Malanga v. Royal Indemnity Company

418 P.2d 396, 4 Ariz. App. 150
CourtCourt of Appeals of Arizona
DecidedJanuary 18, 1967
Docket2 CA-CIV 135
StatusPublished
Cited by7 cases

This text of 418 P.2d 396 (Malanga v. Royal Indemnity Company) is published on Counsel Stack Legal Research, covering Court of Appeals of Arizona primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.

Bluebook
Malanga v. Royal Indemnity Company, 418 P.2d 396, 4 Ariz. App. 150 (Ark. Ct. App. 1967).

Opinion

KRUCKER, Chief Judge.

This is an appeal from a judgment denying recovery in a suit for death benefits under a policy insuring against losses “ * * resulting directly and independently of all other causes from accidental bodily injuries * *

The insured under the policy was John S. Ellis, who died on March 16, 1963, during the term of the policy, from an over-ingestion of barbiturates and alcohol. His widow, Mary M. Ellis Malanga, was the beneficiary under the policy for death benefits. The death benefits provided for in the policy were in the sum of $30,000 for which an annual premium of $37,50 was charged.

The facts leading up. to the death of the deceased, viewed favorably, to support the judgment below, are as follows. The deceased was thirty-two (32) years of age at the time of his death. Excessive use of alcohol had led to marital problems with his wife, the plaintiff below and the appellant here. He separated from his wife in January, 1963, and a divorce case was pending at the time of his death. For approximately two months prior to his death he had abstained from drinking alcohol, but on the evening in question he. had been drinking. He had been examined by the family physician nine days before his death and was found to be in excellent health, with the exception of being somewhat weak from dieting and not eating because of worrying about matrimonial problems. On the evening in question, he came to the home of his estranged wife where he collapsed. The family physician was called, the deceased was taken to his own abode and placed in bed, where he was later found dead. In his pockets were two tablets of sodium amytal.

The family physician testified that he had never prescribed any barbiturates or other drugs for the deceased, that the deceased considered taking medication a sign of weakness, and that as far as the witness knew the deceased had no other attending physician.

Blood tests taken after death indicated the presence of ethyl alcohol and sodium amytal in the bloodstream in such quantities'that *152 neither one alone would ordinarily cause death but in such quantities that together they could have been the cause of the death. Autopsy revealed ho traumatic injuries that could have brought about death, and it was the opinion of both the family physician and the pathologist who performed an autopsy that the ingestion of alcohol and barbiturates combined were the cause of death.

The pathologist testified in part:

“Any type of drugs or medication or liquid of this type produces essentially a common factor. The common factor here with any of these agents is central nervous system depression so that you have each one individually acting as a central nervous system depressant. Now when you take these in various forms or multiple forms, for example with barbiturates, with tranquilizers and so on, you, what we call compound the effect. You not only have the action of the one; you have a total composite action of the many. You can take actually lower doses of any of these agents, but the cumulative effect is the same as if one massive dose had been taken.
* * * * * *
“So these agents in large doses are toxic. This is true of all drugs. The primary site of action is the central nervous system. It is a target organ. It causes a depression of the brain stem. In the brain stem are two important centers, one that controls respiration and one that controls heart rate. This is why we can breathe and ■ our heart will beat automatically. Whether you like it or not, you breathe, your heart beats. These are called automatic. You don’t have to think about this to have it happen. Now when you start raising the level of either one of these two agents, these centers which normally have an automatic regulated stimulus, it becomes interfered with so that the stimulation that normally occurs becomes depressed as the level of these agents goes higher and you can eventually completely remove the stimulus by these agents so there is no stimulation to respiration and there would be no stimulation to heartbeat so that death then occurs by depressing these areas sufficiently so that the heart rate would be slow enough or the respiratory center would be slow enough so that the rate of breathing or the heartbeat rate would not be sufficient to sustain life.
❖ * * * * *
“Q So when we talk about this particular death, we are talking about a man who took enough alcohol and barbiturates into his system as to slow down the function of the central nervous system to a point where his heart stopped beating, is that essentially it, Doctor?
“A Yes. Yes.
“Q Now was this cause of his death,, in your opinion, traumatic in nature ?
“A No, sir.
“Q There was nothing traumatic that you found in your examination which, in-your opinion, contributed to his death ?
“A . That’s correct.
“Q In your opinion, was his death due to a bodily injury ?
“A ’No, sir.
‡ # ■ # . . -‡ :|c ?}c
“Q But the barbiturate and - alcohol acting together in the case become poisonous, is that what you are telling us ?
“A No, I don’t like the term poisonous. I am trying to separate for you, as best I can, poisons from medications. I think I have to because I think this is a big point. We are not talking about poisons here. This is a prescribed type medicine. Alcohol is prescribed as a medicine and it is good in a specific amount, so I can’t.use the word poison for this type of agent because over here I have poisons that we see every day in town that in and by themselves, when ingested, produce toxic effects and may produce death. These do not produce toxic or poisonous effects. It is more or less a depression that results from internal cellular changes. I can’t answer this is a poisonous change. We don’t see this cellular change in the laboratory. It is something that we theorize must occur.” (Emphasis added.)

*153 Pertinent provisions of the policy in question are as follows:

“This insurance is against loss indicated as covered in the Schedule of Benefits resulting directly and independently of all other causes from accidental bodily injuries sustained during the term of the policy, herein referred to as such injury, subject to all the provisions, exceptions, limitations and reductions of the policy.
******
“Part II Death Benefit
“If within ninety days after the date of accident, if the Insured is not continuously totally disabled, or within two hundred weeks after the date of accident and during a period of continuous total disability for which weekly indemnity shall have been payable under Part I of this policy, such injury

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Bluebook (online)
418 P.2d 396, 4 Ariz. App. 150, Counsel Stack Legal Research, https://law.counselstack.com/opinion/malanga-v-royal-indemnity-company-arizctapp-1967.