Kalsbeck v. Westview Clinic, P.A.

375 N.W.2d 861, 1985 Minn. App. LEXIS 4748
CourtCourt of Appeals of Minnesota
DecidedOctober 22, 1985
DocketC1-85-446
StatusPublished
Cited by15 cases

This text of 375 N.W.2d 861 (Kalsbeck v. Westview Clinic, P.A.) is published on Counsel Stack Legal Research, covering Court of Appeals of Minnesota primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.

Bluebook
Kalsbeck v. Westview Clinic, P.A., 375 N.W.2d 861, 1985 Minn. App. LEXIS 4748 (Mich. Ct. App. 1985).

Opinion

OPINION

RANDALL, Judge.

In this medical malpractice action, plaintiff appeals from the trial court’s order denying a new trial and from the judgment entered following a jury verdict for defendant. Appellant contends that the trial court erred in refusing to give certain requested jury instructions, and that the verdict was not supported by the evidence. We affirm.

FACTS

Appellant’s decedent, her husband Thomas Kalsbeck (Kalsbeck), died on January 27, 1983, after a short illness. He was 47 years old. While the direct cause of death was cardiac arrest, the death certificate and the attending physician’s death summary indicate the following final diagnosis:

1. Overwhelming pneumonia, penicillin-resistant staphylococci;
2. Diabetes mellitus, out of control;
3. Respiratory arrest, secondary to No. 1 and No. 2;
4. Cerebral anoxia and resulting coma, secondary to No. 3;
5. Repeated cardiac arrests secondary to Nos. 3 and 4.

The events of the few days preceding Kalsbeck’s death are as follows: Kalsbeck and appellant were examined by their family doctor, respondent Dr. James Haight, on January 24, 1983, a Monday. Both Kals-beck and appellant had been ill since the previous Thursday. Appellant described their symptoms as including sore throats, coughing, and a general aching “down to the bone.” By Monday, Kalsbeck was suffering more than appellant. He was congested, coughing, and had even coughed up some blood that morning. His right side was painful, making it difficult to cough.

According to Dr. Haight, however, Kals-beck did not look “that sick,” and the doctor was surprised when x-rays showed pneumonia. Dr. Haight testified that his impression at the time of the office visit was that Kalsbeck’s pneumonia was probably viral rather than bacterial. Pneumonia caused by a viral organism cannot be treated effectively by antibiotics or medication of any kind. Dr. Haight favored viral pneumonia because Kalsbeck’s white blood count was normal. This count is usually elevated with bacterial pneumonia. However, the blood test also disclosed a “shift to the left” of the differential count, which caused Dr. Haight to believe it was possible the pneumonia was bacterial. He therefore decided to treat Kalsbeck with two antibiotics — Bicillin, which was injected in the office, and Ampicillin, which was prescribed. These two antibiotics are ordinarily effective against approximately 80-90% of the bacteria which cause pneumonia. They are not effective against most types of staphylococcal bacterial pneumonia (staph), which was the type of pneumonia that was ultimately diagnosed after Kalsbeck was admitted to the hospital, and which is listed on the death summary as the primary cause of death.

Viral pneumonias account for approximately 50% of all pneumonias. Of the bacterial pneumonias that make up the remaining 50%, only 1-3% are staph. That figure may actually be even lower for patients such as Kalsbeck, since staph acquired by *864 already hospitalized patients constitutes most of its occurrences. In fact, of the 4 family practitioners who gave expert testimony, only one had ever diagnosed a case of non-hospital acquired staph, even though each of them had at least 10 years experience. Dr. Haight had never seen a patient with staph, and he testified that it never occurred to him that Kalsbeck might have staph.

Expert testimony established that the incidence of staph increases following a bout of influenza. While one testifying doctor stated that he believed that the Kalsbecks had influenza, other experts disputed his conclusion, and testified that the illness could have been the common cold. In any case, Dr. Haight testified that he was not aware of the increase of staph following influenza.

An additional and essential element of Kalsbeck’s condition is that he was diabetic. He contracted diabetes only 3 years earlier, at the age of 44, and was therefore a Type II, or adult-onset diabetic. Type II diabetics have residual pancreatic function, and so their bodies continue to produce some insulin. These diabetics may or may not require additional insulin to supplement their natural supply. Type I, or juvenile diabetes, is acquired in childhood and involves an absolute absence of insulin. These diabetics have no reserve capacity to produce insulin, and they react quickly and strongly to excess insulin as well as to the lack of insulin. Their blood sugar levels are usually more volatile than those of a Type II diabetic.

Kalsbeck was an insulin dependent Type II diabetic. This may have been at least partially due to his mild obesity, which adds stress to the body and can cause the diabetic’s insulin requirement to increase. If Kalsbeck had been within the normal weight limits for his age, he might not have needed insulin.

A primary consequence of diabetes is a reduced ability to fight infections. Also, the presence of an infection aggravates a diabetic condition, making it more difficult to control. The most typical aggravating result of an infection in a diabetic is an increase in the blood sugar. Once the blood sugar is elevated, the resistance to infection decreases even further. Thus, the relationship between diabetes and infection is circular. When a diabetic has an infection they must monitor their condition more closely than they do on a regular basis, and must make an extra effort to keep their blood sugar level down.

As most diabetics do, Kalsbeck monitored his diabetes at home, using chemical strips which test the amount of sugar being spilled over into the urine from the blood. The test does not give an accurate reading of the blood sugar level, since each diabetic has a different “spill threshold,” i.e., they spill sugar into the urine at blood sugar levels which differ among diabetics. Nevertheless, for years it was the only practical way for diabetics to have any idea of their blood sugar levels without visiting a medical laboratory. In just the last few years, home blood testing kits became available so that many diabetics have now abandoned the less accurate urinalysis method.

The facts are not clear on how well controlled Kalsbeck’s diabetes was. He had not been hospitalized except when he was initially diagnosed, and he rarely missed a day of work because of illness. However, he did not regularly inform Dr. Haight of the results of his urine tests, and he resisted the doctor’s recommendation that he come in every 3 months to have a blood sugar taken. The few times he did come in to the office, his blood sugar was often elevated. The normal range for blood sugar is 70-120 mg. percent. On one occasion, Kalsbeck’s blood sugar was 400, and another time it was 188. Dr. Haight testified that Kalsbeck was “basically fairly well controlled,” and “reasonably well controlled.” Again, one difficulty in determining whether a diabetic is in good control is that even consistently elevated blood sugars do not always noticeably affect their general feeling of well being and good health, (particularly in Type II diabetics.) Here, without a more detailed history of *865 Kalsbeck’s blood sugar levels, it is impossible to know how well he managed his disease.

When Dr.

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Bluebook (online)
375 N.W.2d 861, 1985 Minn. App. LEXIS 4748, Counsel Stack Legal Research, https://law.counselstack.com/opinion/kalsbeck-v-westview-clinic-pa-minnctapp-1985.