Remick v. SSA

2011 DNH 176
District Court, D. New Hampshire·Decided October 21, 2011·No. CV-10-578-PB·Published·Cited by 3 cases

Opinion

Remick v. SSA CV-10-578-PB 10/21/11

UNITED STATES DISTRICT COURT FOR THE DISTRICT OF NEW HAMPSHIRE

Gary Arlon Remick

v. Case N o . 10-cv-578-PB Opinion N o . 2011 DNH 176 Michael J. Astrue, Commissioner, Social Security Administration

MEMORANDUM AND ORDER

Gary Arlon Remick filed a complaint, pursuant to 42 U.S.C.

§ 405(g), seeking judicial review of the decision of the Commissioner denying his application for supplemental social security benefits. Remick contends that the Administrative Law Judge (“ALJ”) failed to consider the combined effect of Remick’s impairments in determining his residual functional capacity (“RFC”), and gave insufficient weight to the opinion of Remick’s treating physician, Dr. Sebastian Strobel. The Commissioner moves to affirm the decision. For the reasons provided below, I affirm the Commissioner’s decision.

I. BACKGROUND1

Remick applied for supplemental social security benefits on April 9, 2008, when he was fifty years old. He alleged an

1 The background information is taken from the parties’ Joint Statement of Material Facts. See L.R. 9.1(b). Citations to the Administrative Transcript are indicated by "Tr."

inability to work as of February 1 8 , 2008, due to multiple impairments, including diabetes mellitus,2 atrial fibrillation,3 neurogenic bladder,4 dysthymic disorder,5 and anxiety disorder.6 He completed the tenth grade of high school, and in the past, he worked as a custodian in a school system and as a laborer in a lumber yard.

A. Medical History Remick was hospitalized on February 1 8 , 2008, following a visit to the emergency room at the Dartmouth Hitchcock Medical Center (“DHMC”), where he requested alcohol detoxification. Upon discharge on March 5 , 2008, the primary diagnoses were: diabetes mellitus, atrial fibrillation, alcohol detoxification, and urinary retention from either bladder outlet obstruction or

2 Diabetes mellitus is “a chronic metabolic disorder in which the use of carbohydrate is impaired and that of lipid and protein enhanced. It is caused by an absolute or relative deficiency of insulin . . . .” Stedman’s Medical Dictionary at 529 (28th ed. 2006) (“Stedman’s”). 3 Atrial fibrillation is “[v]ermicular twitching . . . of individual muscular fibers . . . in which the normal rhythmic contractions of the cardiac atria are replaced by rapid irregular twitchings of the muscular wall . . . .” Stedman’s at 722-23. 4 Neurogenic bladder is “any defective functioning of bladder due to impaired innervation . . . .” Stedman’s at 226. 5 Dysthymic disorder is “a chronic disturbance of mood characterized by mild depression or loss of interest in usual activities.” Stedman’s at 569. 6 Anxiety disorder is characterized by “chronic, repeated episodes of anxiety reactions.” Stedman’s at 569.

urinary tract infection (“UTI”). During this admission, he was placed on insulin, and at the time of discharge, his glucose was well controlled. He also began taking diltiazem, a medication for atrial fibrillation. Lastly, a catheter was inserted to address urinary retention and he was taught to straight- catheterize.

1. Atrial Fibrillation On March 1 1 , 2008, at a first doctor’s visit following his hospitalization, Remick reported that he had not noticed problems with his heart rate being too fast or slow. Dr. Dhaval Parikh, who saw Remick on April 1 7 , 2008 at the DHMC, noted that at that time, Remick was completely asymptomatic with atrial fibrillation and that his heart rate was mostly controlled. During a May 1 9 , 2008 visit with Dr. Strobel, Remick’s primary care provider, no cardiovascular symptoms were noted. The assessment was that Remick’s atrial fibrillation rate was controlled. The only symptom noted was gravity dependent edema in the afternoon, most likely from diltiazem. At a subsequent visit, on July 1 6 , 2008, Remick again reported no difficulties with his heart and no chest pain, but reported that he still had lower extremity edema that would be gone in the morning, and that while exercising he experienced shortness of breath. Dr. Strobel indicated that Remick did a lot of walking. Dr. Strobel also noted that on June 1 3 , 2008, while following a Bruce

protocol,7 Remick had to stop because of fatigue, the target rate was not reached, and Remick developed atrial flutter8 during recovery. A Holter monitor9 test was performed on August 1 8 , 2008. The physician’s interpretation was periods of normal sinus rhythm with multiple episodes of fibrillation, flutter, and supraventricular tachycardia10.

In a subsequent visit with Dr. Strobel on August 2 6 , 2008, Remick’s heart rate was normal and there was no edema in his extremities. There was a follow-up cardiology visit on August 2 7 , 2008, during which Remick reported that he continued to go for walks and expressed no functional limitations. On October 1 5 , 2008, Dr. Strobel again found that Remick’s heart rate was well controlled. Upon examination, there was no edema in his extremities. During the next visit, on January 2 1 , 2009, Remick reported to Dr. Strobel that he was exercising well and had

7 Bruce protocol is “a standardized protocol for electrocardiogram-monitored exercise using increasing speeds and elevations of the treadmill.” Stedman’s at 1584. 8 Atrial flutter is characterized by “rapid regular atrial contractions occurring usually at rates between 250 and 330 per minute . . . .” Stedman’s at 749. 9 Holter monitor is “a technique for long-term, continuous[,] usually ambulatory, recording of electrocardiographic signals on magnetic tape for scanning and selection of significant but fleeting changes that might otherwise escape notice.” Stedman’s at 1222. 10 Tachycardia is “[r]apid beating of the heart, conventionally applied to rates over 90 beats per minute.” Stedman’s at 1931. Supraventricular tachycardia occurs “anywhere above the ventricular level, i.e., sinus node, atrium, atrioventricular junction.” Id.

decreased leg swelling. Dr. Strobel again noted that there was no edema in his extremities and that his heart rate was well controlled. On the same date, Dr. Grossman reported that Remick was exercising without difficulty and that he was not experiencing shortness of breath.

When Dr. Strobel saw Remick on May 1 3 , 2009, Remick reported swelling in his right leg, especially at night. Dr. Strobel found that his heart rate seemed to be well controlled but that Remick was flipping in and out of fibrillation and flutter. During a cardiology visit on June 3 , 2009, Remick reported that he had more dependent edema over the past year and that although his legs were usually free of fluid in the morning, the fluid accumulated progressively during the day. The assessment was that his ventricular responses to atrial arrhythmias appeared to be well controlled, and that the swelling was related to diltiazem.

Remick saw Dr. Strobel again on July 2 1 , 2009 and reported that he was “okay” on metoprolol and diltiazem, the two atrial fibrillation prescriptions he was taking, and that he had less swelling in his ankles. At follow-up visits on October 1 6 , 2009, November 3 , 2009, January 1 5 , 2010, and March 5 , 2010, Dr. Strobel noted that Remick’s heart rate continued to be well controlled and that there was no edema in his extremities.

2. Bladder Dysfunction During his March 1 1 , 2008 visit at the DHMC, Remick reported straight-catheterizing every three hours to address his bladder dysfunction. At a subsequent visit, on April 1 7 , 2008, a urodynamic study showed that he had atonic bladder with impaired sensation and the resulting treatment plan was to continue to cath often enough to keep his urine volume under 500 cc. The diagnosis was confirmed in a GI/Hepatology consultation on May 6, 2008.

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