Capital Hilton Hotel v. District of Columbia Department of Employment Services

565 A.2d 981, 1989 D.C. App. LEXIS 222, 1989 WL 132202
CourtDistrict of Columbia Court of Appeals
DecidedNovember 6, 1989
Docket88-704
StatusPublished
Cited by6 cases

This text of 565 A.2d 981 (Capital Hilton Hotel v. District of Columbia Department of Employment Services) is published on Counsel Stack Legal Research, covering District of Columbia Court of Appeals primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.

Bluebook
Capital Hilton Hotel v. District of Columbia Department of Employment Services, 565 A.2d 981, 1989 D.C. App. LEXIS 222, 1989 WL 132202 (D.C. 1989).

Opinion

FARRELL, Associate Judge:

In April 1985, claimant Napoleon Davila (claimant or Davila) was lifting and carrying tables while engaged in his duties as a banquet houseman for the Capital Hilton Hotel (petitioner or Hilton) when he suffered a rupture of a berry aneurysm at the base of his brain. Following an emergency operation and Davila’s partial recovery, the Department of Employment Services (DOES) awarded him temporary total disability benefits from the date of the injury to the present time. Hilton has petitioned for review, raising claims that relate to both the “arising out of” and “accidental injury” elements of a compensable injury under the District of Columbia Workers' Compensation Act of 1979, D.C.Code §§ 36-301 to -345 (1988). The principal among these contentions is that before compensation can be awarded to an injured employee suffering from a preexisting heart or vascular disorder, the claimant must offer proof that the injuries were precipitated by an exertion unusual for that employee. We affirm the order awarding compensation.

I.

Claimant’s duties as a banquet houseman consisted of setting up tables, assembling speaker platforms, and putting up room dividers for banquet functions, at the Hilton. As a normal part of his job, he carried tables and platform sections each weighing forty pounds or more and up to eight feet in length. The most intensive work took place between 10:00 a.m. and 2:00 p.m., and during these hours — at least at the busy time of year, which included April — a crew of six to eight housemen might have to set up a room in as little as five or ten minutes for each such “turnover”. Claimant had worked for Hilton for more than five years. 1 Apparently to his employer’s satisfaction, he prided himself on working faster than anyone else; 2 unlike his coworkers, he regularly carried two tables at a time and a single platform section by himself.

On April 24, 1985, claimant was part of a work crew consisting of four men instead of the usual six to eight, which required him, as the hearing examiner found, to work faster than usual. 3 Shortly before 11:45 a.m., claimant and two coworkers were transporting tables to the storage room. Claimant was carrying two forty *983 pound tables by himself. Carlos Velasquez, who was second in line behind him, testified that the worker immediately behind claimant came running and told him that Davila was on the floor. A paramedic was summoned and claimant was rushed to the hospital, where Dr. Arthur I. Kobrine examined him and concluded that he had suffered a subarachnoid hemorrhage or rupture of a berry aneurysm. Dr. Kobrine operated on Davila and removed or “clipped” two unruptured aneurysms as well as the one that had caused the injury.

At the workers’ compensation hearing, the expert medical testimony was provided by Dr. Kobrine for the claimant and Dr. Raymond B. Jenkins for the employer. 4 The experts agreed in describing the nature and formation of berry aneurysms, but they disagreed sharply on whether a causal relationship exists between physical exertion and increased blood pressure, on the one hand, and the rupture of aneurysms on the other. A berry aneurysm is a blistering or ballooning out of an arterial wall, usually at the base of the brain, which develops as a result of a congenital weakness of the muscle wall of the artery. Although some aneurysms remain static and never burst, others enlarge from the constant pressure of blood across the arterial wall, and as they do so the wall becomes thinner. If bleeding occurs it may be slow, causing headaches, stiff necks or backaches, or it may be apoplectic, resulting in a stroke with cataclysmic results. Claimant’s ruptured aneurysm measured llk centimeters, which both doctors agreed was moderately big.

On the basis of claimant's medical record and relevant studies in the field, Dr. Jenkins expressed the opinion that there was no causal connection between the work claimant was performing and the rupture of his aneurysm. Ultimately, in Dr. Jenkins’ view, the fact that claimant was at work when the aneurysm burst was a matter of “statistics” or happenstance. He agreed that claimant had been diagnosed as having hypertension. He explained, however, that increased blood pressure does not correlate statistically with the rupture of aneurysms, and that patients clinically studied who had suffered from hypertension did not experience a greater number of ruptured aneurysms than other people. Dr. Jenkins was similarly unable to link physical exertion to ruptured aneurysms, pointing out that “when you take a series of people who have burst their aneurysms, it is spread across all activities, from sitting watching television, to being fast asleep in bed, to cheering on the Redskins.” He cited a clinical study of 140 patients in which, for example, 13 had experienced a ruptured aneurysm while sleeping, 24 had done so while at relative rest, and 23 during more strenuous activity. Dr. Jenkins disagreed, therefore, with the “usual wisdom” that increased exertion and blood pressure are a major mechanism in causing aneurysms to burst. At the same time, he agreed that “in certain specific instances a rise of blood pressure is correlated with the bleeding of an aneurysm,” giving the example of people cheering for the Redskins; and he noted that persons awaiting operation for a leaking aneurysm will commonly be given stool softeners to avoid the strain that can produce an aneurysmal rupture. 5

*984 Dr. Jenkins also described the symptoms and likely consequences of a “sentinel” or premonitory bleed of an aneurysm. A sentinel bleed usually produces a severe headache and possibly back stiffness, and most aneurysms that have bled in this manner go on to rupture. Noting that Davila had recently complained of headaches and neck stiffness, and was not a “headachy” person, Dr. Jenkins concluded that he had experienced a sentinel bleed. This fact, however, merely fortified the doctor’s ultimate conclusion: Davila’s aneurysm was “of such a size with a wall of such thinness” that it was “in a state of imminent bleeding” whether or not it had previously bled. Because a rupture of the aneurysm had “reached the point of inevitability,” it was irrelevant in the end whether Davila was at work or what kind of activity he was engaged in when the rupture occurred. The aneurysm would have ruptured “minutes later or hours later,” at most within “a number of tens of hours,” and not “days and weeks later.” It “ruptured because it was ready to.”

Dr. Kobrine differed sharply with Dr. Jenkins about the correlation between ruptured aneurysms, increased blood pressure, and physical exertion. In his opinion, the activity Davila was engaged in on the date of his injury was the direct precipitating cause of the rupture of his aneurysm. Dr. Kobrine explained that an aneurysm enlarges and its walls grow thin because the arterial wall of the blood vessel cannot withstand the pressures of the blood in the blood vessel.

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Bluebook (online)
565 A.2d 981, 1989 D.C. App. LEXIS 222, 1989 WL 132202, Counsel Stack Legal Research, https://law.counselstack.com/opinion/capital-hilton-hotel-v-district-of-columbia-department-of-employment-dc-1989.