Perry v. Industrial Commission of Arizona

741 P.2d 693, 154 Ariz. 226
CourtCourt of Appeals of Arizona
DecidedJuly 16, 1987
Docket1 CA-IC 3600
StatusPublished
Cited by8 cases

This text of 741 P.2d 693 (Perry v. Industrial Commission of Arizona) is published on Counsel Stack Legal Research, covering Court of Appeals of Arizona primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.

Bluebook
Perry v. Industrial Commission of Arizona, 741 P.2d 693, 154 Ariz. 226 (Ark. Ct. App. 1987).

Opinion

*227 SHELLEY, Presiding Judge.

This is a special action review of an Industrial Commission award denying reopening of a 1977 claim on the basis of preclusion. We agree that preclusion applies and therefore affirm the award.

Curtis Perry, the petitioner employee (claimant) is a former professional basketball player. In approximately January 1977, while playing for the Phoenix Suns (Suns), the respondent employer, claimant developed severe low back pain and was unable to complete the remainder of the season. He was then twenty-nine years old and had never had back problems before.

Compensability was accepted. The Suns’ team physician, Paul M. Steingard, D.O., diagnosed a lumbosacral strain with possible nerve root irritation. Dr. Steingard consulted two orthopedic surgeons, Paul James Nichols, M.D. and William C. Brainard, M.D. The claimant also saw orthopedic surgeon Thomas Henry Tabor, Jr., M.D., who supervised active treatment.

- In February 1977, Dr. Nichols noted muscle spasm, a scoliotic tilt, muscle atrophy of the right thigh, and absent right patellar reflex. X-rays confirmed a first degree spondylolisthesis at L3-L4, bilateral breaks in the pars interartieularis at L3, and disc degeneration and narrowing between L3 and L4. Dr. Nichols subsequently reported that the spondylolisthesis, rather than a herniated disc, accounted for the claimant’s symptoms, but he did not specify the cause of the spondylolisthesis itself. Dr. Brainard, however, reported on February 24th that the pars defects “appear non-sclerotic, and I would b'e concerned about this being an acute injury, i.e. a stress fracture through the pars interartieularis with the particular activities mentioned. The patient may have had a unilateral defect without slip, (and may have had the long-standing degenerative disc process without symptomotology, and then developed stress fracture in the area with his activities, producing the symptomotology and further strain on the disc space itself.” Dr. Brainard also recommended a bone scan, which revealed an abnormal area of increased uptake at L3. A followup bone scan approximately one year later was negative.

The claimant’s symptoms improved and he attempted to resume his career the following season. The pain returned, and he was again disabled in January 1978. He failed to pass the fall 1978 team physical and never played professional basketball again.

In October 1978, Dr. Tabor reported that the claimant was again asymptomatic and had been discharged without a permanent impairment. Relying on Dr. Tabor’s opinion, the respondent carrier (Carrier) closed the claim. The claimant protested the termination notice. A hearing was then conducted.

The claimant called only one medical witness, Dr. Steingard. Referring to Dr. Brainard’s report and to the bone scans, he acknowledged “there’s a question medically as to whether or not spondylolisthesis and the pars defect is a congenital thing, and then the flareup [of symptoms] is due to an acute injury ..., or whether or not it is, in fact, a stress fracture, a fresh new injury____” But in his opinion, the spondylolisthesis most likely was preexistent and there was no way of knowing the etiology of the pars defect. He considered these questions “academic” because “[t]here’s no question that the accident caused the symptoms____” He also testified that this symptomatic aggravation permanently disabled the claimant from playing professional basketball.

Dr. Tabor testified that the industrial injury was a temporary soft tissue sprain in the area of the bony defects. The defects themselves were developmental abnormalities. To support this opinion, Dr. Tabor relied on the x-rays taken immediately after the industrial injury, which indicated long-standing changes, and x-rays taken when the claim was closed, which-showed no degeneration since the industrial injury. He testified that these were findings inconsistent with a traumatic etiology. Dr. Tabor did rate a 5% permanent impairment for the underlying condition, but this rating was unrelated to the industrial injury. He also testified that the *228 prognosis was unpredictable—the claimant might remain asymptomatic or might have significant difficulty in the future.

On May 24, 1979, the administrative law judge issued the award terminating the claim without permanent impairment. He found that the structural defects were developmental, that the industrial injury had only temporarily aggravated the underlying condition, and that this condition rated a 5% impairment unrelated to the 1977 injury. The claimant did not seek review and the award accordingly became final. E.g., Sandoval v. Salt River Project, 117 Ariz. 209, 571 P.2d 706 (App.1977).

In February 1985, the claimant returned to Dr. Nichols, complaining of increased pain. X-rays revealed a significant collapse of the L3-L4 disc space. Dr. Nichols attributed this degeneration to the industrial injury.

Relying on this opinion, the claimant petitioned to reopen the 1977 injury claim. The Carrier denied the petition. The claimant timely protested the denial. Pending the scheduled hearings, Drs. Brainard and Tabor reexamined the claimant.

At these hearings, the claimant called both Dr. Nichols and Dr. Brainard. Dr. Nichols confirmed the additional degeneration of the disc space. He conceded that the spondylolisthesis caused this degeneration and therefore that the 1977 injury caused the degeneration only if it also caused the spondylolisthesis. In his opinion, this causal link was established because a stress fracture had caused the spondylolisthesis. He testified that the degeneration between 1977 and 1985, the two bone scans, and the claimant’s history substantiated this etiology.

Dr. Brainard concurred with Dr. Nichols for the same reasons. He, however, admitted that he had diagnosed the stress fracture in 1977. He also acknowledged that the stress fracture caused the spondylolisthesis and that this condition in turn was responsible for the degenerative narrowing.

Dr. Tabor’s opinion was unchanged. Although he had independently confirmed the markedly narrowed disc space, in his opinion this degeneration was an expected consequence of the nonindustrially related spondylolisthesis. He also disagreed that the bone scans were diagnostic of a stress fracture; to the contrary, he interpreted them to be consistent with his diagnosis.

The administrative law judge then issued the award denying reopening. He found that although the claimant’s condition had worsened since closing, the cause of this worsening was the underlying spondylolisthesis. He also found that Drs. Nichols and Brainard’s opinions were “nothing more nor less than new evidence/opinions on matters which were previously known/considered/litigated and/or which could have been litigated at the ... prior ‘closing’ proceedings in 1979____” He accordingly concluded that the prior adjudication that the spondylolisthesis was unrelated to the industrial injury was final and precluded reopening. This denial was affirmed on administrative review. The current special action followed.

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Bluebook (online)
741 P.2d 693, 154 Ariz. 226, Counsel Stack Legal Research, https://law.counselstack.com/opinion/perry-v-industrial-commission-of-arizona-arizctapp-1987.