Inland Steel Co. v. Johnson

439 S.W.2d 562, 1969 Ky. LEXIS 372
CourtCourt of Appeals of Kentucky
DecidedMarch 7, 1969
StatusPublished
Cited by8 cases

This text of 439 S.W.2d 562 (Inland Steel Co. v. Johnson) is published on Counsel Stack Legal Research, covering Court of Appeals of Kentucky primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.

Bluebook
Inland Steel Co. v. Johnson, 439 S.W.2d 562, 1969 Ky. LEXIS 372 (Ky. Ct. App. 1969).

Opinion

PALMORE, Judge.

Leo Johnson died of coronary thrombosis while working in the appellant company’s coal mine. A workmen’s compensation claim by his widow and children culminated in a dismissal based on a finding by the board that they had failed to prove “that the decedent’s work activities caused or brought about the coronary thrombosis which resulted in his death.” The company appeals from a judgment of the Knott Circuit Court setting aside the final order of the board and remanding the case for further consideration.

The circuit court found as a fact that there was a partial causal relationship between Johnson’s work and his death. Though we might readily agree with that conclusion, in order to sustain it we must find that the evidence was of such persuasive force that it was clearly unreasonable for the board not to be convinced by it. Lee v. International Harvester Company, Ky., 373 S.W.2d 418, 420 (1963).

At the time of his death Johnson was 47 years old. He had been a coal miner all of his adult life and had been employed by the appellant company for 17 years. According to his widow he had. never lost any time from work by reason of disability except for a brief period of hospitalization for a hernia operation. She may, however, have been mistaken in this respect, because the record shows that in 1957 he was in the hospital for six days with pain in the left lower chest, which was finally diagnosed as pleurisy, and in 1960 spent several days in the hospital with a fever of unknown etiology and acute rheumatoid arthritis of the right thumb and index finger.

Johnson was stricken suddenly and without previous warning just after midnight of February 1-2, 1965, and within 15 to 30 minutes after he had beaten a large stone to pieces with a 16-pound sledgehammer. He complained of “hurting all over,” particularly in his arms, and after resting for a few minutes attempted to [563]*563resume his regular task of operating a mine car but almost immediately suffered another or further seizure and fell off the car to the side of the tracks. He was dead within moments. An autopsy disclosed a fresh thrombotic occlusion of the right coronary artery, evidence of a pre-existing arterio-sclerotic condition, and an old myocardial infarction adjacent to the left coronary artery. There was no evidence that he had consciously experienced any previous symptoms of heart disease unless the condition for which he was hospitalized in 1957 actually was a heart attack (as it probably was) instead of pleurisy.

The autopsy was performed by Dr. W. C. Morris, a pathologist, of Pikeville, Kentucky. After describing the clinical details, he was asked whether “overexertion or hard work immediately before he had the coronary would have brought about the coronary more suddenly,” to which he replied as follows: “That is entirely possible, yes. In a heart case, as we term a heart case that has had previous heart trouble, or is under treatment, has more restricted activity. That is the general practice.” He further testified as follows:

Q- “Do I understand you to say it is quite possible that the work of the employee or the overexertion could have possibly caused the coronary occlusion and the death here of this claimant ?”
A- “Yes, that is entirely conceivable. In other words, I have never had any symptom of any previous trouble, but I don’t know, in the type work I do I couldn’t turn around and go and do what this man was doing because I would probably have a coronary, too. Or, if I went out and tried to play a game of tennis I would probably have a coronary. You can have one without any previous damage, too, and it can be due to sudden exertion or it can — well, the heart is just a very important organ and an awful lot of things can happen to it.”

On cross-examination Dr. Morris explained his position in this manner:

Q- “Doctor, did you feel that the examination you made, and the diagnosis you made as to the cause of death being coronary occlusion, was caused by or brought about by his man’s work ?”
A- “This is drawing a conclusion which the anatomic pathologist is not supposed to do, actually, Mr. Francis. We report the findings, and then it is up to the interpretation of the attending physician. In other words, we are a doctor’s doctor. We do the strict anatomical and clinical testing, and, of course, we see so much, and we read, but our job is to report just what we find.”

The principal medical witness for the claimants was Dr. Franklin B. Moosnick, of Lexington, Kentucky, a member of the faculty at the University of Kentucky Medical School and a specialist in internal medicine. The opinion and order of the board denying the claim quotes certain excerpts from Dr. Moosnick’s testimony with the comment that at best it “merely shows that the work in which the decedent was engaged could have precipitated the coronary thrombosis. This is not sufficient testimony upon which to base an award. Kelly Contracting Company v. Robinson, Ky., 377 S.W.2d 892.” It is our view that this interpretation rather underrates the evidence in question. Though Dr. Moosnick was careful not to assume a dogmatic position, we think the fair import of his testimony was that Johnson’s strenuous physical activity just prior to the fatal onset probably was a precipitating factor. We quote, for example, this concluding statement given on direct examination:

“In this instance I think it is within the realm of probability that this man, with coronary disease and with some fresh occlusion, had that occlusion converted into a clinically evident situation by extreme work or discrete or extreme demand upon himself of whatever type, [564]*564that the resulting ischemia or lack of adequate blood flow, or anoxia, which is lack of proper oxygen content, may have disturbed the pacemaker and conduction system to the point that a fatal arrhythmia resulted. I can state on the basis of the ramifications I have already spoken about that such a sequence in events may indeed have occurred, and that one could not deny that such a sequence of events did occur.”

And on cross-examination:

“It could have happened at any time. I’ll go further, I’ll grant you it could have caused his death sitting in his own living room. The fact that this man was working and apparently — working hard, would, it seems to me, — make him more liable to converting this potential situation. In other words, he has an actual occlusion, but it has not infarcted, it has not produced symptoms. Then something occurs which converts this latent situation into an actual situation, which we have testimony which did occur as far as the bystanders could see, and then in a rapid sequence this man underwent some acute happening which took his life.”

Dr. J. L. Patterson, a general practitioner of Wheelwright, Kentucky, was the physician who was called to the scene and who pronounced Johnson dead. He testified as follows:

Q- “In your opinion, sir, was this condition which caused Mr. Johnson’s death related to his work in any manner ?”
A- “No, sir.”
Q- “Why do you say that ?”

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Bluebook (online)
439 S.W.2d 562, 1969 Ky. LEXIS 372, Counsel Stack Legal Research, https://law.counselstack.com/opinion/inland-steel-co-v-johnson-kyctapp-1969.