is localized, and if it is accessible to the surgeon.

Vasil dilator drugs can temporarily relax blood vessels, allowing increased flow, if the inner walls are not covered by calcium plaque which restricts elasticity of the vessels. Another factor which contributes to the advancement of vascular occlusive disease is diet. Thus, if a patient reduces his intake of calcium and cholesterol the progress of the disease may be slowed. The only other alternative treatment known is chelation. Chelation is intended to remove the calcium that is the cause of the disease.

There is no doubt that the state has the authority, even duty, to control admission of persons to the practice of medicine and to prevent incompetent doctors from injuring patients. (Page v. State Board of Medical Examiners, 141 Fla. 294, *1040 193 So. 82, 83 (Fla. 1940) See also Page v. Watson, 140 Fla. 536, 192 So. 205 (1938)) The state has broad powers where the health of its citizens are concerned. (Barsky v. Board of Regents of University of State of New York, 347 U.S. 442, 74 S.Ct. 650, 98 L.Ed. 829 (1954)). The legislature has a right to prescribe reasonable rules and regulations that shall control the practice of medicine. (Page v. State Board of Medical Examiners, supra.) Such authority is, however, limited in that such rules and regulations must bear a reasonable relationship to the public safety, health, morals and general welfare. (Stadnik v. Shell's City, Inc., 140 So.2d 871 (Fla. 1962))

The Florida case most explicit on the issue of state authority to control a profession is Golden v. McCarty, 337 So.2d 388 (Fla. 1976), in which the Supreme Court upheld a statute which required tattooing to be performed by a licensed doctor or dentist or by a person under the direction of one so licensed.

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