Norris v. Industrial Commission

61 P.2d 413, 90 Utah 256, 1936 Utah LEXIS 16
CourtUtah Supreme Court
DecidedOctober 22, 1936
DocketNo. 5657.
StatusPublished
Cited by58 cases

This text of 61 P.2d 413 (Norris v. Industrial Commission) is published on Counsel Stack Legal Research, covering Utah Supreme Court primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.

Bluebook
Norris v. Industrial Commission, 61 P.2d 413, 90 Utah 256, 1936 Utah LEXIS 16 (Utah 1936).

Opinion

WOLFE, Justice.

Certiorari to the Industrial Commission to review an order denying compensation to lone W. Norris, widow of Thomas E. Norris, the latter having died on January 13, 1934, from what the attending physician diagnosed as occlusion of the coronary artery. On December 9, 1932, while working for the Publix Theatres Corporation, Norris fell and among other severe injuries sustained a fracture of the right leg near the hip joint and injuries about the right knee. After removal of a cast extending from the ankle to the hip, which had been on for nine weeks, the right leg began to swell, accompanied with pains in the right knee. Norris for the next year and until his death suffered from this swelling. There is no dispute that it was caused by a thrombosis of the longus saphenous vein (one of the two large superficial veins of the leg), probably caused by an irritation of the vein, in turn probably caused by the accident or by the cast made necessary by the accident. Norris obtained compensation for total temporary disability and obtained an award for permanent partial disability of the right leg for 52-14 weeks. There is no dispute involving these awards. The dispute arises on account of what subsequently happened. On January 3, 1934,, about one year and one month after the accident, Norris was taken to the hospital suffering with what was diagnosed as a fecal impaction with acute appendicitis. He was operated on and was apparently making good recovery until the morning of January 13th, when he was suddenly stricken with what *258 the doctor diagnosed as a beginning thrombosis of the coronary artery, caused, it was surmised, by emboli. On the afternoon of January 13th he died. The attending physician gave in his report as the cause of death,

“Coronary artery occlusion; myocardial degeneration. Other contributing causes of importance, operation chronic appendicitis January 3rd, 1934; condition for which performed fecal impaction.”

More accurately used, an occlusion seems to be the general term for a closing of the vein or blood vessel to the passage of the blood. It may be caused by a thrombosis or an embolus; a thrombosis, the plugging of a blood vessel through the formation of a clot or obstruction on the walls of the vessel or vein; an embolus, a plug which floats along until it becomes lodged so as to obstruct the passage of the blood. It consists usually of a clot of fibrin, a shred from a morbid growth, a globule of fat, air bubbles, or a microorganism. Thrombosis and occlusion are often used interchangeably because a thrombus causes occlusion. An em-bolus or floating particle by attaching itself or becoming wedged may form a thrombosis or occlusion, but ordinarily a thrombus is caused by an inflammation of the walls of the vessel. In this case the cause of the death being conceded to be an occlusion of the coronary artery, the question was: Was it caused by an embolus which broke from the thrombosis of the longus saphenous vein of the right leg or from the appendicitis operation, or from some other cause? If from the first, the death would be traceable to the accident and the defendants would be liable. If from the second or third causes, it would not be traceable to the accident, at least with sufficient conviction to rise to the dignity of proof.

The Commission found that the death was caused by “coronary artery occlusion and myocardial degeneration. The contributing cause of importance was operation for chronic appendicitis on January 3rd, 193U.” (Italics ours). On rehearing the decision was affirmed, but the findings did not include the specific sentence above italicized, but *259 stated that it “was not caused by any accident or injury arising out of or in the course of the employment with the Publix Theatres Corporation, as heretofore found in its Findings of April 24th, 1934.”

The applicant founds her appeal on the familiar contention that the “uncontradicted testimony clearly shows that said Norris died as a result of the injuries received on December 9th, 1932, while in the course of his employment and that the decision of the Industrial Commission is contrary to and not sustained by [any substantial] evidence.” To this we cannot accede. A reading of the record convinces us that the commission might well have found that the death was caused by the accident. We are not prepared to say that the record does not show that the more probable cause of the coronary occlusion was the breaking away of an embolus from the thrombosis of the longus saphenous vein of the right leg rather than that it originated from the area of the appendix, but it is not our province to measure the relative probabilities. That is for the commission. Moreover, in its decision on the rehearing, the commission did not specify that the contributing cause of importance was the operation for chronic appendicitis of January 3, 1934. It might have concluded that there was not sufficiently definite proof that the coronary occlusion was caused by either an embolus from the leg or the region of the appendix. It was testified to by the doctors that in both cases to reach the coronary artery, emboli from either region would have to go from the veins to the right auricle, thence to the right ventricle, thence through the pulmonary artery (which carries venous blood) to the lungs, thence to the left auricle, left ventricle, through the aorta to the coronary artery; that it was an exceedingly rare thing, if not impossible, for an embolus to do this, as they were almost without exception filtered out by the lungs.

*260 *259 The commission may have concluded that the coronary occlusion was caused by a thrombosis originating in the coronary artery; that is, that a thrombus or clot became *260 organized by some local cause in this artery. There was conflicting expert testimony on the question of the relative probability of the cause being an embolus from the leg or from the region of the iliac fossa (lower right quadrant of the abdomen). The brief of plaintiff is devoted to demonstrating the lack of credibility to be given to the opinion of the doctors for the defendants in order to show it to be unsubstantial evidence. But the evidence of Dr. Hummer cannot be called unsubstantial. Again, therefore, we have the old case of a conflict of evidence which it is for the commission to resolve. It may be well to sum up the principles laid down in a number of previous cases regarding the jurisdiction of this court over awards and orders of the commission.

The Legislature has, in effect, said:

“The Commission is the final arbiter of the facts. If there is error in judgment or conclusions of or from facts, it must be the Commission’s error and remain there. We give the Supreme Court the right to speak only by warrant of law in compensation cases when it speaks in reference to errors of law alleged to have been made by the Commission.”

An apparent exception is on the finding by the commission of jurisdictional facts. On these this court examines the evidence to see whether it clearly preponderates against the conclusions of the commission.

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Bluebook (online)
61 P.2d 413, 90 Utah 256, 1936 Utah LEXIS 16, Counsel Stack Legal Research, https://law.counselstack.com/opinion/norris-v-industrial-commission-utah-1936.