Cauthorn v. Owens Corning Fiberglas Corp.

840 A.2d 1028, 2004 Pa. Super. 1, 2004 Pa. Super. LEXIS 2
CourtSuperior Court of Pennsylvania
DecidedJanuary 5, 2004
StatusPublished
Cited by20 cases

This text of 840 A.2d 1028 (Cauthorn v. Owens Corning Fiberglas Corp.) is published on Counsel Stack Legal Research, covering Superior Court of Pennsylvania primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.

Bluebook
Cauthorn v. Owens Corning Fiberglas Corp., 840 A.2d 1028, 2004 Pa. Super. 1, 2004 Pa. Super. LEXIS 2 (Pa. Ct. App. 2004).

Opinion

OLSZEWSKI, J.

¶ 1 Francis E. Cauthorn brought suit against John Crane, Inc. and other companies that either sold or manufactured asbestos products. Plaintiff Cauthorn, the appellee in this case, alleged that he is suffering from symptomatic asbestos-related diseases caused by exposure to asbestos products. This trial was conducted in a reverse-bifurcated fashion before a jury, with the jury first determining appellee’s damages and then apportioning liability for these damages between the defendant companies. John Crane, Inc. was held liable for a portion of Mr. Cauthorn’s injuries and now appeals. 1

FACTS

The Phase I Trial: Determination of Damages

¶ 2 At the time of trial, Mr. Cauthorn was 64 years old. For 14 years of his life (from 1966 until 1980) he built and inspected furnaces for the Leeds and Northrup Company. He testified that he handled and was surrounded by asbestos-containing products every day during this time. Further, the building of furnaces required him to cut, or inspect others while they were cutting, these asbestos-containing products. It was the cutting of the products that created dust he would then breathe in. This dust also covered his *1031 work uniform so that at the end of the day he would have to take an airhose and blow the dust off of his clothing, thus causing the dust to recirculate and him to breathe in even more dust.

¶ 3 In 1999, Mr. Cauthorn began to experience trouble with his breathing. This prevented him from walking “just about anyplace.. .without getting out of breath or short of breath.” Because of this, he was and still is forced to drive everywhere he wishes to go, even if it is a couple of blocks away. Mr. Cauthorn is also suffering from quite a few other serious health conditions. He testified that he is taking medication for high blood pressure, diabetes, high cholesterol, a kidney disorder, a heart condition, and an occasional attack of gout. In his youth he contracted pneumonia, and in 1999 he underwent triple bypass heart surgery. Further, he testified that he smoked a pack and a half to two packs of cigarettes a day from the time he was in high school until he quit around 1997 or 1999 (which is about 40 years of heavy smoking).

¶ 4 In preparation for trial, Dr. Stanley L. Altschuler examined Mr. Cauthorn. Dr. Altschuler testified as an expert at trial and told the jury of his examination findings, as well as his opinions regarding his examination of Mr. Cauthorn. The doctor declared that when listening to Mr. Cauthorn’s lungs, he heard “crackles or rales” in the bottom of both lungs. As it was occurring in the bottoms of both lungs, Dr. Altschuler declared that this finding suggested plaintiff was suffering from interstitial fibrosis, a type of which is asbestosis.

¶ 5 The doctor followed this by describing three breathing tests he performed on Mr. Cauthorn and found an abnormal diffusion capacity in his lungs. The doctor declared: “I can’t say that [Mr. Cauthorn] has abnormal, but I have a hesitancy to say that this is actually a normal lung volume.”

¶ 6 Finally, Dr. Altschuler testified as to his review of a number of Mr. Cauthorn’s chest x-rays as well as a CAT scan of Mr. Cauthorn’s chest. On the basis of this evidence, Dr. Altschuler testified that Mr. Cauthorn:

[h]ad evidence of pleural thickening. He had abnormalities on both diaphragms with a calcification, which is very characteristic of asbestos disease. On one diaphragm the calcification wasn’t present. On the other diaphragm, on the CAT scan, we can see also some calcification on the chest wall on the right side and scarring. That’s for the pleural portion of it. In addition on the chest x-ray there is scarring that we can see in the lung tissue and we see scarring on the CAT scan also.

