Moe v. Springfield Milling Corp.

394 N.W.2d 582, 1986 Minn. App. LEXIS 4880
CourtCourt of Appeals of Minnesota
DecidedOctober 21, 1986
DocketC0-86-660
StatusPublished
Cited by6 cases

This text of 394 N.W.2d 582 (Moe v. Springfield Milling Corp.) is published on Counsel Stack Legal Research, covering Court of Appeals of Minnesota primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.

Bluebook
Moe v. Springfield Milling Corp., 394 N.W.2d 582, 1986 Minn. App. LEXIS 4880 (Mich. Ct. App. 1986).

Opinion

OPINION

SEDGWICK, Judge.

Norman and Genevieve Moe appeal from a summary judgment dismissing their personal injury claims against Springfield Milling Corporation for dioxin poisoning traceable to a cattle feed supplement. The trial court decided the absence of an established dioxin level in humans and appellant’s failure to show with reasonable medical certainty a dioxin-related injury to Moe removed all questions of material fact. We believe a causal connection establishing a question of material fact has been demonstrated regardless of the nature of dioxin’s background level in the human population and reasonable medical certainty is not required at the pre-trial stage. We reverse and remand for trial.

FACTS

Appellants commenced this action on October 31, 1977, claiming that a feed supplement purchased from Springfield Milling Corp. was toxic and harmed their personal health and the health of their cattle. Appellants seek compensation for property damage and personal injury. Following hearing on respondents’ motion for summary judgment, the trial court dismissed the claims against Hubbard Milling Co. with prejudice. The personal injury claim against Springfield Milling was also dismissed with prejudice and final partial summary judgment was entered. The Moes’ appeal the personal injury summary judgment.

Norman Moe is a successful farmer who raised cattle. In late 1973, appellant used a protein supplement formulated and produced by respondent, Springfield Milling Corporation. The supplement was mixed with homegrown feed. The cattle did not thrive on the mixture. Moe noticed most cattle experienced drastically reduced weight gain in comparison to normal levels on his feedlot. Twenty-five head also died on a feedlot where cattle losses were rare. The vets could not determine the cause. After experimenting on his own, Moe decided the feed supplement was responsible and discontinued its use.

After his “resolution” of the feed supplement dilemma, Moe had some cattle slaughtered for personal consumption. Moe claims that after eating the meat he experienced “fever, high blood pressure, slow healing skin sores, chest pains, abdominal cramps, diarrhea, and lack of ability to concentrate.” Moe continues to exhibit some symptoms. His wife experienced some health problems but not to the same degree or extent.

Appellant claims the feed supplement was responsible not only for the cattle’s problems, but his own. Moe states that the feed killed rats and mice and that family pets fed meat scraps sickened and died. Initially, a contaminant such as PCB’s was suspected as the “poison” but tests failed *584 to reveal dangerous concentrations of those toxic elements.

Further tests revealed the presence of 2, 3, 7, 8 TCDD (dioxin) in the feed as well as the meat. The feed tested at 13 parts per trillion (ppt). The meat was analyzed at 18 ppt in a hamburger sample, while 415 and 431 ppt were found in a piece of fat. Tests were also conducted on fat samples taken from Norman Moe. A test made by Wright State University (Ohio) found 10 ppt; the University of Nebraska discovered 15 ppt and 30 ppt.

Expert testimony exists to the effect that Mr. Moe’s symptoms are consistent with dioxin exposure.

Professor Jeffrey Stevens of the University of Minnesota found:

3. That the detection of 13 parts per trillion of TCDD in the feed supplement is a positive finding. That such a level of TCDD in the feed supplement can produce a much higher level of TCDD in cattle which consume the feed supplement, and is therefore consistent with the detection of 415 parts per trillion in a beef sample. That it could be expected that an even higher level of TCDD would be found in the tissue of humans who consumed such beef. That TCDD is an extremely toxic element and its effect on human health would be consistent with symptoms exhibited by Norman Moe, including the diagnosis of an immune deficiency problem.

Dr. Paul Johnson of Occupational Health Services at St. Paul-Ramsey Medical Center stated:

My impression is that we have strong evidence that Mr. Moe was exposed to meat which was contaminated with 2, 3, 7, 8 TCDD. This is substantiated by the presence of measurable levels of this substance in cattle feed and the meat from these cattle which he states that he subsequently consumed. * * *
******
Mr. Moe also relates a history that is certainly suggestive of clinical problems consistent with exposure to 2, 3, 7, 8 TCDD. The many subjective symptoms which he complained of, the fatty liver changes noted on two biopsies, and the abnormalities noted on EMG all are consistent with problems that could be expected in a person exposed to this substances.
******
In summary I feel that Mr. Norman Moe has had exposure to beef which was contaminated with 2, 3, 7, 8 TCDD. This exposure has been documented. Mr. Moe has manifested health problems in the past which are consistent with human exposure to this substance.

An allergist also believed Moe’s illness was consistent with exposure to “some toxic chemical.”

Moe has a history of past health problems. Some of the symptoms he experienced after eating the meat had been experienced on prior occasions because of other chemical exposure. Some medical reports suggest a psychosomatic basis for his symptoms. Dr. Johnson touches on these problems as well as the general nature of his complaints.

The problem in this case is that although the exposure seems definite and documented, all of Mr. Moe’s health complaints and abnormal findings which have been documented to this time, are extremely nonspecific, that is; although they could be produced by this specific chemical substance, they also could be manifestations of many other medical conditions.

There is no testimony as to symptoms experienced by Mrs. Moe and no tests or expert testimony substantiating her exposure to any chemical.

ISSUE

Did the trial court err in granting respondent summary judgment on the personal injury claims?

ANALYSIS

Pursuant to Minn.R.Civ.P. 56.03 respondent was granted a summary judg *585 ment dismissing, with prejudice, the Moes’ personal injury claims. Summary judgment is proper when no genuine issue arises as to a material fact. Normally, summary judgment should not be granted with regard to questions of negligence. Kaczor v. Murrow, 354 N.W.2d 524, 525 (Minn.Ct.App.1984). Negligence issues, such as proximate cause, are “usually a question of fact and seldom can be disposed of on a motion for summary judgment.” Hamilton v. Independent School District No. 114, 355 N.W.2d 182, 184 (Minn.Ct.App.1984). Such factual questions, deducing causation for example, are the province of the factfinder, the jury. Id. at 185; Illinois Farmers Insurance Co. v. Tapemark Co.,

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Bluebook (online)
394 N.W.2d 582, 1986 Minn. App. LEXIS 4880, Counsel Stack Legal Research, https://law.counselstack.com/opinion/moe-v-springfield-milling-corp-minnctapp-1986.