Parsons v. Wood

1978 OK 84, 584 P.2d 1332, 1978 Okla. LEXIS 412
CourtSupreme Court of Oklahoma
DecidedJune 6, 1978
Docket49630
StatusPublished
Cited by7 cases

This text of 1978 OK 84 (Parsons v. Wood) is published on Counsel Stack Legal Research, covering Supreme Court of Oklahoma primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.

Bluebook
Parsons v. Wood, 1978 OK 84, 584 P.2d 1332, 1978 Okla. LEXIS 412 (Okla. 1978).

Opinion

IRWIN, Justice.

The principal defendant in this proceeding is appellee, Julian D. Wood, M.D., and the appellees will be referred to as appellee.

Appellant is the mother of decedent, Ralph W. Parson, Jr., who died in 1972 at the age of 25, and she is also the adminis-tratrix of his estate. Appellant commenced a malpractice action against appellee to recover damages resulting from appellee’s alleged negligence. Appellee filed a motion for summary judgment and submitted with such motion were the depositions of the appellant, the appellee, and Drs. M. and L., two other physicians also involved in Ralph’s examination and treatment. Also submitted were certain items of documentary evidence, such as medical reports. The trial court sustained appellee’s motion for summary judgment and appellant appealed.

On assignment to the Court of Appeals the judgment of the trial court was reversed and remanded on grounds that the evidence presented to the trial court demonstrated unresolved material factual controversies. Appellees seek certiorari.

Briefly summarized, appellant alleged in her petition that on January 29, 1970, and continuing through March 31,1970, appellee negligently examined, diagnosed and treated Ralph, and that appellee allowed Ralph’s appendix to burst resulting in peritonitis and the spreading of the deadly carcinoma cells through Ralph’s abdominal cavity, either expediting or causing Ralph’s death.

According to the record presented, appel-lee had been Ralph’s personal physician since Ralph’s early childhood. For many years Ralph had experienced classic sympto-mology of a chronic colon disease, ulcerative colitis. There is no dispute as to the pre-ex-isting condition of ulcerative colitis and Ralph’s earlier sicknesses and hospitalizations. 1 Symptoms of the disease in Ralph were cramping abdominal pain, diarrhea, fever, loss of appetite and, in episodes of acute aggravation, bloody bowel movements. After extensive diagnostic evaluation in the Mayo Clinic at age twelve, Ralph was referred back to appellee for continued out patient treatment consisting of combined dietary and medical management of the disease. Despite treatment, Ralph continued to have periodic episodes of distress from his condition, and such recurrent problems are typical of ulcerative colitis patients.

In late February of 1970, when approximately 23, Ralph again saw appellee complaining of upper abdominal pain. Appellee gave Ralph a routine physical examination and concluded he was experiencing a recurrence of his colitis condition combined with *1334 either indigestion or peptic ulcer. A muscle relaxant was prescribed and Ralph was sent home with instructions to report back in a few days. Ralph returned on March 2nd complaining of increased upper abdominal pain, chills, fever and other generalized aches and pains. Again appellee gave Ralph a physical examination and had a urinalysis run. Appellee did not take Ralph’s temperature assuming it was elevated. The diagnosis after this second visit was chronic ulcerative colitis and influenza. Ralph was given a penicillin injection, other medication and scheduled for gastrointestinal x-rays the following day. Before the x-rays could be taken, Ralph complained of acute abdominal pain and was brought to the emergency room of the local hospital on the appellee’s direction. Diagnosis by ap-pellee was that of a perforated viscus (abdominal organ) most probably perforated peptic ulcer or possibly as a consequence of recurrent aggravation of existing ulcerative colitis. Ralph was immediately transferred to Presbyterian Hospital in Oklahoma City for surgical consultation with Dr. M.

Dr. M. examined Ralph and after a standard physical, blood work and other tests, concluded that cause of Ralph’s condition was acute appendicitis with probable rupture and attendant peritonitis. Immediate surgery confirmed Dr. M.’s diagnosis and resulted in an additional finding. Ralph had adenocarcinoma, cancer of the colon. Dr. M. removed Ralph’s appendix and treated him for the peritonitic infection. Ralph’s post-operative recovery was “reasonably smooth”, and he was discharged to continue his convalescence at home.

The convalescence was not “smooth”. Ralph experienced an intestinal obstruction and was readmitted for further surgery in less than a month. At this time, Dr. M. removed Ralph’s colon entirely in an attempt to check the spread of the cancer, and found no evidence of peritonitis. This surgery and subsequent surgical andsmedical treatment were not successful in arresting the spread of the cancer. More than a year and a half later, Ralph died. In answer to the following question: “Is it possible that due to the ruptured appendix and peritonitis, is it possible that this could have spread the cancer cells to other parts of the abdomen; namely, to the colon?”, and Dr. M. said “No”. There is absolutely no evidence to indicate the cause of death was anything other than adenocarcinoma, cancer of the colon.

All the foregoing facts were presented to the trial court in the depositions and other evidentiary materials submitted in conjunction with appellee’s motion for summary judgment. Appellant offered no other evidence on her behalf. In Weeks v. Wedge-wood Village Inc., Okl., 554 P.2d 780 (1976), we said:

“ * * * Rule 13 is intended to permit a party to pierce the allegations of the pleadings to show that the facts are otherwise than alleged.
If the defendant introduces evidence which indicates there is no substantial controversy as to a fact material to plaintiff’s cause of action, and this fact is in the defendant’s favor, the plaintiff has the burden of showing that evidence is available which would justify trial of the ■issue.”

Appellant’s failure to put on evi-dentiary materials of her own does not necessarily preclude her from demonstrating that an actual controversy exists as to a material fact issue in the case. The party against whom the motion for summary judgment is directed can always show through the movant’s own evidence the existence of controverted fact issues. This is precisely what appellant attempted to do in the case at bar. Using appellee’s deposition and clinical notes, appellant documented the diagnostic procedures employed in examining Ralph upon the advent of his abdominal complaints. Using the deposition of Dr. M. appellant documented the diagnostic procedures employed by the surgeon upon receiving Ralph as a patient. The diagnostic approaches of the two physicians dif *1335 fered. Dr. M. as it turns out, correctly diagnosed Ralph’s condition as acute, probably ruptured, appendix, while appellee thought the condition was recurrence of pre-existent ulcerative colitis or peptic disease. Appellant, in effect, argues that since the evidence shows that appellee incorrectly diagnosed Ralph’s illness as a recurrence of his ulcerative colitis condition, the evidence would necessitate reversing the trial court’s summary judgment ruling and submission of the cause to the jury. It does not necessarily follow from the evidence presented that Dr. M.’s correct diagnosis was the product of a proper adherence to the prevailing professional standard of care or that appellee’s diagnostic procedures were below the prevailing professional standard of care.

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Bluebook (online)
1978 OK 84, 584 P.2d 1332, 1978 Okla. LEXIS 412, Counsel Stack Legal Research, https://law.counselstack.com/opinion/parsons-v-wood-okla-1978.