Leo v. Long Island Railroad

307 F.R.D. 314, 2015 U.S. Dist. LEXIS 56953, 2015 WL 1958906
CourtDistrict Court, S.D. New York
DecidedApril 30, 2015
DocketNo. 13cv7191 (MHD)
StatusPublished
Cited by8 cases

This text of 307 F.R.D. 314 (Leo v. Long Island Railroad) is published on Counsel Stack Legal Research, covering District Court, S.D. New York primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.

Bluebook
Leo v. Long Island Railroad, 307 F.R.D. 314, 2015 U.S. Dist. LEXIS 56953, 2015 WL 1958906 (S.D.N.Y. 2015).

Opinion

MEMORANDUM & ORDER

MICHAEL H. DOLINGER, United States Magistrate Judge.

Plaintiff Brian Leo commenced this lawsuit under the Federal Employers Liability Act (“FELA”), 45 U.S.C. § 51 et seq., seeking recovery for physical and other injuries suffered while employed by the defendant Long Island Railroad Company (“LIRR”). Following trial, a jury returned verdicts finding defendant liable and awarding plaintiff a total of $3,189,122.64 in past and future damages.

Following entry of judgment for that amount, defendant has moved for a new trial or a remittitur of portions of the damages award. Plaintiff has opposed. For the reasons that follow, defendant’s motion is granted in part.

FACTUAL AND PROCEDURAL BACKGROUND

I. The Pertinent Trial Evidence

A. The Accident

Mr. Leo was employed by the LIRR as an assistant signalman. On November 2, 2011 he was working to install innerduct1 under a platform at the Kew Gardens station in Queens. While crawling on hands and knees under the platform, he encountered a substantial pile of large broken pieces of concrete, apparently rubble from a pre-existing platform that had never been removed. As he crawled over the broken concrete, a heavy piece of it dislodged and fell onto his right wrist and arm, trapping him in that position. He attempted to pull his hand from under the concrete but was unable to do so. Shortly after, a fellow worker pulled the concrete slab away, freeing his arm. (Tr. 137-38, 142-55, 251, 373).

[317]*317B. Medical Evidence

Mr. Leo remained at the worksite until the end of his shift. That evening, however, encountering stiffness and swelling, he visited the emergency room at Good Samaritan Hospital, where an X-ray of his wrist showed no fracture. (Id. at 155-56; Pl.’s Ex. [“PX”] 23 at pp. 1-6 [Record of Nov. 2, 2011 visit to Good Samaritan Hospital]).

The next day plaintiff went to the LIRR medical facility and was taken off work. (Tr. 157; see also PX 26 pp. 27 [LIRR Medical Center receipt dated Nov. 3, 2011], 74-75 [Medical notations for Nov. 3, 2011 visit to LIRR Medical Center]). Two days later he consulted Dr. Arthur Pallotta, an orthopedic surgeon to whom he had been referred by the hospital. The doctor observed swelling, tenderness and abrasions on the wrist as well as sensitivity on the median nerve.2 He diagnosed a “sprain/crush” injury and put a splint on the arm. (Tr. 50-54,157; PX 13 p. 5 [Medical chart dated Nov. 4, 2011]).

On November 28, 2011—nearly four weeks after the accident—Dr. Pallotta found some swelling and tenderness over the back of the wrist. (Tr. 54-55). On December 16, 2011, he again saw plaintiff and noted continuing median-nerve sensitivity and also observed that the ring and small fingers of the hand hung further down than the normal cascade of the fingers on the hand, a phenomenon known as clawing. (Id. at 55-57, 64). Noting some weakness in the first dorsal interos-sei and loss of sensation in the affected fingers, Dr. Pallotta suspected possible ulnar-nerve injury3 and ordered an electromyelo-gram (“EMG”) and nerve conduction study.4 The results of these tests were normal. (Id. at 57-64; see also PX 13 pp. 6 [Medical chart dated Nov. 28, 2011], 12 [Letter to LIRR Medical Department from Dr. Pallotta signed Nov. 28, 2011]).