¶ 7 Dr. Altschuler was familiar with plaintiffs extensive list of physical ailments as well as plaintiffs former smoking habit. Even so, the doctor declared that, taking everything together, “Mr. Cauthorn has asbestosis and asbestos-related pleural disease.” According to the doctor, “[e]ach breath of asbestos that [Mr. Cauthorn] inhaled contributed to his development of [these] asbestos diseases.” Further, the doctor declared that Mr. Cauthorn’s asbestos diseases are “a contributing factor to his shortness of breath” and his “abnormal diffusion capacity.”

¶ 8 While on cross-examination Dr. Alt-schuler admitted that he could not distinguish “what percentage of [Mr. Cauthorn’s diffusion capacity abnormality] is caused by cigarettes and how much is caused by asbestos,” the doctor reiterated his opinion that “cigarette smoking and asbestos exposure [were] contributing factors in [Mr. Cauthorn’s] diffusion abnormality.”

*1032 ¶ 9 John Crane, Inc. presented opposing expert testimony of Dr. Thaddeus C. Bart-ter. Dr. Bartter had also examined Mr. Cauthorn in preparation for the trial. Dr. Bartter testified, however, that he did not hear any “crackling” sounds emanating from plaintiffs chest. The doctor also performed breathing tests on Mr. Cauthorn and found that his lungs were “minimally restrictive.” Since three months prior to this test Mr. Cauthorn had “totally normal” lung volume, Dr. Bartter concluded that plaintiffs lung restriction was not asbestos-related, but rather due to “something like smoking or asthma.”

¶ 10 In reviewing Mr. Cauthorn’s x-rays and CAT scans, Dr. Bartter found only one-sided pleural calcification, suggesting that Mr. Cauthorn does not have asbestosis. Finally, Dr. Bartter concluded: “in this particular case ... there is no evidence of asbestosis.” The doctor further declared that plaintiffs shortness of breath was due, not to an asbestos-related disease, but rather to plaintiffs long history of smoking and the doctor’s conclusion that plaintiff had tuberculosis in his past.

¶ 11 The jury found that Mr. Cauthorn had indeed suffered an asbestos-related injury and awarded him $150,000 in damages. This concluded the Phase I trial. The Phase II Trial: Determination of Liability

¶ 12 Two days after this verdict, Phase II of the trial began. Plaintiff began by calling Dr. Altschuler. The doctor testified as to the harmful and cumulative effect of asbestos. He testified that “[b]e-cause any asbestos fiber will cause some degree of injury.. .each fiber will have some small effect and it’s the cumulative effect of all the different fibers.” Further, he declared “[e]ach breath of air that contained asbestos fibers substantially contributed to the development of [Mr. Caut-horn’s] diseases.” As to Mr. Cauthorn’s shortness of breath, Dr. Altschuler found “the asbestos fibers.. .would have caused small amounts of injury which would accumulate to reach a point that it has in Mr. Cauthorn where we can visibly see it on chest x-rays and he can experience it by his symptoms of shortness of breath.”

¶ 13 Mr. Cauthorn testified next as to product identification. He stated that both John Crane, Inc. and PARS supplied asbestos rope to his workplace. He knew this because he saw the names of both corporations on the packages of rope with the word “asbestos” printed on the packages.

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Cite This Page — Counsel Stack

Bluebook (online)
840 A.2d 1028, 2004 Pa. Super. 1, 2004 Pa. Super. LEXIS 2, Counsel Stack Legal Research, https://law.counselstack.com/opinion/cauthorn-v-owens-corning-fiberglas-corp-pasuperct-2004.