Beaucoudray v. Walsh

9 So. 3d 916, 2007 La.App. 4 Cir. 0818, 2009 La. App. LEXIS 388, 2009 WL 635711
CourtLouisiana Court of Appeal
DecidedMarch 12, 2009
Docket2007-CA-0818
StatusPublished
Cited by13 cases

This text of 9 So. 3d 916 (Beaucoudray v. Walsh) is published on Counsel Stack Legal Research, covering Louisiana Court of Appeal primary law. Counsel Stack provides free access to over 12 million legal documents including statutes, case law, regulations, and constitutions.

Bluebook
Beaucoudray v. Walsh, 9 So. 3d 916, 2007 La.App. 4 Cir. 0818, 2009 La. App. LEXIS 388, 2009 WL 635711 (La. Ct. App. 2009).

Opinion

PAUL A. BONIN, Judge.

|2In this medical malpractice case, plaintiffs, Todd Beaucoudray and his parents, appeal a jury verdict in favor of the defendants, Dr. John Walsh and Dr. James T. Bennett. 1 Finding that there was a reasonable basis for the jury’s findings of fact and determination that the plaintiffs failed to bear their burden of proof on the standard of care required of the defendants in their treatment of Todd Beaucoudray (“Todd”), for the reasons given below, we affirm the trial court judgment dismissing the case with prejudice.

TODD BEAUCOUDRAY’S MEDICAL HISTORY

An understanding of Todd Beaucoud-ray’s complex congenital medical and developmental conditions that existed prior to his treatment by Dr. Walsh and Dr. Bennett is helpful in evaluating the claim of medical malpractice made in this case. Todd Beaucoudray was born in 1982 with multiple health problems, including but not limited to, congenital spinal abnormalities. At age eleven, he was of short stature for his age. He received growth hormone therapy. He had previously had |3surgery to correct his club feet. His hearing was partially impaired. Past medical history indicated that he suffered from hernias. He appeared somewhat barrel chested due to rib prominence.

Todd was a good student. He was ambulatory and active, participating in all his activities except contact sports. His upper *919 extremities were normal and he had no problems with his hands. X-rays of Todd’s spine revealed abnormalities; however, there was no evidence of scoliosis.

Todd and his parents were eager that he lead a full life and sought medical care that would help them realize their hopes for their son. He and his parents sought medical treatment to give him the most mobile life possible, for as much of his life as possible. In 1993, the Beaucoudrays were referred to Dr. Bennett, an orthopae-dist, for his opinion. Records indicate that Todd had already begun showing signs of reduced motor functionality, and his mother expressed that fear to Dr. Bennett. X-rays taken in 1994 showed spinal abnormalities but no evidence of scoliosis or abnormal kyphosis. However, X-rays taken in April 1997, prior to the surgery at issue, showed a marked change in Todd’s spinal alignment, including significant scoliosis and major kyphosis changes. 2

Todd’s scoliosis was classified as congenital, i.e., caused by vertebral anomalies present at birth. In the upper thoracic or chest area of the spine, his spine curved sideways, i.e. “left-directed” scoliosis. The curve of his spine from |4front to back was more pronounced than normal, which doctors identified as congenital acute angled thoracic kyphosis. Rather than lying in a gradual curve, his spine arched sharply forward. Particularly in x-rays of the thoracic spine, his vertebrae were not clearly defined.

In his cervical ■ spine, individual vertebrae were almost completely normal. However, his lumbar spine was noted to be very short and his lower ribs were less than an inch from his pelvic bone, reducing space for his internal organs. X-rays of the pelvic area indicated that Todd’s left leg was about one-half inch shorter than his right leg. Further diagnostic studies showed that some vertebrae were triangle-shaped rather than stacked like rectangular blocks. Todd’s lumbar vertebrae lacked pedicles, whose primary function is to act as a bridge to protect the spinal cord and give stability to the vertebrae.

In August of 1997, Todd reported having weakness in his legs which could indicate neurologic symptoms due to spinal cord problems. As Todd grew, his spinal cord was pulled over the “angular kink” of the kyphosis and became irritated. MRI’s showed the spinal cord had normal room in the cervical area, passed through an extremely narrow and tight area in the thoracic area, and then once again had normal room in the lumbar area. One vertebra at the sharpest point of the “kink” was almost one-half normal size. If the kyphosis or “kink” were to be straightened out, a large gap in the bony structure would leave the spinal cord unprotected. Additional studies of his spine showed that with the exception of vertebrae in his neck, Todd did not have one normal vertebra.

|r,Because he had not finished growing at age fifteen, Todd’s condition was expected to worsen. Without surgical intervention, his spinal cord would eventually become compressed, and almost certainly Todd would ultimately become totally paralyzed.

Treatment by Defendants, Dr. Bennett and Dr. Walsh

Dr. Bennett examined Todd in September of 1993 and again in 1994, and noted his congenital spinal deformities. He both offered and recommended regular followup. In 1997, at age fifteen, Todd returned *920 to Dr. Bennett. He wanted to be evaluated prior to beginning a weight-lifting program, as well as to have his flared ribs corrected. Also, his mother expressed her concern that Todd’s spine might be deteriorating.

During the 1997 examination, Dr. Bennett noted abnormal reflexes, three beats of clonus (muscular contractions which may indicate spinal cord damage), and abnormal x-rays. As a result, Dr. Bennett ordered an MRI to assess any spinal cord impingement. In layman’s terms, the MRI revealed that Todd’s spinal cord was irritated as it was pulled over the “kink”, and significantly flattened out as it made a sharp turn over that “kink”.

Dr. Bennett suggested options to stabilize Todd’s spine, avoid further cord irritation, and avoid development of other neu-rologic problems. He referred the family to Dr. John Walsh, a pediatric neurosurgeon, for a determination of appropriate surgical intervention(s).

| (iAt that time, there were two surgical options: first, in situ fusion and second, decompression. An in situ fusion fuses the spine in its current position so that the existing curvature does not worsen. A decompression removes bone which is causing compression to the spinal cord. Neither option corrects or straightens the spine. Due to Todd’s unusual physical deformities which could complicate surgery, numerous additional specialists, including a neurosurgeon, a pediatric general surgeon, a pulmonologist, a neurologist, and a neuroradiologist, were consulted and participated in the evaluation of medically appropriate treatment.

Dr. Walsh performed a neurological assessment and found that at that point Todd had normal neurological function, indicative that the cord had sufficient room. This finding essentially excluded decompression as an initial surgical option. Dr. Walsh also confirmed that as Todd’s spinal cord passed over the top of the “kink”, it became markedly thin and displaced to one side. He recommended in situ fusion, to be performed with the assistance of a pediatric general surgeon, as a conservative approach. 3

A Choice of Procedures

As previously noted, complete work-ups were performed by numerous specialists prior to surgery being authorized.

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9 So. 3d 916, 2007 La.App. 4 Cir. 0818, 2009 La. App. LEXIS 388, 2009 WL 635711, Counsel Stack Legal Research, https://law.counselstack.com/opinion/beaucoudray-v-walsh-lactapp-2009.