Dr. Pallotta saw plaintiff again on March 7, 2012. (Tr. 64). Mr. Leo reported little improvement, and the doctor again found decreased sensation in the fingers and continued clawing. He also observed increased sensitivity in the ulnar nerve and diagnosed a right wrist sprain/crush injury with median- and ulnar-nerve dysfunction, as well as neuritis.5 (Id. at 64-66; see also PXs 13 p. 8 [Medical chart dated Mar. 7, 2012] & 15 pp. 7-8 [Results of test dated Feb. 1,2012]). On plaintiffs next visit, on April 4, 2012, the doctor observed increased drooping of the two affected fingers. (Tr. 66). He found that sensation in the two affected fingers had decreased “a little bit”, that the first dorsal interossei muscle was “slightly weak”, and that another muscle innervated by the ulnar nerve—the flexor digitorum profundus—“appeared to be weak, as well.” (Id. at 66-67; see also PX 13 p. 9 [Medical chart dated Apr. 4. 2012]).

In connection with the April 4 visit, Dr. Pallotta ordered another nerve-conduction study. That study was inconclusive as to whether the symptoms were attributable to an ulnar-nerve injury, as the doctor had originally surmised. It did show, however, a right c8-tl radiculopathy6 and spontaneous activity at the right opponens pollieis muscle. (Tr. 67-68; PXs 13 p. 9 & 15 pp. 4-6 [Results of test dated May 11, 2012]). As [318]*318Dr. Pallotta explained these findings, they could indicate that the source of the injury was located at a point in the nerve system above the forearm and elbow, and that a lack of innervation, whether of the median nerve or the ulnar nerve, might trigger these findings. (Tr. 68-72). Further, the doctor noted that such injury to the nerve could have been caused by an excess of pressure on the nerve, for example if the arm is pulled too high over the head. (Id. at 72-73).

On a May 23, 2012 visit, Dr. Pallotta found increased atrophy and weakness of the first dorsal interossei. He also noted decreased sensation in the fingers. (Id. at 73-74; PX 13 p. 1 [Medical chart dated May 23, 2012]). These findings were at least consistent with injury to the ulnar nerve or the brachial plexus, which feeds into the ulnar nerve. (Tr. 74; see also id. at 72).

Because of his concern that the injury might originate in the brachial plexus, Dr. Pallotta referred plaintiff to a Dr. Patrick Reid, a neurosurgeon. (Id. at 74-76). Dr. Reid opined that the injury was traceable to the posterior interosseus nerve, which is responsible for the extension of the fingers. (Id. at 75-76; PXs 16-17 [Medical records from Drs. Patrick Reid & Joseph Fein-berg]).

Dr. Pallotta disagreed with that assessment based on a subsequent examination, on October 23, 2012, when he tested the strength of muscles innervated by the posterior interosseus nerve. (Tr. 76-78).7 On that visit he also observed increased clawing of the two affected fingers. (Id. at 77). He then referred plaintiff to a brachial plexus specialist, a Dr. Christopher Winfree. (Id. at 78-79). Dr. Winfree diagnosed dysfunction of the ulnar nerve, probably at the wrist. (Id. at 79-80; PX 20 pp. 1-3 [Letter dated Dec. 10, 2012 from Dr. Winfree to Dr. Pallot-ta]).

Dr. Pallotta next saw plaintiff on January 2, 2013. He observed a further worsening in the clawing. As for sensation, it had marginally improved. (Tr. 81). As Dr. Pallotta explained, plaintiffs effort to pull his arm out from under the concrete slab may well have injured his brachial plexus, leading to the symptoms that he observed. (Id. at 82-83).

Dr. Pallotta saw Mr. Leo again on March 8, 2013 and in July 2013, with similar results. (Id. at 83-85; PX 13 p. 2 [Medical chart dated Mar. 8, 2013]).

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Bluebook (online)
307 F.R.D. 314, 2015 U.S. Dist. LEXIS 56953, 2015 WL 1958906, Counsel Stack Legal Research, https://law.counselstack.com/opinion/leo-v-long-island-railroad-nysd-2015.