Ray Oaks Machine Shop v. Workmen's Compensation Appeal Board
This text of 600 A.2d 1305 (Ray Oaks Machine Shop v. Workmen's Compensation Appeal Board) is published on Counsel Stack Legal Research, covering Commonwealth Court of Pennsylvania primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.
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Ray Oaks Machine Shop (Employer) appeals from an order of the Workmen’s Compensation Appeal Board (Board) which affirmed the referee’s award of fatal claim benefits to Mary E. Bubenko (Claimant), the widow of Alex J. Bubenko (Decedent).
Decedent worked as a coal miner for seven and a half years beginning in 1936, where he was exposed to coal dust. Later, Decedent worked as a grinder in several machine shops for twenty-four years, where he was exposed to silica dust. Decedent’s last employer was Ray Oaks, where he worked for one and a half years. Decedent’s last day of work was July 3, 1980.
During his lifetime, Decedent had filed a claim petition under Section 108 of the Pennsylvania Workmen’s Compensation Act (the Act),1 alleging total disability or, in the alternative, partial disability as a result of coal workers’ pneumoconiosis and bronchitis. He began receiving compensation benefits for partial disability from silicosis commencing on August 9,1983, at the rate of $100.00 per week. Neither Employer nor Decedent appealed the award.
On December 22, 1986 Decedent died of an acute myocardial infarction. On March 7, 1988 Claimant filed a fatal claim petition alleging that her husband’s death was second[183]*183arily caused by, but not limited to, his work-related inhalation of coal dust, silica dust, grinding wheel dust and fumes. Employer filed a timely answer denying every allegation set forth in the fatal claim petition.
After the matter was heard, the referee, in a decision dated January 12,1990, made the following relevant finding of fact:
15. After careful review, I find the testimony and opinions of Dr. Sidney A. Goldblatt more persuasive than those expressed by Dr. Harvey Mendelow. I, therefore, find that mixed dust pneumoconiosis; i.e., coal workers’ pneumoconiosis and silicosis, was a co-equal cause of death with the myocardial infarction/coronary artery disease; and therefore, a significant contributing factor. (Emphasis added).
The referee found that Claimant sustained her burden of proving that her husband’s hazardous exposure during the course of his employment with Employer was a significant contributing factor in bringing about Decedent’s death and, accordingly, awarded fatal claim benefits. On Employer’s appeal, the Board, by decision dated February 26, 1991, found that the referee’s decision was supported by substantial evidence and affirmed the fatal claim award. Employer’s appeal to this Court followed.2
Employer contends that it is not liable to compensate Claimant because the referee’s finding that “mixed dust pneumoconiosis” was a substantial contributing factor in bringing about Decedent’s death is not supported by the record. Where there are multiple causes of death and the immediate cause was non-compensable, the requirements of Section 301(c)(2) of the Act, 77 P.S. § 411(2), may be met by a showing with unequivocal medical evidence that the deceased suffered from an occupational disease and that it [184]*184was a substantial, contributing factor among the secondary causes in bringing about death. Kusenko v. Republic Steel Corp., 506 Pa. 104, 107, 484 A.2d 374, 376 (1984) (quoting McCloskey v. Workmen’s Compensation Appeal Board, 501 Pa. 93, 101, 460 A.2d 237, 241 (1983)).
In reaching her conclusion, the referee considered the testimony of both parties’ medical experts. Dr. Goldblatt, Claimant’s medical expert, testified that he had reviewed microscopic slides, the protocol of the autopsy performed by Dr. Monteleone, the certificate of death, and various medical reports and records. Based on his review, Dr. Goldblatt was of the professional opinion that Decedent died of co-equal causes of death, namely, a myocardial infarction related to a cardiac condition and severe coal workers’ pneumoconiosis of the macular and micronodular type. Dr. Goldblatt also noted that an abundance of birefringent silica crystals were present on autopsy, and testified that silicosis was “strategically important” (R.R. 124a) in bringing about Decedent’s death.
Dr. Mendelow, Employer’s medical expert, testified that based on his review of various hospital reports, testimony and the autopsy report, he was of the professional opinion that Decedent’s death was a direct result of a sudden cardiac arrest due to advanced coronary artery disease. He did not believe that the coronary artery disease was related to Decedent’s employment. On cross-examination, Dr. Mendelow opined that Decedent’s exposure to mixed dust was not a substantial factor as evidenced by the pathologic findings, but was instead a modest contributing factor. Dr. Mendelow further testified that he found no silicosis and stated that the hypoxia was a direct result of Decedent’s coronary disease.
Of the two conflicting medical opinions, the referee found Dr. Goldblatt’s opinions to be more persuasive than those expressed by Dr. Mendelow.3 The referee then concluded [185]*185that “mixed dust pneumoconiosis,” which she described as a combination of coal workers’ pneumoconiosis and silicosis, was a co-equal cause of death with the myocardial infarction and coronary artery disease and was therefore a significant contributing factor in bringing about Decedent’s death.
Our review of the record, however, discloses that neither Dr. Goldblatt nor Dr. Mendelow diagnosed “mixed dust pneumoconiosis” in Decedent. Instead, Dr. Goldblatt diagnosed coal workers’ pneumoconiosis from coal mine employment and silicosis from machine shop employment and Dr. Mendelow, while referring to the presence of silica and carbide contained in grinding wheels as “mixed dust,” did not believe that the “mixed dust” contributed to death and further testified that Decedent did not have coal workers’ pneumoconiosis or silicosis.
In the absence of a diagnosis of “mixed dust pneumoconiosis”, the presence of two enumerated occupational diseases 4 cannot create a third disease caused by a mixture of undefined dusts which is compensable under the omnibus provision of Section 108(n) of the Act. Koppers Co., Inc. v. Workmen’s Compensation Appeal Board (Boyle), 113 Pa.Commonwealth Ct. 161, 536 A.2d 509, 511 (1988), overruled on other grounds, Bell v. Workmen’s Compensation Appeal Board (Gateway Coal Co.), 118 Pa.Commonwealth Ct. 320, 545 A.2d 430 (1988). As Employer points out, the referee cannot combine the medical opinions of Dr. Goldblatt and Dr. Mendelow to find “mixed dust pneumoconiosis” resulting from the presence of coal workers’ pneumoconiosis and silicosis where Dr. Goldblatt did not diagnose “mixed dust” and where Dr. Mendelow did not diagnose coal workers’ pneumoconiosis or silicosis.5 Accordingly, we [186]*186find that there is no medical support in the record and no legal basis for the referee to award benefits for “mixed dust pneumoconiosis” under Section 108(n) of the Act.
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Cite This Page — Counsel Stack
600 A.2d 1305, 143 Pa. Commw. 180, 1991 Pa. Commw. LEXIS 710, Counsel Stack Legal Research, https://law.counselstack.com/opinion/ray-oaks-machine-shop-v-workmens-compensation-appeal-board-pacommwct-1991.