Remick v. SSA

2011 DNH 176
CourtDistrict Court, D. New Hampshire
DecidedOctober 21, 2011
DocketCV-10-578-PB
StatusPublished
Cited by3 cases

This text of 2011 DNH 176 (Remick v. SSA) is published on Counsel Stack Legal Research, covering District Court, D. New Hampshire primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.

Bluebook
Remick v. SSA, 2011 DNH 176 (D.N.H. 2011).

Opinion

Remick v. SSA CV-10-578-PB 10/21/11

UNITED STATES DISTRICT COURT FOR THE DISTRICT OF NEW HAMPSHIRE

Gary Arlon Remick

v. Case N o . 10-cv-578-PB Opinion N o . 2011 DNH 176 Michael J. Astrue, Commissioner, Social Security Administration

MEMORANDUM AND ORDER

Gary Arlon Remick filed a complaint, pursuant to 42 U.S.C.

§ 405(g), seeking judicial review of the decision of the

Commissioner denying his application for supplemental social

security benefits. Remick contends that the Administrative Law

Judge (“ALJ”) failed to consider the combined effect of Remick’s

impairments in determining his residual functional capacity

(“RFC”), and gave insufficient weight to the opinion of Remick’s

treating physician, Dr. Sebastian Strobel. The Commissioner

moves to affirm the decision. For the reasons provided below, I

affirm the Commissioner’s decision.

I. BACKGROUND1

Remick applied for supplemental social security benefits on

April 9, 2008, when he was fifty years old. He alleged an

1 The background information is taken from the parties’ Joint Statement of Material Facts. See L.R. 9.1(b). Citations to the Administrative Transcript are indicated by "Tr."

1 inability to work as of February 1 8 , 2008, due to multiple

impairments, including diabetes mellitus,2 atrial fibrillation,3

neurogenic bladder,4 dysthymic disorder,5 and anxiety disorder.6

He completed the tenth grade of high school, and in the past, he

worked as a custodian in a school system and as a laborer in a

lumber yard.

A. Medical History

Remick was hospitalized on February 1 8 , 2008, following a

visit to the emergency room at the Dartmouth Hitchcock Medical

Center (“DHMC”), where he requested alcohol detoxification.

Upon discharge on March 5 , 2008, the primary diagnoses were:

diabetes mellitus, atrial fibrillation, alcohol detoxification,

and urinary retention from either bladder outlet obstruction or

2 Diabetes mellitus is “a chronic metabolic disorder in which the use of carbohydrate is impaired and that of lipid and protein enhanced. It is caused by an absolute or relative deficiency of insulin . . . .” Stedman’s Medical Dictionary at 529 (28th ed. 2006) (“Stedman’s”). 3 Atrial fibrillation is “[v]ermicular twitching . . . of individual muscular fibers . . . in which the normal rhythmic contractions of the cardiac atria are replaced by rapid irregular twitchings of the muscular wall . . . .” Stedman’s at 722-23. 4 Neurogenic bladder is “any defective functioning of bladder due to impaired innervation . . . .” Stedman’s at 226. 5 Dysthymic disorder is “a chronic disturbance of mood characterized by mild depression or loss of interest in usual activities.” Stedman’s at 569. 6 Anxiety disorder is characterized by “chronic, repeated episodes of anxiety reactions.” Stedman’s at 569. 2 urinary tract infection (“UTI”). During this admission, he was

placed on insulin, and at the time of discharge, his glucose was

well controlled. He also began taking diltiazem, a medication

for atrial fibrillation. Lastly, a catheter was inserted to

address urinary retention and he was taught to straight-

catheterize.

1. Atrial Fibrillation

On March 1 1 , 2008, at a first doctor’s visit following his

hospitalization, Remick reported that he had not noticed

problems with his heart rate being too fast or slow. Dr. Dhaval

Parikh, who saw Remick on April 1 7 , 2008 at the DHMC, noted that

at that time, Remick was completely asymptomatic with atrial

fibrillation and that his heart rate was mostly controlled.

During a May 1 9 , 2008 visit with Dr. Strobel, Remick’s primary

care provider, no cardiovascular symptoms were noted. The

assessment was that Remick’s atrial fibrillation rate was

controlled. The only symptom noted was gravity dependent edema

in the afternoon, most likely from diltiazem. At a subsequent

visit, on July 1 6 , 2008, Remick again reported no difficulties

with his heart and no chest pain, but reported that he still had

lower extremity edema that would be gone in the morning, and

that while exercising he experienced shortness of breath. Dr.

Strobel indicated that Remick did a lot of walking. Dr. Strobel

also noted that on June 1 3 , 2008, while following a Bruce

3 protocol,7 Remick had to stop because of fatigue, the target rate

was not reached, and Remick developed atrial flutter8 during

recovery. A Holter monitor9 test was performed on August 1 8 ,

2008. The physician’s interpretation was periods of normal

sinus rhythm with multiple episodes of fibrillation, flutter,

and supraventricular tachycardia10.

In a subsequent visit with Dr. Strobel on August 2 6 , 2008,

Remick’s heart rate was normal and there was no edema in his

extremities. There was a follow-up cardiology visit on August

2 7 , 2008, during which Remick reported that he continued to go

for walks and expressed no functional limitations. On October

1 5 , 2008, Dr. Strobel again found that Remick’s heart rate was

well controlled. Upon examination, there was no edema in his

extremities. During the next visit, on January 2 1 , 2009, Remick

reported to Dr. Strobel that he was exercising well and had

7 Bruce protocol is “a standardized protocol for electrocardiogram-monitored exercise using increasing speeds and elevations of the treadmill.” Stedman’s at 1584. 8 Atrial flutter is characterized by “rapid regular atrial contractions occurring usually at rates between 250 and 330 per minute . . . .” Stedman’s at 749. 9 Holter monitor is “a technique for long-term, continuous[,] usually ambulatory, recording of electrocardiographic signals on magnetic tape for scanning and selection of significant but fleeting changes that might otherwise escape notice.” Stedman’s at 1222. 10 Tachycardia is “[r]apid beating of the heart, conventionally applied to rates over 90 beats per minute.” Stedman’s at 1931. Supraventricular tachycardia occurs “anywhere above the ventricular level, i.e., sinus node, atrium, atrioventricular junction.” Id. 4 decreased leg swelling. Dr. Strobel again noted that there was

no edema in his extremities and that his heart rate was well

controlled. On the same date, Dr. Grossman reported that Remick

was exercising without difficulty and that he was not

experiencing shortness of breath.

When Dr. Strobel saw Remick on May 1 3 , 2009, Remick

reported swelling in his right leg, especially at night. Dr.

Strobel found that his heart rate seemed to be well controlled

but that Remick was flipping in and out of fibrillation and

flutter. During a cardiology visit on June 3 , 2009, Remick

reported that he had more dependent edema over the past year and

that although his legs were usually free of fluid in the

morning, the fluid accumulated progressively during the day.

The assessment was that his ventricular responses to atrial

arrhythmias appeared to be well controlled, and that the

swelling was related to diltiazem.

Remick saw Dr. Strobel again on July 2 1 , 2009 and reported

that he was “okay” on metoprolol and diltiazem, the two atrial

fibrillation prescriptions he was taking, and that he had less

swelling in his ankles. At follow-up visits on October 1 6 , 2009,

November 3 , 2009, January 1 5 , 2010, and March 5 , 2010, Dr.

Strobel noted that Remick’s heart rate continued to be well

controlled and that there was no edema in his extremities.

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